David M. Lee

2.5k total citations · 1 hit paper
8 papers, 1.2k citations indexed

About

David M. Lee is a scholar working on Molecular Biology, Cancer Research and Genetics. According to data from OpenAlex, David M. Lee has authored 8 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 5 papers in Cancer Research and 1 paper in Genetics. Recurrent topics in David M. Lee's work include RNA modifications and cancer (5 papers), Cancer-related molecular mechanisms research (5 papers) and RNA Research and Splicing (5 papers). David M. Lee is often cited by papers focused on RNA modifications and cancer (5 papers), Cancer-related molecular mechanisms research (5 papers) and RNA Research and Splicing (5 papers). David M. Lee collaborates with scholars based in United States and Switzerland. David M. Lee's co-authors include Dhavalkumar D. Patel, Daniel I. Schenkman, Barton F. Haynes, Perry J. Blackshear, Gary S. Gilkeson, Gregory A. Taylor, Wi S. Lai, Michael J. Thompson, Hal E. Broxmeyer and Ester Carballo and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Nature Genetics.

In The Last Decade

David M. Lee

8 papers receiving 1.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A Pathogenetic Role for TNFα in the Syndrome of Cachexia, Arthritis, and Autoimmunity Resulting from Tristetraprolin (TTP) Deficiency

1996 666 citations Gregory A. Taylor, Ester Carballo et al. Immunity profile →

Peers

Countries citing papers authored by David M. Lee

Since Specialization

Citations

This map shows the geographic impact of David M. Lee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David M. Lee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David M. Lee more than expected).

Fields of papers citing papers by David M. Lee

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David M. Lee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David M. Lee. The network helps show where David M. Lee may publish in the future.

Co-authorship network of co-authors of David M. Lee

This figure shows the co-authorship network connecting the top 25 collaborators of David M. Lee. A scholar is included among the top collaborators of David M. Lee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David M. Lee. David M. Lee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

#	Work	Indexed citations
1	Elements at the 5′ end of Xist harbor SPEN-independent transcriptional antiterminator activity 2020 ·Nucleic Acids Research ·(unknown), David M. Lee, Rachel E. Cherney, Susan O. Kim, Kaoru Inoue, Megan D. Schertzer, Steve Bischoff, Dale O. Cowley, J. Mauro Calabrese	11
2	SHAPE reveals transcript-wide interactions, complex structural domains, and protein interactions across the Xist lncRNA in living cells 2020 ·UNC Libraries ·David M. Lee, Matthew Friedersdorf, Jack D. Keene, (unknown), Cindo O. Nicholson, Matthew J. Smola, (unknown), Kevin M. Weeks, Kaoru Inoue	6
3	A piggyBac-based toolkit for inducible genome editing in mammalian cells 2019 ·RNA ·Megan D. Schertzer, (unknown), (unknown), David M. Lee, (unknown), Ryan M. Murphy, Susan O. Kim, (unknown), J. Mauro Calabrese	28
4	lncRNA-Induced Spread of Polycomb Controlled by Genome Architecture, RNA Abundance, and CpG Island DNA 2019 ·Molecular Cell ·Megan D. Schertzer, (unknown), Joshua Starmer, Rachel E. Cherney, David M. Lee, Gabriela Salazar, (unknown), Steve Bischoff, Dale O. Cowley, Pablo Ariel, Mark J. Zylka, Jill M. Dowen, Terry Magnuson, J. Mauro Calabrese	98
5	Functional classification of long non-coding RNAs by k-mer content 2018 ·Nature Genetics ·(unknown), Susan O. Kim, Kaoru Inoue, Matthew J. Smola, David M. Lee, Megan D. Schertzer, Joshua S. Wooten, Allison R. Baker, Daniel J. Sprague, David W. Collins, (unknown), Shuo Wang, Qidi Chen, Kevin M. Weeks, Peter J. Mucha, J. Mauro Calabrese	167
6	SHAPE reveals transcript-wide interactions, complex structural domains, and protein interactions across the Xist lncRNA in living cells 2016 ·Proceedings of the National Academy of Sciences ·Matthew J. Smola, (unknown), Kaoru Inoue, Cindo O. Nicholson, Matthew Friedersdorf, Jack D. Keene, David M. Lee, J. Mauro Calabrese, Kevin M. Weeks	174
7	Granulocyte chemotaxis and disease expression are differentially regulated by GRK subtype in an acute inflammatory arthritis model (K/BxN) 2008 ·Clinical Immunology ·Teresa K. Tarrant, Rishi R. Rampersad, Denise Esserman, (unknown), Peng Liu, Richard T. Premont, Robert J. Lefkowitz, David M. Lee, Dhavalkumar D. Patel	32
8	A Pathogenetic Role for TNFα in the Syndrome of Cachexia, Arthritis, and Autoimmunity Resulting from Tristetraprolin (TTP) Deficiency breakdown → 1996 ·Immunity ·Gregory A. Taylor, Ester Carballo, David M. Lee, Wi S. Lai, Michael J. Thompson, Dhavalkumar D. Patel, Daniel I. Schenkman, Gary S. Gilkeson, Hal E. Broxmeyer, Barton F. Haynes, Perry J. Blackshear	666

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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