Debra Weinstat‐Saslow
- Molecular Biology
- Oncology top 10%
- Cancer Research top 10%
- Genetics top 10%
- Cell Biology top 10%
- Co-authors
- Patricia S. SteegDavid D. RobertsP. S. SteegWilliam A. FrazierVivian ZabrenetzkyStephen T. ReedersDavid L. PageJean F. Simpson
- Topics
- Genetic and Kidney Cyst Diseases (9 papers)Renal and related cancers (6 papers)Hedgehog Signaling Pathway Studies (5 papers)
- Partner nations
- United StatesUnited KingdomNetherlands
In The Last Decade
Debra Weinstat‐Saslow
11 papers receiving 915 citations
Peers
Comparison fields: 5 of 76
- Molecular Biology 671
- Oncology 354
- Cancer Research 284
- Genetics 213
- Cell Biology 110
Countries citing papers authored by Debra Weinstat‐Saslow
This map shows the geographic impact of Debra Weinstat‐Saslow's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Debra Weinstat‐Saslow with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Debra Weinstat‐Saslow more than expected).
Fields of papers citing papers by Debra Weinstat‐Saslow
This network shows the impact of papers produced by Debra Weinstat‐Saslow. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Debra Weinstat‐Saslow. The network helps show where Debra Weinstat‐Saslow may publish in the future.
Co-authorship network of co-authors of Debra Weinstat‐Saslow
This figure shows the co-authorship network connecting the top 25 collaborators of Debra Weinstat‐Saslow. A scholar is included among the top collaborators of Debra Weinstat‐Saslow based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Debra Weinstat‐Saslow. Debra Weinstat‐Saslow is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 48 | |
| 3 | 270 | |
| 4 | 138 | |
| 5 | Transfection of thrombospondin 1 complementary DNA into a human breast carcinoma cell line reduces primary tumor growth, metastatic potential, and angiogenesis. | 294 |
| 6 | 5 | |
| 7 | Positional cloning approach to the dominant polycystic kidney disease gene, PKD1. | 9 |
| 8 | 22 | |
| 9 | 29 | |
| 10 | 65 | |
| 11 | 47 | |
| 12 | 8 |
About Debra Weinstat‐Saslow
Debra Weinstat‐Saslow is a scholar working on Genetics, Molecular Biology and Biochemistry, having authored 12 papers that have together received 935 indexed citations. Recurring topics across this work include Genetic and Kidney Cyst Diseases (9 papers), Renal and related cancers (6 papers) and Hedgehog Signaling Pathway Studies (5 papers). The work is most often cited by research in Cancer Research (284 citations), Oncology (354 citations) and Molecular Biology (671 citations). Debra Weinstat‐Saslow has collaborated with scholars based in United States, United Kingdom and Netherlands. Frequent co-authors include Patricia S. Steeg, David D. Roberts, P. S. Steeg, William A. Frazier, Vivian Zabrenetzky, Stephen T. Reeders, David L. Page, Jean F. Simpson, Maria J. Merino and Richard E. Manrow. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Nature Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.