Helen Kiriazis

6.5k total citations · 1 hit paper
137 papers, 4.9k citations indexed

About

Helen Kiriazis is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Surgery. According to data from OpenAlex, Helen Kiriazis has authored 137 papers receiving a total of 4.9k indexed citations (citations by other indexed papers that have themselves been cited), including 85 papers in Cardiology and Cardiovascular Medicine, 58 papers in Molecular Biology and 23 papers in Surgery. Recurrent topics in Helen Kiriazis's work include Cardiovascular Function and Risk Factors (35 papers), Cardiac Fibrosis and Remodeling (26 papers) and Cardiomyopathy and Myosin Studies (20 papers). Helen Kiriazis is often cited by papers focused on Cardiovascular Function and Risk Factors (35 papers), Cardiac Fibrosis and Remodeling (26 papers) and Cardiomyopathy and Myosin Studies (20 papers). Helen Kiriazis collaborates with scholars based in Australia, United States and China. Helen Kiriazis's co-authors include Xiao‐Jun Du, Julie R. McMullen, Anthony M. Dart, Yidan Su, Xiao‐Ming Gao, Rebecca H. Ritchie, Bianca C. Bernardo, Yow Keat Tham, Xiao‐Ming Gao and Xiao‐Jun Du and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

Helen Kiriazis

125 papers receiving 4.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Relaxin Reverses Cardiac and Renal Fibrosis in Spontaneously Hypertensive Rats

2005 164 citations Helen Kiriazis, Yidan Su et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Helen Kiriazis Australia	41	2.4k	2.3k	711	611	544	137	4.9k
Xiao‐Jun Du Australia	41	1.9k 0.8×	1.8k 0.8×	658 0.9×	389 0.6×	508 0.9×	135	4.5k
Burns C. Blaxall United States	43	3.6k 1.5×	3.2k 1.4×	879 1.2×	519 0.8×	419 0.8×	88	6.3k
Karen E. Porter United Kingdom	44	2.5k 1.0×	1.9k 0.8×	1.3k 1.8×	750 1.2×	460 0.8×	131	5.6k
Mark Aronovitz United States	37	2.1k 0.9×	2.3k 1.0×	809 1.1×	375 0.6×	494 0.9×	98	5.5k
Djahida Bedja United States	35	2.2k 0.9×	2.3k 1.0×	492 0.7×	260 0.4×	602 1.1×	67	4.6k
Deborah A. Siwik United States	37	2.0k 0.8×	2.3k 1.0×	686 1.0×	530 0.9×	530 1.0×	64	5.0k
Yasukatsu Izumi Japan	33	1.7k 0.7×	1.1k 0.5×	569 0.8×	420 0.7×	400 0.7×	87	3.5k
Haruhiro Toko Japan	38	3.5k 1.5×	2.3k 1.0×	1.3k 1.9×	473 0.8×	423 0.8×	71	6.0k
Rajesh Katare New Zealand	45	2.5k 1.0×	1.3k 0.6×	1.0k 1.4×	789 1.3×	231 0.4×	134	5.2k
Clifford D.L. Folmes United States	26	2.9k 1.3×	1.1k 0.5×	615 0.9×	645 1.1×	456 0.8×	37	4.9k

Countries citing papers authored by Helen Kiriazis

Since Specialization

Citations

This map shows the geographic impact of Helen Kiriazis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Helen Kiriazis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Helen Kiriazis more than expected).

Fields of papers citing papers by Helen Kiriazis

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Helen Kiriazis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Helen Kiriazis. The network helps show where Helen Kiriazis may publish in the future.

Co-authorship network of co-authors of Helen Kiriazis

This figure shows the co-authorship network connecting the top 25 collaborators of Helen Kiriazis. A scholar is included among the top collaborators of Helen Kiriazis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Helen Kiriazis. Helen Kiriazis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Jackson, Kristy L., Haoyun Fang, Feng Tang, et al.. (2025). Annexin-A1 deficiency uncovers female-specific pathways in blood pressure control and cardiovascular remodeling in mice. Communications Biology. 8(1). 955–955.

Jackson, Kristy L., Haoyun Fang, Bethany Claridge, et al.. (2024). Novel formylpeptide receptor 1/2 agonist limits hypertension-induced cardiovascular damage. Cardiovascular Research. 120(11). 1336–1350. 6 indexed citations

Mohan, Muthukumar, Eoin Brennan, Helen Kiriazis, et al.. (2024). Lipoxin A4 improves cardiac remodeling and function in diabetes-associated cardiac dysfunction. Cardiovascular Diabetology. 23(1). 413–413. 5 indexed citations

Cohen, Charles D., et al.. (2024). Sex-Specific Mechanisms Driving Cardiac Remodelling in Type 2 Diabetes. Heart Lung and Circulation. 33. S483–S484.

Jackson, Kristy L., Feng Tang, Cameron J. Nowell, et al.. (2024). The pro‐resolving mediator, annexin A1 regulates blood pressure, and age‐associated changes in cardiovascular function and remodeling. The FASEB Journal. 38(3). e23457–e23457. 7 indexed citations

Deo, Minh, Helen Kiriazis, D. Donner, et al.. (2023). A high-sucrose diet exacerbates the left ventricular phenotype in a high fat-fed streptozotocin rat model of diabetic cardiomyopathy. American Journal of Physiology-Heart and Circulatory Physiology. 324(2). H241–H257. 10 indexed citations

Zhuang, Aowen, Yingying Liu, Christine Yang, et al.. (2023). Deletion of the muscle enriched lncRNA Oip5os1 induces atrial dysfunction in male mice with diabetes. Physiological Reports. 11(23). e15869–e15869. 4 indexed citations

Tham, Yow Keat, Bianca C. Bernardo, Bethany Claridge, et al.. (2022). Estrogen receptor α deficiency in cardiac myocytes reprograms heart-derived extracellular vesicle proteome and induces obesity in female mice. Journal of Molecular and Cellular Cardiology. 173. S104–S104. 1 indexed citations

Horlock, Duncan, David M. Kaye, Catherine E. Winbanks, et al.. (2021). Old Drug, New Trick: Tilorone, a Broad-Spectrum Antiviral Drug as a Potential Anti-Fibrotic Therapeutic for the Diseased Heart. Pharmaceuticals. 14(3). 263–263. 6 indexed citations

10.

Prakoso, Darnel, Shiang Y. Lim, Jeffrey R. Erickson, et al.. (2021). Fine-tuning the cardiac O-GlcNAcylation regulatory enzymes governs the functional and structural phenotype of the diabetic heart. Cardiovascular Research. 118(1). 212–225. 66 indexed citations

11.

Weeks, Kate L., Yow Keat Tham, Suzan Yıldız, et al.. (2021). FoxO1 is required for physiological cardiac hypertrophy induced by exercise but not by constitutively active PI3K. American Journal of Physiology-Heart and Circulatory Physiology. 320(4). H1470–H1485. 23 indexed citations

12.

Qin, Cheng Xue, Sarah Rosli, Minh Deo, et al.. (2019). Cardioprotective Actions of the Annexin-A1 N-Terminal Peptide, Ac2-26, Against Myocardial Infarction. Frontiers in Pharmacology. 10. 269–269. 31 indexed citations

13.

Nguyen, My‐Nhan, Helen Kiriazis, Xiao‐Ming Gao, & Xiao‐Jun Du. (2017). Cardiac Fibrosis and Arrhythmogenesis. Comprehensive physiology. 7(3). 1009–1049. 6 indexed citations

14.

Heywood, Sarah, Adèle Richart, Darren C. Henstridge, et al.. (2017). High-density lipoprotein delivered after myocardial infarction increases cardiac glucose uptake and function in mice. Science Translational Medicine. 9(411). 44 indexed citations

15.

Bernardo, Bianca C., Xiao‐Ming Gao, Yow Keat Tham, et al.. (2016). Inhibition of miR-154 Protects Against Cardiac Dysfunction and Fibrosis in a Mouse Model of Pressure Overload. Scientific Reports. 6(1). 22442–22442. 44 indexed citations

16.

Bernardo, Bianca C., Jenny Y. Y. Ooi, Aya Matsumoto, et al.. (2016). Sex differences in response to miRNA‐34a therapy in mouse models of cardiac disease: identification of sex‐, disease‐ and treatment‐regulated miRNAs. The Journal of Physiology. 594(20). 5959–5974. 38 indexed citations

17.

Blasio, Miles J. De, Darnel Prakoso, Cheng Xue Qin, et al.. (2016). Abstract 15267: Cardiac-Specific Insulin-Like Growth Factor-1 Receptor (IGF-1R) Expression Targets Maladaptive Hexosamine Biosynthesis and O-Linked GlcNAc Modification of Sarco/Endoplasmic Reticulum Ca2+-ATPase (SERCA2a) in Diabetic Myocardium. Circulation. 134. 1 indexed citations

18.

Blasio, Miles J. De, Karina Huynh, Cheng Xue Qin, et al.. (2015). Therapeutic targeting of oxidative stress with coenzyme Q10 counteracts exaggerated diabetic cardiomyopathy in a mouse model of diabetes with diminished PI3K(p110α) signaling. Free Radical Biology and Medicine. 87. 137–147. 69 indexed citations

19.

White, David, Fang Lü, William Chan, et al.. (2013). Correction: Pro-Inflammatory Action of MIF in Acute Myocardial Infarction via Activation of Peripheral Blood Mononuclear Cells. PLoS ONE. 8(11). 16 indexed citations

20.

Pretorius, Lynette, Xiao‐Jun Du, Elizabeth A. Woodcock, et al.. (2009). Reduced Phosphoinositide 3-Kinase (p110α) Activation Increases the Susceptibility to Atrial Fibrillation. American Journal Of Pathology. 175(3). 998–1009. 152 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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