David R. Emlet

3.4k total citations · 1 hit paper
24 papers, 2.1k citations indexed

About

David R. Emlet is a scholar working on Molecular Biology, Nephrology and Oncology. According to data from OpenAlex, David R. Emlet has authored 24 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 5 papers in Nephrology and 5 papers in Oncology. Recurrent topics in David R. Emlet's work include Acute Kidney Injury Research (4 papers), Dialysis and Renal Disease Management (3 papers) and Protein Kinase Regulation and GTPase Signaling (3 papers). David R. Emlet is often cited by papers focused on Acute Kidney Injury Research (4 papers), Dialysis and Renal Disease Management (3 papers) and Protein Kinase Regulation and GTPase Signaling (3 papers). David R. Emlet collaborates with scholars based in United States, India and China. David R. Emlet's co-authors include Albert J. Wong, Marina Holgado-Madruga, David K. Moscatello, Andrew K. Godwin, Bruce Montgomery, John A. Kellum, Isamu Okamoto, Lawrence C. Kenyon, Andrew Shaw and Xiao‐Yan Wen and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

David R. Emlet

24 papers receiving 2.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A Grb2-associated docking protein in EGF- and insulin-receptor signalling

1996 578 citations Marina Holgado-Madruga, David R. Emlet et al. Nature profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David R. Emlet United States	16	1.3k	598	261	234	231	24	2.1k
Bryan K. McCune United States	17	1.1k 0.8×	557 0.9×	138 0.5×	142 0.6×	253 1.1×	26	2.0k
Levi J. Beverly United States	28	1.7k 1.3×	601 1.0×	345 1.3×	277 1.2×	403 1.7×	65	2.7k
Masayuki Koizumi Japan	25	1.6k 1.2×	1.1k 1.9×	296 1.1×	183 0.8×	406 1.8×	53	3.0k
Géraldine Siegfried France	23	1.2k 0.9×	633 1.1×	210 0.8×	429 1.8×	469 2.0×	45	2.3k
Nina Jones Canada	30	2.1k 1.6×	440 0.7×	379 1.5×	379 1.6×	320 1.4×	59	3.3k
Natalia S. Pellegata Germany	33	1.5k 1.2×	1.8k 2.9×	255 1.0×	241 1.0×	716 3.1×	94	4.3k
Donna Hylton United States	8	1.2k 0.9×	544 0.9×	347 1.3×	64 0.3×	356 1.5×	12	2.3k
Jeffrey Kiefer United States	24	1.2k 0.9×	588 1.0×	180 0.7×	149 0.6×	513 2.2×	60	2.0k
Yuki Hamano Japan	15	814 0.6×	354 0.6×	396 1.5×	80 0.3×	460 2.0×	27	2.0k
Vesselina G. Cooke United States	11	1.0k 0.8×	434 0.7×	234 0.9×	152 0.6×	431 1.9×	12	2.1k

Countries citing papers authored by David R. Emlet

Since Specialization

Citations

This map shows the geographic impact of David R. Emlet's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David R. Emlet with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David R. Emlet more than expected).

Fields of papers citing papers by David R. Emlet

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David R. Emlet. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David R. Emlet. The network helps show where David R. Emlet may publish in the future.

Co-authorship network of co-authors of David R. Emlet

This figure shows the co-authorship network connecting the top 25 collaborators of David R. Emlet. A scholar is included among the top collaborators of David R. Emlet based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David R. Emlet. David R. Emlet is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Friedman, Peter A., W. Bruce Sneddon, Tatyana Mamonova, et al.. (2022). RGS14 regulates PTH- and FGF23-sensitive NPT2A-mediated renal phosphate uptake via binding to the NHERF1 scaffolding protein. Journal of Biological Chemistry. 298(5). 101836–101836. 14 indexed citations

Al‐bataineh, Mohammad M., Carol L. Kinlough, Zaichuan Mi, et al.. (2021). KIM-1-mediated anti-inflammatory activity is preserved by MUC1 induction in the proximal tubule during ischemia-reperfusion injury. American Journal of Physiology-Renal Physiology. 321(2). F135–F148. 14 indexed citations

Jobbagy, Soma, Darío A. Vitturi, Sonia R. Salvatore, et al.. (2020). Nrf2 activation protects against lithium-induced nephrogenic diabetes insipidus. JCI Insight. 5(1). 12 indexed citations

Wen, Xiao‐Yan, Shengnan Li, Xiukai Chen, et al.. (2020). Time-dependent effects of histone deacetylase inhibition in sepsis-associated acute kidney injury. Intensive Care Medicine Experimental. 8(1). 9–9. 13 indexed citations

Jin, Kui, Carlos L. Manrique‐Caballero, Hui Li, et al.. (2020). Activation of AMP‐activated protein kinase during sepsis/inflammation improves survival by preserving cellular metabolic fitness. The FASEB Journal. 34(5). 7036–7057. 57 indexed citations

Emlet, David R., Andrew Shaw, & John A. Kellum. (2015). Sepsis-Associated AKI: Epithelial Cell Dysfunction. Seminars in Nephrology. 35(1). 85–95. 66 indexed citations

Jin, Kui, David R. Emlet, Núria M. Pastor‐Soler, et al.. (2015). Is acute kidney injury in the early phase of sepsis a sign of metabolic downregulation in tubular epithelial cells?. Critical Care. 19(S1). 3 indexed citations

Emlet, David R., Puja Gupta, Marina Holgado-Madruga, et al.. (2013). Targeting a Glioblastoma Cancer Stem-Cell Population Defined by EGF Receptor Variant III. Cancer Research. 74(4). 1238–1249. 119 indexed citations

Papathanassiu, Adonia E., Nicholas J. MacDonald, David R. Emlet, & Hong Vu. (2010). Antitumor activity of efrapeptins, alone or in combination with 2-deoxyglucose, in breast cancer in vitro and in vivo. Cell Stress and Chaperones. 16(2). 181–193. 19 indexed citations

10.

Smith, Charles A., Agnese A. Pollice, David R. Emlet, & Stanley E. Shackney. (2006). A simple correction for cell autofluorescence for multiparameter cell‐based analysis of human solid tumors. Cytometry Part B Clinical Cytometry. 70B(2). 91–103. 9 indexed citations

11.

Emlet, David R., Russell Schwartz, Kathryn Brown, et al.. (2006). HER2 expression as a potential marker for response to therapy targeted to the EGFR. British Journal of Cancer. 94(8). 1144–1153. 27 indexed citations

12.

Shackney, Stanley E., et al.. (2005). Guidelines for improving the reproducibility of quantitative multiparameter immunofluorescence measurements by laser scanning cytometry on fixed cell suspensions from human solid tumors. Cytometry Part B Clinical Cytometry. 70B(1). 10–19. 2 indexed citations

13.

Okamoto, Isamu, Lawrence C. Kenyon, David R. Emlet, et al.. (2003). Expression of constitutively activated EGFRvlll in non‐small cell lung cancer. Cancer Science. 94(1). 50–56. 107 indexed citations

14.

Tsuiki, Hiromasa, Mehdi Tnani, Isamu Okamoto, et al.. (2003). Constitutively active forms of c-Jun NH2-terminal kinase are expressed in primary glial tumors.. PubMed. 63(1). 250–5. 85 indexed citations

15.

Okamoto, Isamu, Hiromasa Tsuiki, Lawrence C. Kenyon, et al.. (2002). Proteolytic Cleavage of the CD44 Adhesion Molecule in Multiple Human Tumors. American Journal Of Pathology. 160(2). 441–447. 132 indexed citations

16.

Antonyak, Marc A., Lawrence C. Kenyon, Andrew K. Godwin, et al.. (2002). Elevated JNK activation contributes to the pathogenesis of human brain tumors. Oncogene. 21(33). 5038–5046. 90 indexed citations

17.

Moscatello, David K., Marina Holgado-Madruga, David R. Emlet, Bruce Montgomery, & Albert J. Wong. (1998). Constitutive Activation of Phosphatidylinositol 3-Kinase by a Naturally Occurring Mutant Epidermal Growth Factor Receptor. Journal of Biological Chemistry. 273(1). 200–206. 245 indexed citations

18.

Emlet, David R., et al.. (1997). Subsets of Epidermal Growth Factor Receptors during Activation and Endocytosis. Journal of Biological Chemistry. 272(7). 4079–4086. 66 indexed citations

19.

Holgado-Madruga, Marina, et al.. (1997). Grb2-associated binder-1 mediates phosphatidylinositol 3-kinase activation and the promotion of cell survival by nerve growth factor. Proceedings of the National Academy of Sciences. 94(23). 12419–12424. 230 indexed citations

20.

Holgado-Madruga, Marina, David R. Emlet, David K. Moscatello, Andrew K. Godwin, & Albert J. Wong. (1996). A Grb2-associated docking protein in EGF- and insulin-receptor signalling. Nature. 379(6565). 560–564. 578 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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