Edgar Bernardo
Impact in
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- Adipose Tissue and Metabolism
- Asthma and respiratory diseases
Papers in
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- Epigenetics and DNA Methylation 3
- Protein Degradation and Inhibitors 1
- Histone Deacetylase Inhibitors Research 1
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- Adipose Tissue and Metabolism 4
- Co-authors
- Mercedes Rincón (2 shared papers)Miguel Marcos (3 shared papers)Roger J. Davis (1 shared paper)Matthew J. Wargo (1 shared paper)Viswas Konasagara Nagaleekar (1 shared paper)Wendy Neveu (1 shared paper)Elianne Burg (1 shared paper)Michael Radermacher (1 shared paper)
- Journals
- The EMBO Journal (1 paper)Frontiers in Cell and Developmental Biology (1 paper)Nature Communications (1 paper)Journal of Gastrointestinal Surgery (1 paper)Developmental Cell (1 paper)
- Partner nations
- SpainUnited StatesUnited Kingdom
In The Last Decade
Edgar Bernardo
8 papers receiving 290 citations
Peers
Comparison fields: 5 of 76
- Physiology 79
- Cancer Research 38
- Molecular Biology 157
- Biological Psychiatry 5
- Epidemiology 62
Countries citing papers authored by Edgar Bernardo
This map shows the geographic impact of Edgar Bernardo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edgar Bernardo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edgar Bernardo more than expected).
Fields of papers citing papers by Edgar Bernardo
This network shows the impact of papers produced by Edgar Bernardo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edgar Bernardo. The network helps show where Edgar Bernardo may publish in the future.
Co-authors
The 25 scholars most cited alongside Edgar Bernardo, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2013 | 81 | |
| 2 | 2017 | 57 | |
| 3 | 2011 | 47 | |
| 4 | 2020 | 46 | |
| 5 | 2015 | 25 | |
| 6 | 2021 | 18 | |
| 7 | 2022 | 12 | |
| 8 | 2022 | 4 |
About Edgar Bernardo
Edgar Bernardo is a scholar working on Molecular Biology, Physiology, Surgery, Oncology and Epidemiology, having authored 8 papers that have together received 290 indexed citations. Recurring topics across this work include Adipose Tissue and Metabolism (4 papers), Epigenetics and DNA Methylation (3 papers), Pancreatic function and diabetes (2 papers), Adipokines, Inflammation, and Metabolic Diseases (2 papers), Cytokine Signaling Pathways and Interactions (1 paper), Protein Degradation and Inhibitors (1 paper), Herbal Medicine Research Studies (1 paper) and Histone Deacetylase Inhibitors Research (1 paper). The work is most often cited by research in Physiology (79 citations), Cancer Research (38 citations), Molecular Biology (157 citations), Biological Psychiatry (5 citations) and Epidemiology (62 citations). Edgar Bernardo has collaborated with scholars based in Spain, United States and United Kingdom. Frequent co-authors include Mercedes Rincón, Miguel Marcos, Roger J. Davis, Matthew J. Wargo, Viswas Konasagara Nagaleekar, Wendy Neveu, Elianne Burg, Michael Radermacher, Juan Anguíta and Nicolás Navasa. Their work appears in journals such as The EMBO Journal, Frontiers in Cell and Developmental Biology, Nature Communications, Journal of Gastrointestinal Surgery and Developmental Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.