Di Qi

1.1k total citations
33 papers, 801 citations indexed

About

Di Qi is a scholar working on Pulmonary and Respiratory Medicine, Immunology and Molecular Biology. According to data from OpenAlex, Di Qi has authored 33 papers receiving a total of 801 indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Pulmonary and Respiratory Medicine, 11 papers in Immunology and 10 papers in Molecular Biology. Recurrent topics in Di Qi's work include Respiratory Support and Mechanisms (12 papers), Neonatal Respiratory Health Research (9 papers) and Immune Response and Inflammation (6 papers). Di Qi is often cited by papers focused on Respiratory Support and Mechanisms (12 papers), Neonatal Respiratory Health Research (9 papers) and Immune Response and Inflammation (6 papers). Di Qi collaborates with scholars based in China. Di Qi's co-authors include Daoxin Wang, Jing He, Xinyu Deng, Wang Deng, Xumao Tang, Yan Zhao, Wen Li, Longhua Feng, Yan Zhao and Qian Yu and has published in prestigious journals such as Scientific Reports, Biochemical and Biophysical Research Communications and Experimental Cell Research.

In The Last Decade

Di Qi

33 papers receiving 793 citations

Peers

Di Qi

PRM

IMMUN

EPIDE

Wang Deng China

Hongguang Nie China

Raquel Herrero Spain

Xiaoyuan Yang United States

Zhigang Chang China

Yimin Sun United States

Jolien Vandewalle Belgium

Heidi Ehrentraut Germany

Martin Witzenrath Germany

Wang Deng China

Di Qi

353 ×1.2

281 ×1.2

PRM

207 ×1.2

IMMUN

152 ×1.1

EPIDE

93 ×0.9

Citations per year, relative to Di Qi Di Qi (= 1×) peers Wang Deng

Countries citing papers authored by Di Qi

Since Specialization

Citations

This map shows the geographic impact of Di Qi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Di Qi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Di Qi more than expected).

Fields of papers citing papers by Di Qi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Di Qi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Di Qi. The network helps show where Di Qi may publish in the future.

Co-authorship network of co-authors of Di Qi

This figure shows the co-authorship network connecting the top 25 collaborators of Di Qi. A scholar is included among the top collaborators of Di Qi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Di Qi. Di Qi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Zhang, Rong, et al.. (2025). CGRP alleviates lipopolysaccharide-induced ARDS inflammation via the HIF-1α signaling pathway. Clinical Science. 139(7). 373–387. 1 indexed citations

Wang, Hanghang, et al.. (2025). MFGE8 regulates the EndoMT of HLMECs through the BMP signaling pathway and fibrosis in acute lung injury. Respiratory Research. 26(1). 142–142. 1 indexed citations

He, Rui, et al.. (2024). Activated Clec4nhi Neutrophils Aggravate Lung Injury in an Endothelial IGFBP7-Dependent Manner. American Journal of Respiratory Cell and Molecular Biology. 71(1). 66–80. 4 indexed citations

Tang, Rui, et al.. (2024). S1PR3 inhibition protects against LPS-induced ARDS by inhibiting NF-κB and improving mitochondrial oxidative phosphorylation. Journal of Translational Medicine. 22(1). 535–535. 8 indexed citations

Chen, Xiaorui, et al.. (2023). Interferon regulatory factor 1 (IRF1) inhibits lung endothelial regeneration following inflammation-induced acute lung injury. Clinical Science. 137(5). 367–383. 7 indexed citations

Yu, Qian, et al.. (2022). Protective effects of adipose-derived biogenic nanoparticles on the pulmonary microvascular endothelial barrier in mice with ventilator-induced lung injury via the TRPV4/ROCK1 signalling pathway. Pulmonary Pharmacology & Therapeutics. 73-74. 102123–102123. 7 indexed citations

Qi, Di, Wang Deng, Xiaorui Chen, et al.. (2022). Adipose‐Derived Circulating Exosomes Promote Protection of the Pulmonary Endothelial Barrier by Inhibiting EndMT and Oxidative Stress through Down‐Regulation of the TGF‐β Pathway: A Potential Explanation for the Obesity Paradox in ARDS. Oxidative Medicine and Cellular Longevity. 2022(1). 5475832–5475832. 12 indexed citations

Qi, Di, et al.. (2021). Vagus nerve stimulation enhances the cholinergic anti-inflammatory pathway to reduce lung injury in acute respiratory distress syndrome via STAT3. Cell Death Discovery. 7(1). 63–63. 52 indexed citations

Yu, Qian, et al.. (2021). High Blood Eosinophil and YKL-40 Levels, as Well as Low CXCL9 Levels, are Associated with Increased Readmission in Patients with Acute Exacerbation of Chronic Obstructive Pulmonary Disease. International Journal of COPD. Volume 16. 795–806. 17 indexed citations

10.

Qi, Di, et al.. (2020). Intermedin alleviates the inflammatory response and stabilizes the endothelial barrier in LPS-induced ARDS through the PI3K/Akt/eNOS signaling pathway. International Immunopharmacology. 88. 106951–106951. 13 indexed citations

11.

He, Jing, Di Qi, Xumao Tang, et al.. (2019). Rosiglitazone promotes ENaC-mediated alveolar fluid clearance in acute lung injury through the PPARγ/SGK1 signaling pathway. Cellular & Molecular Biology Letters. 24(1). 35–35. 29 indexed citations

12.

Zhao, Yan, et al.. (2019). Tangeretin attenuates lipopolysaccharide-induced acute lung injury through Notch signaling pathway via suppressing Th17 cell response in mice. Microbial Pathogenesis. 138. 103826–103826. 22 indexed citations

13.

Tang, Xumao, Qian Yu, Di Qi, et al.. (2019). Circulating Exosomes From Lipopolysaccharide-Induced Ards Mice Trigger Endoplasmic Reticulum Stress in Lung Tissue. Shock. 54(1). 110–118. 11 indexed citations

14.

Zhu, Tao, Guihua Chen, Shuo Tang, et al.. (2018). Extent of Lung Involvement and Serum Cryptococcal Antigen Test in Non-Human Immunodeficiency Virus Adult Patients with Pulmonary Cryptococcosis. Chinese Medical Journal. 131(18). 2210–2215. 10 indexed citations

15.

Chen, Yan, et al.. (2018). p300 promotes differentiation of Th17 cells via positive regulation of the nuclear transcription factor RORγt in acute respiratory distress syndrome. Immunology Letters. 202. 8–15. 9 indexed citations

16.

Cheng, Li, Yan Zhao, Di Qi, Wen Li, & Daoxin Wang. (2017). Wnt/β-catenin pathway promotes acute lung injury induced by LPS through driving the Th17 response in mice. Biochemical and Biophysical Research Communications. 495(2). 1890–1895. 21 indexed citations

17.

Liu, Daishun, et al.. (2017). Effect of sivelestat sodium in patients with acute lung injury or acute respiratory distress syndrome: a meta-analysis of randomized controlled trials. BMC Pulmonary Medicine. 17(1). 148–148. 43 indexed citations

18.

Qi, Di, Xumao Tang, Jing He, et al.. (2016). Omentin protects against LPS-induced ARDS through suppressing pulmonary inflammation and promoting endothelial barrier via an Akt/eNOS-dependent mechanism. Cell Death and Disease. 7(9). e2360–e2360. 72 indexed citations

19.

He, Jing, Di Qi, Daoxin Wang, et al.. (2014). Insulin upregulates the expression of epithelial sodium channel in vitro and in a mouse model of acute lung injury: Role of mTORC2/SGK1 pathway. Experimental Cell Research. 331(1). 164–175. 23 indexed citations

20.

Qi, Di, Jing He, Daoxin Wang, et al.. (2014). 17β-estradiol suppresses lipopolysaccharide-induced acute lung injury through PI3K/Akt/SGK1 mediated up-regulation of epithelial sodium channel (ENaC) in vivo and in vitro. Respiratory Research. 15(1). 159–159. 55 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact