Dustin E. Bosch

44 papers receiving 1.3k citations

Hit Papers

A bacterial cytidine deaminase toxin enables CRISPR-free mitochondrial base editing 2020 · 483 citations
4832020202620222024100200300400

Peers

Dustin E. Bosch
Comparison fields: 5 of 94
  • Endocrinology 109
  • Clinical Biochemistry 92
  • Molecular Biology 886
  • Aging 19
  • Molecular Medicine 47
Replace Peter Schotte with:
Peter Schotte Belgium
Cécile Arrieumerlou France
Nobumichi Furuta Japan
Andreas Lengeling Germany
Esteban A. Roberts United States
Jürg M. Sommer United States
Saı̈d Taouji France
Carmela Galli Switzerland
Ryuji Yamaguchi Japan
Lucas T. Jae Germany
Dustin E. Bosch relative to Peter Schotte Belgium Peter Schotte's profile →
Citations per field
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Peter Schotte · 1×
Citations per year

Countries citing papers authored by Dustin E. Bosch

Since Specialization
Citations

This map shows the geographic impact of Dustin E. Bosch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dustin E. Bosch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dustin E. Bosch more than expected).

Fields of papers citing papers by Dustin E. Bosch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dustin E. Bosch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dustin E. Bosch. The network helps show where Dustin E. Bosch may publish in the future.

Co-authors

The 25 scholars most cited alongside Dustin E. Bosch, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Dustin E. Bosch Line = papers co-authored together Dustin E. Bosch links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
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11 20191
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13 20182
14 201726
15 201340
16 201215
17 201230
18 20127
19 201115
20 200842

About Dustin E. Bosch

Dustin E. Bosch is a scholar working on Infectious Diseases, Endocrinology, Hepatology, Surgery and Oncology, having authored 48 papers that have together received 1.4k indexed citations. Recurring topics across this work include Amoebic Infections and Treatments (9 papers), Protein Kinase Regulation and GTPase Signaling (7 papers), Pancreatitis Pathology and Treatment (6 papers), Pancreatic and Hepatic Oncology Research (5 papers), Liver Disease and Transplantation (4 papers), Liver Disease Diagnosis and Treatment (4 papers), Helicobacter pylori-related gastroenterology studies (4 papers) and Receptor Mechanisms and Signaling (4 papers). The work is most often cited by research in Endocrinology (109 citations), Clinical Biochemistry (92 citations), Molecular Biology (886 citations), Aging (19 citations) and Molecular Medicine (47 citations). Dustin E. Bosch has collaborated with scholars based in United States, Israel and Singapore. Frequent co-authors include David P. Siderovski, Adam J. Kimple, S. Brook Peterson, Joseph D. Mougous, Philippe Giguère, Matthew C. Radey, FoSheng Hsu, Jun Zeng, Marcos H. de Moraes and Beverly Mok. Their work appears in journals such as Journal of Biological Chemistry, Human Pathology, Applied immunohistochemistry & molecular morphology, Biochemistry and Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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