Mark E. Ewen

12.2k total citations · 6 hit papers
54 papers, 10.4k citations indexed

About

Mark E. Ewen is a scholar working on Oncology, Molecular Biology and Genetics. According to data from OpenAlex, Mark E. Ewen has authored 54 papers receiving a total of 10.4k indexed citations (citations by other indexed papers that have themselves been cited), including 41 papers in Oncology, 35 papers in Molecular Biology and 17 papers in Genetics. Recurrent topics in Mark E. Ewen's work include Cancer-related Molecular Pathways (38 papers), Virus-based gene therapy research (12 papers) and Ocular Oncology and Treatments (8 papers). Mark E. Ewen is often cited by papers focused on Cancer-related Molecular Pathways (38 papers), Virus-based gene therapy research (12 papers) and Ocular Oncology and Treatments (8 papers). Mark E. Ewen collaborates with scholars based in United States, Japan and Australia. Mark E. Ewen's co-authors include David M. Livingston, Jun‐ya Kato, C J Sherr, James A. DeCaprio, Hayla K. Sluss, Hitoshi Matsushime, Jeanne B. Lawrence, H Matsushime, Scott W. Hiebert and Richard Eckner and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Mark E. Ewen

54 papers receiving 10.2k citations

Hit Papers

Direct binding of cyclin D to the retinoblastoma gene pro... 1992 2026 2003 2014 1993 1994 1993 1992 1992 250 500 750 1000

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mark E. Ewen United States 38 7.1k 6.7k 1.9k 1.4k 1.1k 54 10.4k
Phang‐Lang Chen United States 53 9.2k 1.3× 4.5k 0.7× 3.0k 1.6× 1.6k 1.2× 769 0.7× 100 11.9k
Douglas C. Dean United States 54 8.4k 1.2× 4.6k 0.7× 1.2k 0.6× 1.3k 1.0× 1.4k 1.2× 107 11.9k
Philip W. Hinds United States 47 7.2k 1.0× 7.2k 1.1× 1.4k 0.8× 1.1k 0.8× 803 0.7× 107 11.8k
Scott W. Hiebert United States 70 14.1k 2.0× 5.4k 0.8× 2.2k 1.2× 1.5k 1.1× 2.5k 2.2× 176 19.1k
E Harlow United States 24 4.6k 0.7× 4.7k 0.7× 1.4k 0.7× 802 0.6× 599 0.5× 29 7.1k
Gordon Peters United Kingdom 48 7.2k 1.0× 4.8k 0.7× 1.1k 0.6× 944 0.7× 912 0.8× 95 10.4k
Ali Fattaey United States 22 4.6k 0.7× 4.0k 0.6× 2.3k 1.2× 689 0.5× 439 0.4× 34 6.6k
Richard A. Ashmun United States 30 5.9k 0.8× 4.5k 0.7× 811 0.4× 1.0k 0.7× 1.0k 0.9× 53 8.4k
James M. Roberts United States 40 7.2k 1.0× 4.8k 0.7× 1.1k 0.6× 2.2k 1.6× 557 0.5× 52 9.6k
Hein te Riele Netherlands 44 6.0k 0.8× 2.3k 0.3× 1.5k 0.8× 550 0.4× 566 0.5× 108 8.2k

Countries citing papers authored by Mark E. Ewen

Since Specialization
Citations

This map shows the geographic impact of Mark E. Ewen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark E. Ewen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark E. Ewen more than expected).

Fields of papers citing papers by Mark E. Ewen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark E. Ewen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark E. Ewen. The network helps show where Mark E. Ewen may publish in the future.

Co-authorship network of co-authors of Mark E. Ewen

This figure shows the co-authorship network connecting the top 25 collaborators of Mark E. Ewen. A scholar is included among the top collaborators of Mark E. Ewen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark E. Ewen. Mark E. Ewen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Liu, Qiang, et al.. (2014). Cyclin D1 and C/EBPβ LAP1 Operate in a Common Pathway To Promote Mammary Epithelial Cell Differentiation. Molecular and Cellular Biology. 34(16). 3168–3179. 13 indexed citations
2.
Chen, Jingqi, Yandan Yao, Fengyan Yu, et al.. (2011). CCL18 from Tumor-Associated Macrophages Promotes Breast Cancer Metastasis via PITPNM3. Cancer Cell. 19(6). 814–816. 18 indexed citations
3.
Takahashi, Chiaki, Bernardo Contreras, Tsuyoshi Iwanaga, et al.. (2005). Nras loss induces metastatic conversion of Rb1-deficient neuroendocrine thyroid tumor. Nature Genetics. 38(1). 118–123. 38 indexed citations
4.
Lamb, Justin & Mark E. Ewen. (2003). Cyclin D1 and Molecular Chaperones: Implications for Tumorigenesis. Cell Cycle. 2(6). 525–527. 26 indexed citations
5.
Lamb, Justin, Sridhar Ramaswamy, Heide L. Ford, et al.. (2003). A Mechanism of Cyclin D1 Action Encoded in the Patterns of Gene Expression in Human Cancer. Cell. 114(3). 323–334. 331 indexed citations
6.
Takahashi, Chiaki, Roderick T. Bronson, Merav Socolovsky, et al.. (2003). Rb and N- ras Function Together To Control Differentiation in the Mouse. Molecular and Cellular Biology. 23(15). 5256–5268. 40 indexed citations
7.
Lamb, Justin, et al.. (2000). Regulation of the Functional Interaction between Cyclin D1 and the Estrogen Receptor. Molecular and Cellular Biology. 20(23). 8667–8675. 73 indexed citations
8.
Ewen, Mark E.. (2000). Where the cell cycle and histones meet: Figure 1.. Genes & Development. 14(18). 2265–2270. 90 indexed citations
9.
McMahon, Christine M., Tuangporn Suthiphongchai, James DiRenzo, & Mark E. Ewen. (1999). P/CAF associates with cyclin D1 and potentiates its activation of the estrogen receptor. Proceedings of the National Academy of Sciences. 96(10). 5382–5387. 147 indexed citations
10.
Shapiro, Geoffrey I., et al.. (1998). p16 INK4A Participates in a G 1 Arrest Checkpoint in Response to DNA Damage. Molecular and Cellular Biology. 18(1). 378–387. 140 indexed citations
11.
Lee, Kwang Y., et al.. (1998). Regulation of Exit from Quiescence by p27 and Cyclin D1-CDK4. Molecular and Cellular Biology. 18(11). 6605–6615. 85 indexed citations
12.
Reed, Michael F., et al.. (1998). Enforced CDK4 expression in a hematopoietic cell line confers resistance to the G1 arrest induced by ionizing radiation. Oncogene. 17(23). 2961–2971. 20 indexed citations
13.
Peeper, Daniel S., Elizabeth Neuman, Juan Zalvide, et al.. (1997). Ras signalling linked to the cell-cycle machinery by the retinoblastoma protein. Nature. 386(6621). 177–181. 315 indexed citations
14.
Ewen, Mark E.. (1996). p53-Dependent repression of cdk4 synthesis in transforming growth factor-β-induced G1 cell cycle arrest. Journal of Laboratory and Clinical Medicine. 128(4). 355–360. 21 indexed citations
15.
Ewen, Mark E. & S.J. Miller. (1996). p53 and translational control. Biochimica et Biophysica Acta (BBA) - Reviews on Cancer. 1242(3). 181–184. 47 indexed citations
16.
Ewen, Mark E.. (1994). The cell cycle and the retinoblastoma protein family. Cancer and Metastasis Reviews. 13(1). 45–66. 251 indexed citations
17.
Kato, Jun‐ya, H Matsushime, Scott W. Hiebert, Mark E. Ewen, & C J Sherr. (1993). Direct binding of cyclin D to the retinoblastoma gene product (pRb) and pRb phosphorylation by the cyclin D-dependent kinase CDK4. Genes & Development. 7(3). 331–342. 1065 indexed citations breakdown →
18.
Ewen, Mark E., et al.. (1993). TGFβ inhibition of Cdk4 synthesis is linked to cell cycle arrest. Cell. 74(6). 1009–1020. 467 indexed citations
19.
Ewen, Mark E., et al.. (1993). Functional interactions of the retinoblastoma protein with mammalian D-type cyclins. Cell. 73(3). 487–497. 894 indexed citations breakdown →
20.
Ewen, Mark E. & Helen R. Revel. (1988). In vitro replication and transcription of the segmented double-stranded RNA bacteriophage φ6. Virology. 165(2). 489–498. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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