Dieter De Coninck
- Molecular Biology
- Health, Toxicology and Mutagenesis top 5%
- Genetics top 10%
- Environmental Chemistry top 5%
- Ecology top 10%
- Co-authors
- Colin JanssenKarel De SchamphelaereFilip Van NieuwerburghJana AsselmanDieter DeforceM. VandegehuchteWim M. De CoenTine Vandenbrouck
- Topics
- Environmental Toxicology and Ecotoxicology (9 papers)Genomic variations and chromosomal abnormalities (5 papers)Epigenetics and DNA Methylation (4 papers)
- Partner nations
- BelgiumUnited StatesNetherlands
In The Last Decade
Dieter De Coninck
31 papers receiving 855 citations
Peers
Comparison fields: 5 of 94
- Molecular Biology 359
- Health, Toxicology and Mutagenesis 193
- Genetics 182
- Environmental Chemistry 139
- Ecology 132
Countries citing papers authored by Dieter De Coninck
This map shows the geographic impact of Dieter De Coninck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dieter De Coninck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dieter De Coninck more than expected).
Fields of papers citing papers by Dieter De Coninck
This network shows the impact of papers produced by Dieter De Coninck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dieter De Coninck. The network helps show where Dieter De Coninck may publish in the future.
Co-authorship network of co-authors of Dieter De Coninck
This figure shows the co-authorship network connecting the top 25 collaborators of Dieter De Coninck. A scholar is included among the top collaborators of Dieter De Coninck based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dieter De Coninck. Dieter De Coninck is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 1 | |
| 3 | 14 | |
| 4 | 13 | |
| 5 | 13 | |
| 6 | 11 | |
| 7 | 52 | |
| 8 | 18 | |
| 9 | 1 | |
| 10 | 10 | |
| 11 | 12 | |
| 12 | 60 | |
| 13 | 39 | |
| 14 | 41 | |
| 15 | 38 | |
| 16 | 47 | |
| 17 | 21 | |
| 18 | 18 | |
| 19 | 32 | |
| 20 | 30 |
About Dieter De Coninck
Dieter De Coninck is a scholar working on Endocrinology, Health, Toxicology and Mutagenesis and Molecular Medicine, having authored 32 papers that have together received 864 indexed citations. Recurring topics across this work include Environmental Toxicology and Ecotoxicology (9 papers), Genomic variations and chromosomal abnormalities (5 papers) and Epigenetics and DNA Methylation (4 papers). The work is most often cited by research in Health, Toxicology and Mutagenesis (193 citations), Environmental Chemistry (139 citations) and Aging (21 citations). Dieter De Coninck has collaborated with scholars based in Belgium, United States and Netherlands. Frequent co-authors include Colin Janssen, Karel De Schamphelaere, Filip Van Nieuwerburgh, Jana Asselman, Dieter Deforce, M. Vandegehuchte, Wim M. De Coen, Tine Vandenbrouck, Michael E. Pfrender and Joseph R. Shaw. Their work appears in journals such as Nature Communications, Environmental Science & Technology and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.