David Pim

4.3k citations

40 papers · 3.1k indexed · 1 hit paper · h-index 25

Impact in

Oncology top 2%
- Cancer-related Molecular Pathways
Epidemiology top 2%
- Cervical Cancer and HPV Research
- Hepatitis B Virus Studies

Papers in

Oncology 22
- Cancer-related Molecular Pathways 17
- Viral-associated cancers and disorders 7
Epidemiology 22
- Cervical Cancer and HPV Research 15
- Hepatitis B Virus Studies 9
- Virology and Viral Diseases 4

Co-authors: Lawrence Banks Miranda Thomas Paola Massimi Greg Matlashewski Sylvie Labrecque Ann Kalita Vjekoslav Tomaić Stephen M. Dilworth
Journals: Oncogene (8 papers)Journal of Virology (7 papers)Virology (3 papers)International Journal of Cancer (2 papers)Scientific Reports (1 paper)
Partner nations: Italy United States Poland

In The Last Decade

David Pim

40 papers receiving 3.0k citations

Hit Papers

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Two Polymorphic Variants of Wild-Type p53 Differ Biochemically and Biologically 1999 · 603 citations

Peers

Countries citing papers authored by David Pim

Since Specialization

Citations

This map shows the geographic impact of David Pim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Pim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Pim more than expected).

Fields of papers citing papers by David Pim

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Pim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Pim. The network helps show where David Pim may publish in the future.

Co-authorship network

The 25 scholars most cited alongside David Pim, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with David Pim Line = papers co-authored together David Pim links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

#	Work
1	The VPS4 component of the ESCRT machinery plays an essential role in HPV infectious entry and capsid disassembly Scientific Reports ·Justyna Broniarczyk, David Pim, Paola Massimi, Martina Bergant, Anna Goździcka‐Józefiak, Colin M. Crump, Lawrence Banks	2017	23
2	Interaction of the Human Papillomavirus E6 Oncoprotein with Sorting Nexin 27 Modulates Endocytic Cargo Transport Pathways PLoS Pathogens ·A.W.P, Paola Massimi, Joaquín Manzo-Merino, Vjekoslav Tomaić, David Pim, Martin P. Playford, Marcela Lizano, Sally Roberts, Christian Kranjec, John Doorbar, Lawrence Banks	2016	44
3	Interaction of HPV E6 oncoproteins with specific proteasomal subunits Virology ·Vjekoslav Tomaić, A.W.P, David Pim, Christina Bauer, Christine Blattner, Lawrence Banks	2013	13
4	Human tumour viruses and the deregulation of cell polarity in cancer Nature reviews. Cancer ·Lawrence Banks, David Pim, Miranda Thomas	2012	50
5	HPV‐16 E2 contributes to induction of HPV‐16 late gene expression by inhibiting early polyadenylation The EMBO Journal ·Cecilia Johansson, Camila Araújo Santos Santana, Xiaoze Li, Irma Ester Palma Manríquez, Joanna Fay, Fergus Ryan, David Pim, Lawrence Banks, Stefan Schwartz	2012	63
6	Human papillomaviruses and the specificity of PDZ domain targeting FEBS Journal ·David Pim, Martina Bergant, Siaw Shi Boon, A.W.P, Christian Kranjec, Paola Massimi, (unknown), Miranda Thomas, Vjekoslav Tomaić, Lawrence Banks	2012	72
7	Interaction of viral oncoproteins with cellular target molecules: infection with high‐risk vs low‐risk human papillomaviruses Apmis ·David Pim, Lawrence Banks	2010	115
8	The mechanisms and implications of hScrib regulation of ERK Small GTPases ·Kazunori Nagasaka, Paola Massimi, David Pim, Sudhir Krishna, Christian Kranjec, Shunsuke Nakagawa, Tetsu Yano, Yuji Taketani, Lawrence Banks	2010	9
9	Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2A Oncogene ·David Pim, Paola Massimi, Stephen M. Dilworth, Lawrence Banks	2005	152
10	HPV‐18 E6I modulates HPV‐18 full‐length E6 functions in a cell cycle dependent manner International Journal of Cancer* ·Ernesto Guccione, David Pim, Lawrence Banks	2004	25
11	p53 polymorphic variants at codon 72 exert different effects on cell cycle progression International Journal of Cancer ·David Pim, Lawrence Banks	2003	341
12	Chimaeric HPV E6 proteins allow dissection of the proteolytic pathways regulating different E6 cellular target proteins Oncogene ·David Pim, Miranda Thomas, Lawrence Banks	2002	29
13	HPV E6 and MAGUK protein interactions: determination of the molecular basis for specific protein recognition and degradation Oncogene ·Miranda Thomas, Britt A. Glaunsinger, David Pim, Jin Seok Lee, Lawrence Banks	2001	92
14	HPV E6 targeted degradation of the discs large protein: evidence for the involvement of a novel ubiquitin ligase Oncogene ·David Pim, Miranda Thomas, Jin Seok Lee, Daniela Gardiol, Lawrence Banks	2000	85
15	Allosteric Activation of Acid α-Glucosidase by the Human Papillomavirus E7 Protein Journal of Biological Chemistry ·Werner Zwerschke, Boris Mannhardt, Paola Massimi, Luo Haichao, David Pim, Walter Nickel, Lawrence Banks, Arnold Reuser, Pidder Jansen‐Dürr	2000	43
16	Two Polymorphic Variants of Wild-Type p53 Differ Biochemically and Biologically Molecular and Cellular Biology ·Miranda Thomas, Ann Kalita, Sylvie Labrecque, David Pim, Lawrence Banks, Greg Matlashewski Hit paper breakdown →	1999	603
17	Interaction between the HPV-16 E2 transcriptional activator and p53 Oncogene ·Paola Massimi, David Pim, Cosetta Bertoli, Véronique Bouvard, Lawrence Banks	1999	71
18	The role of the E6-p53 interaction in the molecular pathogenesis of HPV Oncogene ·Miranda Thomas, David Pim, Lawrence Banks	1999	354
19	HPV-18 E6I protein modulates the E6-directed degradation of p53 by binding to full-length HPV-18 E6 Oncogene* ·David Pim, Lawrence Banks	1999	76
20	Alternatively spliced HPV-18 E6* protein inhibits E6 mediated degradation of p53 and suppresses transformed cell growth Oncogene ·David Pim, Paola Massimi, Lawrence Banks	1997	102

About David Pim

David Pim is a scholar working on Oncology, Epidemiology, Cell Biology, Genetics and Biotechnology, having authored 40 papers that have together received 3.1k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (17 papers), Cervical Cancer and HPV Research (15 papers), Virus-based gene therapy research (11 papers), Hepatitis B Virus Studies (9 papers), Viral-associated cancers and disorders (7 papers), Ubiquitin and proteasome pathways (6 papers), Virology and Viral Diseases (4 papers) and Hippo pathway signaling and YAP/TAZ (4 papers). The work is most often cited by research in Oncology (1.5k citations), Epidemiology (1.2k citations), Cancer Research (507 citations), Otorhinolaryngology (119 citations) and Molecular Biology (1.6k citations). David Pim has collaborated with scholars based in Italy, United States and Poland. Frequent co-authors include Lawrence Banks, Miranda Thomas, Paola Massimi, Greg Matlashewski, Sylvie Labrecque, Ann Kalita, Vjekoslav Tomaić, Stephen M. Dilworth, Justyna Broniarczyk and L. Crawford. Their work appears in journals such as Oncogene, Journal of Virology, Virology, International Journal of Cancer and Scientific Reports.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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