D. Pim

4.3k total citations · 2 hit papers
23 papers, 3.8k citations indexed

About

D. Pim is a scholar working on Oncology, Epidemiology and Genetics. According to data from OpenAlex, D. Pim has authored 23 papers receiving a total of 3.8k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Oncology, 11 papers in Epidemiology and 9 papers in Genetics. Recurrent topics in D. Pim's work include Cervical Cancer and HPV Research (10 papers), Cancer-related Molecular Pathways (10 papers) and Virus-based gene therapy research (9 papers). D. Pim is often cited by papers focused on Cervical Cancer and HPV Research (10 papers), Cancer-related Molecular Pathways (10 papers) and Virus-based gene therapy research (9 papers). D. Pim collaborates with scholars based in Italy, United Kingdom and Canada. D. Pim's co-authors include L.V. Crawford, Ed Harlow, Lawrence Banks, L. Crawford, Greg Matlashewski, R.D. Bulbrook, Peter Lamb, Samuel Benchimol, Alan Storey and Simon Tuck and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The EMBO Journal and The Journal of Immunology.

In The Last Decade

D. Pim

23 papers receiving 3.5k citations

Hit Papers

Monoclonal antibodies specific for simian virus 40 tumor ... 1981 2026 1996 2011 1981 1982 250 500 750 1000

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
D. Pim Italy 20 2.2k 1.6k 1.1k 891 550 23 3.8k
L. Crawford United Kingdom 33 2.0k 0.9× 1.8k 1.1× 1.3k 1.1× 2.1k 2.4× 403 0.7× 67 4.6k
Sumitra Deb United States 32 2.3k 1.1× 2.2k 1.3× 441 0.4× 392 0.4× 463 0.8× 79 3.5k
Peter Whyte United States 23 3.2k 1.5× 3.1k 1.9× 2.4k 2.1× 508 0.6× 323 0.6× 43 5.2k
Karen Buchkovich United States 11 2.2k 1.0× 1.9k 1.1× 1.2k 1.0× 316 0.4× 203 0.4× 14 3.5k
E Harlow United States 24 4.7k 2.1× 4.6k 2.8× 1.4k 1.2× 1.1k 1.2× 393 0.7× 29 7.1k
G W Demers United States 23 1.7k 0.8× 1.8k 1.1× 700 0.6× 972 1.1× 307 0.6× 29 3.6k
Robert Ralston United States 37 655 0.3× 1.8k 1.1× 823 0.7× 1.9k 2.1× 214 0.4× 78 4.8k
Kah-Whye Peng United States 43 2.2k 1.0× 2.1k 1.3× 4.6k 4.0× 1.7k 2.0× 548 1.0× 102 6.2k
Carole Yee United States 29 1.3k 0.6× 1.3k 0.8× 962 0.8× 1.9k 2.1× 119 0.2× 59 4.1k
Susan M. Astrin United States 27 888 0.4× 2.4k 1.5× 1.3k 1.1× 897 1.0× 69 0.1× 51 4.5k

Countries citing papers authored by D. Pim

Since Specialization
Citations

This map shows the geographic impact of D. Pim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by D. Pim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites D. Pim more than expected).

Fields of papers citing papers by D. Pim

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by D. Pim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by D. Pim. The network helps show where D. Pim may publish in the future.

Co-authorship network of co-authors of D. Pim

This figure shows the co-authorship network connecting the top 25 collaborators of D. Pim. A scholar is included among the top collaborators of D. Pim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with D. Pim. D. Pim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Nagasaka, Kazunori, D. Pim, Paola Massimi, et al.. (2010). The cell polarity regulator hScrib controls ERK activation through a KIM site-dependent interaction. Oncogene. 29(38). 5311–5321. 62 indexed citations
2.
Thomas, Miranda, Nisha Narayan, D. Pim, et al.. (2008). Human papillomaviruses, cervical cancer and cell polarity. Oncogene. 27(55). 7018–7030. 141 indexed citations
3.
Massimi, Paola, D. Pim, & Lawrence Banks. (1997). Human papillomavirus type 16 E7 binds to the conserved carboxy-terminal region of the TATA box binding protein and this contributes to E7 transforming activity.. Journal of General Virology. 78(10). 2607–2613. 73 indexed citations
4.
Thomas, Miranda, et al.. (1996). Induction of apoptosis by p53 is independent of its oligomeric state and can be abolished by HPV-18 E6 through ubiquitin mediated degradation.. PubMed. 13(2). 265–73. 58 indexed citations
5.
Gu, Zhengming, D. Pim, Sylvie Labrecque, Lawrence Banks, & Greg Matlashewski. (1994). DNA damage induced p53 mediated transcription is inhibited by human papillomavirus type 18 E6.. PubMed. 9(2). 629–33. 38 indexed citations
6.
Bouvard, Véronique, Alan Storey, D. Pim, & Lawrence Banks. (1994). Characterization of the human papillomavirus E2 protein: evidence of trans-activation and trans-repression in cervical keratinocytes.. The EMBO Journal. 13(22). 5451–5459. 139 indexed citations
7.
Pim, D., Mary Collins, & Lawrence Banks. (1992). Human papillomavirus type 16 E5 gene stimulates the transforming activity of the epidermal growth factor receptor.. PubMed. 7(1). 27–32. 190 indexed citations
8.
Pim, D. & Lawrence Banks. (1991). Loss of HPV-16 E7 dependence in cells transformed by HPV-16 E7 plus EJ-ras correlates with increased c-myc expression.. PubMed. 6(4). 589–94. 4 indexed citations
9.
Banks, Lawrence, Flora Moreau, Karen H. Vousden, D. Pim, & Greg Matlashewski. (1991). Expression of the human papillomavirus E7 oncogene during cell transformation is sufficient to induce susceptibility to lysis by activated macrophages. The Journal of Immunology. 146(6). 2037–2042. 16 indexed citations
10.
Storey, Alan, D. Pim, Anne M. Murray, et al.. (1988). Comparison of the in vitro transforming activities of human papillomavirus types.. The EMBO Journal. 7(6). 1815–1820. 273 indexed citations
11.
Matlashewski, Greg, Simon Tuck, D. Pim, et al.. (1987). Primary Structure Polymorphism at Amino Acid Residue 72 of Human p53. Molecular and Cellular Biology. 7(2). 961–963. 56 indexed citations
12.
Banks, Lawrence, et al.. (1987). Expression of Human Papillomavirus Type 6 and Type 16 Capsid Proteins in Bacteria and Their Antigenic Characterization. Journal of General Virology. 68(12). 3081–3089. 27 indexed citations
13.
Matlashewski, Greg, Simon Tuck, D. Pim, et al.. (1987). Primary structure polymorphism at amino acid residue 72 of human p53.. Molecular and Cellular Biology. 7(2). 961–963. 406 indexed citations
14.
Matlashewski, Greg, Peter Lamb, D. Pim, et al.. (1984). Isolation and characterization of a human p53 cDNA clone: expression of the human p53 gene.. The EMBO Journal. 3(13). 3257–3262. 225 indexed citations
15.
Crawford, L.V., D. Pim, & Patricia W. Lamb. (1984). The cellular protein p53 in human tumours.. PubMed. 2(4). 261–72. 88 indexed citations
16.
Benchimol, Samuel, D. Pim, & L. Crawford. (1982). Radioimmunoassay of the cellular protein p53 in mouse and human cell lines.. The EMBO Journal. 1(9). 1055–1062. 148 indexed citations
17.
Harlow, Ed, et al.. (1981). Monoclonal antibodies specific for simian virus 40 tumor antigens. Journal of Virology. 39(3). 861–869. 1064 indexed citations breakdown →
18.
Crawford, L.V., D. Pim, E G Gurney, P.N. Goodfellow, & Joyce Taylor‐Papadimitriou. (1981). Detection of a common feature in several human tumor cell lines--a 53,000-dalton protein.. Proceedings of the National Academy of Sciences. 78(1). 41–45. 196 indexed citations
19.
Crawford, L.V., et al.. (1980). Characterization of the Complex between SV40 Large T Antigen and the 53K Host Protein in Transformed Mouse Cells. Cold Spring Harbor Symposia on Quantitative Biology. 44(0). 179–187. 28 indexed citations
20.
Crawford, L.V., D. Pim, & David P. Lane. (1980). An immunochemical investigation of SV40 T-antigens 2. Quantitation of antigens and antibody activities. Virology. 100(2). 314–325. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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