David R. Derksen
- Molecular Biology
- Cellular and Molecular Neuroscience
- Oncology
- Endocrinology, Diabetes and Metabolism
- Cell Biology
- Co-authors
- David W. LitchfieldNicole St‐DenisGreg VilkPaula M. LoriaDavid P. FairlieDavid J. EdmondsSpiros LirasDavid A. Griffith
- Topics
- Receptor Mechanisms and Signaling (4 papers)Protein Kinase Regulation and GTPase Signaling (3 papers)Diabetes Treatment and Management (3 papers)
- Journals
- Journal of Biological ChemistryMolecular and Cellular BiologyJournal of Medicinal Chemistry
- Partner nations
- United StatesCanadaAustralia
In The Last Decade
David R. Derksen
12 papers receiving 249 citations
Peers
Comparison fields: 5 of 59
- Molecular Biology 168
- Cellular and Molecular Neuroscience 55
- Oncology 44
- Endocrinology, Diabetes and Metabolism 39
- Cell Biology 26
Countries citing papers authored by David R. Derksen
This map shows the geographic impact of David R. Derksen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David R. Derksen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David R. Derksen more than expected).
Fields of papers citing papers by David R. Derksen
This network shows the impact of papers produced by David R. Derksen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David R. Derksen. The network helps show where David R. Derksen may publish in the future.
Co-authorship network of co-authors of David R. Derksen
This figure shows the co-authorship network connecting the top 25 collaborators of David R. Derksen. A scholar is included among the top collaborators of David R. Derksen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David R. Derksen. David R. Derksen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 20 | |
| 2 | 8 | |
| 3 | 5 | |
| 4 | 21 | |
| 5 | 38 | |
| 6 | 10 | |
| 7 | 10 | |
| 8 | 45 | |
| 9 | 19 | |
| 10 | 21 | |
| 11 | 47 | |
| 12 | 11 |
About David R. Derksen
David R. Derksen is a scholar working on Cellular and Molecular Neuroscience, Endocrinology, Diabetes and Metabolism and Biochemistry, having authored 12 papers that have together received 255 indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (4 papers), Protein Kinase Regulation and GTPase Signaling (3 papers) and Diabetes Treatment and Management (3 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (55 citations), Molecular Biology (168 citations) and Endocrinology, Diabetes and Metabolism (39 citations). David R. Derksen has collaborated with scholars based in United States, Canada and Australia. Frequent co-authors include David W. Litchfield, Nicole St‐Denis, Greg Vilk, Paula M. Loria, David P. Fairlie, David J. Edmonds, Spiros Liras, David A. Griffith, James S. Duncan and Stefania Sarno. Their work appears in journals such as Journal of Biological Chemistry, Molecular and Cellular Biology and Journal of Medicinal Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.