David R Grubb

2.2k total citations · 1 hit paper
22 papers, 1.9k citations indexed

About

David R Grubb is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Epidemiology. According to data from OpenAlex, David R Grubb has authored 22 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 9 papers in Cardiology and Cardiovascular Medicine and 4 papers in Epidemiology. Recurrent topics in David R Grubb's work include Ion channel regulation and function (6 papers), Cardiac electrophysiology and arrhythmias (6 papers) and Cell death mechanisms and regulation (5 papers). David R Grubb is often cited by papers focused on Ion channel regulation and function (6 papers), Cardiac electrophysiology and arrhythmias (6 papers) and Cell death mechanisms and regulation (5 papers). David R Grubb collaborates with scholars based in Australia, United States and Switzerland. David R Grubb's co-authors include Alfons Lawen, Jennifer Ly, Elizabeth A. Woodcock, Thomas Kaufmann, Christoph Borner, Martin Schüler, Anna C. Schinzel, Theresa M. Filtz, Ernst J. Wolvetang and Karina L Johnson and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Cell Biology and The Journal of Immunology.

In The Last Decade

David R Grubb

22 papers receiving 1.9k citations

Hit Papers

The mitochondrial membrane potential (Δψm) in apoptosis; ... 2003 2026 2010 2018 2003 400 800 1.2k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David R Grubb Australia 13 1.2k 195 164 161 151 22 1.9k
Myeong‐Sok Lee South Korea 26 1.2k 1.0× 239 1.2× 87 0.5× 162 1.0× 272 1.8× 83 2.1k
Yanna Cheng China 29 933 0.8× 266 1.4× 153 0.9× 102 0.6× 218 1.4× 66 2.0k
Sandra Incerpi Italy 29 1.1k 0.9× 291 1.5× 260 1.6× 209 1.3× 190 1.3× 105 2.8k
Hüveyda Başağa Türkiye 28 1.2k 1.0× 234 1.2× 198 1.2× 183 1.1× 261 1.7× 63 2.4k
Tong-Shin Chang South Korea 19 1.7k 1.4× 119 0.6× 149 0.9× 89 0.6× 134 0.9× 26 2.6k
Jon Holy United States 24 986 0.8× 221 1.1× 104 0.6× 88 0.5× 217 1.4× 59 1.8k
Jeong Ho Seok South Korea 26 980 0.8× 151 0.8× 103 0.6× 161 1.0× 217 1.4× 64 1.7k
Jamshid Davoodi Iran 19 1.1k 0.9× 167 0.9× 285 1.7× 90 0.6× 115 0.8× 46 1.7k
Yizhong Zhang China 22 1.3k 1.1× 232 1.2× 168 1.0× 299 1.9× 175 1.2× 56 2.1k
Guang Xu China 28 1.4k 1.1× 321 1.6× 131 0.8× 141 0.9× 295 2.0× 54 2.5k

Countries citing papers authored by David R Grubb

Since Specialization
Citations

This map shows the geographic impact of David R Grubb's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David R Grubb with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David R Grubb more than expected).

Fields of papers citing papers by David R Grubb

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David R Grubb. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David R Grubb. The network helps show where David R Grubb may publish in the future.

Co-authorship network of co-authors of David R Grubb

This figure shows the co-authorship network connecting the top 25 collaborators of David R Grubb. A scholar is included among the top collaborators of David R Grubb based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David R Grubb. David R Grubb is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Grubb, David R, Xiao‐Ming Gao, Helen Kiriazis, et al.. (2016). Expressing an inhibitor of PLCβ1b sustains contractile function following pressure overload. Journal of Molecular and Cellular Cardiology. 93. 12–17. 2 indexed citations
2.
Matkovich, Scot J., David R Grubb, Julie R. McMullen, & Elizabeth A. Woodcock. (2016). Chronic Contractile Dysfunction without Hypertrophy Does Not Provoke a Compensatory Transcriptional Response in Mouse Hearts. PLoS ONE. 11(6). e0158317–e0158317. 3 indexed citations
3.
Grubb, David R, Yi Ma, Hong Qian, et al.. (2015). The atypical ‘b’ splice variant of phospholipase Cβ1 promotes cardiac contractile dysfunction. Journal of Molecular and Cellular Cardiology. 84. 95–103. 10 indexed citations
4.
Grubb, David R, et al.. (2015). Phospholipase Cβ1b directly binds the SH3 domain of Shank3 for targeting and activation in cardiomyocytes. Biochemical and Biophysical Research Communications. 461(3). 519–524. 4 indexed citations
5.
Woodcock, Elizabeth A. & David R Grubb. (2015). Novel Therapeutic Targets in Heart Failure: The Phospholipase Cβ1b–Shank3 Interface. 7. 3 indexed citations
7.
Wong, Albert J., et al.. (2012). Regulation of autophagy in cardiomyocytes by Ins(1,4,5)P3 and IP3-receptors. Journal of Molecular and Cellular Cardiology. 54. 19–24. 25 indexed citations
8.
Pepe, Salvatore, David R Grubb, J.C. Komen, et al.. (2012). Tissue-specific splicing of an Ndufs6 gene-trap insertion generates a mitochondrial complex I deficiency-specific cardiomyopathy. Proceedings of the National Academy of Sciences. 109(16). 6165–6170. 48 indexed citations
9.
Woodcock, Elizabeth A., David R Grubb, & Peter Iliades. (2010). Potential Treatment of Cardiac Hypertrophy and Heart Failure by Inhibiting the Sarcolemmal Binding of Phospholipase Cβ1b. Current Drug Targets. 11(8). 1032–1040. 10 indexed citations
10.
Grubb, David R, et al.. (2010). Phospholipase Cβlb associates with a Shank3 complex at the cardiac sarcolemma. The FASEB Journal. 25(3). 1040–1047. 22 indexed citations
11.
Woodcock, Elizabeth A., David R Grubb, Theresa M. Filtz, et al.. (2009). Selective activation of the “b” splice variant of phospholipase Cβ1 in chronically dilated human and mouse atria. Journal of Molecular and Cellular Cardiology. 47(5). 676–683. 26 indexed citations
12.
Grubb, David R, et al.. (2008). Ins(1,4,5)P3 regulates phospholipase Cβ1 expression in cardiomyocytes. Journal of Molecular and Cellular Cardiology. 45(5). 679–684. 6 indexed citations
14.
Schinzel, Anna C., et al.. (2004). Conformational control of Bax localization and apoptotic activity by Pro168. The Journal of Cell Biology. 164(7). 1021–1032. 124 indexed citations
15.
Ly, Jennifer, David R Grubb, & Alfons Lawen. (2003). The mitochondrial membrane potential (Δψm) in apoptosis; an update. APOPTOSIS. 8(2). 115–128. 1405 indexed citations breakdown →
16.
Baker, Mark A., David R Grubb, & Alfons Lawen. (2002). Didemnin B induces apoptosis in proliferating but not resting peripheral blood mononuclear cells. APOPTOSIS. 7(5). 407–412. 19 indexed citations
17.
Grubb, David R, Jennifer Ly, François Vaillant, Karina L Johnson, & Alfons Lawen. (2001). Mitochondrial cytochrome c release is caspase-dependent and does not involve mitochondrial permeability transition in didemnin B-induced apoptosis. Oncogene. 20(30). 4085–4094. 37 indexed citations
18.
Johnson, Karina L, David R Grubb, & Alfons Lawen. (1999). Unspecific activation of caspases during the induction of apoptosis by didemnin B in human cell lines. Journal of Cellular Biochemistry. 72(2). 269–278. 10 indexed citations
19.
Grubb, David R, Ernst J. Wolvetang, & Alfons Lawen. (1995). Didemnin B Induces Cell Death by Apoptosis: The Fastest Induction of Apoptosis Ever Described. Biochemical and Biophysical Research Communications. 215(3). 1130–1136. 42 indexed citations
20.
Ralph, Stephen J., Bruce D. Wines, Miranda Payne, et al.. (1995). Resistance of melanoma cell lines to interferons correlates with reduction of IFN-induced tyrosine phosphorylation. Induction of the anti-viral state by IFN is prevented by tyrosine kinase inhibitors.. The Journal of Immunology. 154(5). 2248–2256. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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