David M. Harris

3.5k total citations · 1 hit paper
64 papers, 2.6k citations indexed

About

David M. Harris is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, David M. Harris has authored 64 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Cardiology and Cardiovascular Medicine, 24 papers in Molecular Biology and 13 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in David M. Harris's work include Cardiac electrophysiology and arrhythmias (12 papers), Ion channel regulation and function (8 papers) and Receptor Mechanisms and Signaling (8 papers). David M. Harris is often cited by papers focused on Cardiac electrophysiology and arrhythmias (12 papers), Ion channel regulation and function (8 papers) and Receptor Mechanisms and Signaling (8 papers). David M. Harris collaborates with scholars based in United States, United Kingdom and Australia. David M. Harris's co-authors include Andrea D. Eckhart, Steven R. Houser, Karl H. Siedentop, Xiongwen Chen, Remus M. Berretta, Walter J. Koch, Risto Kerkelä, Thomas Force, Kathleen C. Woulfe and Jey-Hsin Chen and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Lancet and Circulation.

In The Last Decade

David M. Harris

60 papers receiving 2.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Cardiotoxicity associated with tyrosine kinase inhibitor sunitinib

2007 814 citations David M. Harris et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David M. Harris United States	24	1.2k	1.2k	429	428	308	64	2.6k
José Vilar France	38	2.1k 1.8×	1.0k 0.9×	342 0.8×	334 0.8×	182 0.6×	89	4.3k
Jens W. Fischer Germany	38	1.7k 1.5×	957 0.8×	394 0.9×	502 1.2×	114 0.4×	139	4.5k
Masahito Tamura Japan	27	3.0k 2.5×	625 0.5×	457 1.1×	678 1.6×	172 0.6×	121	4.9k
Ralph V. Shohet United States	38	1.9k 1.6×	806 0.7×	256 0.6×	285 0.7×	110 0.4×	93	4.7k
Satoru Yamazaki Japan	28	1.1k 1.0×	575 0.5×	230 0.5×	382 0.9×	208 0.7×	97	2.6k
Lin Zhang China	35	2.2k 1.9×	1.7k 1.4×	384 0.9×	203 0.5×	285 0.9×	140	3.7k
Thierry Couffinhal France	34	2.4k 2.0×	766 0.6×	392 0.9×	355 0.8×	194 0.6×	139	4.7k
Martin Moser Germany	37	1.9k 1.6×	931 0.8×	429 1.0×	168 0.4×	129 0.4×	121	3.9k
David E. Gutstein United States	32	1.9k 1.6×	1.5k 1.2×	183 0.4×	129 0.3×	208 0.7×	80	3.3k
W. Matthijs Blankesteijn Netherlands	32	2.2k 1.9×	1.6k 1.4×	225 0.5×	487 1.1×	222 0.7×	80	4.0k

Countries citing papers authored by David M. Harris

Since Specialization

Citations

This map shows the geographic impact of David M. Harris's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David M. Harris with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David M. Harris more than expected).

Fields of papers citing papers by David M. Harris

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David M. Harris. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David M. Harris. The network helps show where David M. Harris may publish in the future.

Co-authorship network of co-authors of David M. Harris

This figure shows the co-authorship network connecting the top 25 collaborators of David M. Harris. A scholar is included among the top collaborators of David M. Harris based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David M. Harris. David M. Harris is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Harris, David M., et al.. (2023). A comparison of virtual and in person delivery of a full meal replacement program for obesity. Obesity Science & Practice. 10(1). e718–e718. 1 indexed citations

Harris, David M., et al.. (2022). Ventricular Tachycardia or Fibrillation Storm in Coronavirus Disease. SHILAP Revista de lepidopterología. 2022. 1–9. 2 indexed citations

Robbins, Nathan, et al.. (2017). Feasibility and safety of exercise stress testing using an anti-gravity treadmill with Tc-99m tetrofosmin single-photon emission computed tomography (SPECT) myocardial perfusion imaging: A pilot non-randomized controlled study. Journal of Nuclear Cardiology. 25(4). 1092–1097. 1 indexed citations

Kim, Joon-Seok, et al.. (2016). Meta-Analysis of Prognostic Implications of Exercise-Induced Ventricular Premature Complexes in the General Population. The American Journal of Cardiology. 118(5). 725–732. 15 indexed citations

Harris, David M., Timothy C. Wong, & Prem Soman. (2015). Direct visualization of regional cardiac sympathetic dysfunction in stress-induced cardiomyopathy. Journal of Nuclear Cardiology. 22(6). 1317–1319. 5 indexed citations

Harris, David M., et al.. (2014). Calcific embolization with infective endocarditis involving the posterior mitral leaflet in a patient with underlying hypertrophic obstructive cardiomyopathy. Journal of Thrombosis and Thrombolysis. 39(2). 241–244.

Chen, Xiongwen, Hiroyuki Nakayama, Xiaoying Zhang, et al.. (2010). Calcium influx through Cav1.2 is a proximal signal for pathological cardiomyocyte hypertrophy. Journal of Molecular and Cellular Cardiology. 50(3). 460–470. 84 indexed citations

Seidl, Armin H., Edwin W. Rubel, & David M. Harris. (2010). Mechanisms for Adjusting Interaural Time Differences to Achieve Binaural Coincidence Detection. Journal of Neuroscience. 30(1). 70–80. 113 indexed citations

Koenig, Judith, et al.. (2009). Evaluation of endotoxin activity in blood measured via neutrophil chemiluminescence in healthy horses and horses with colic. American Journal of Veterinary Research. 70(10). 1183–1186. 8 indexed citations

10.

Martini, Jeffrey S., Philip Raake, Leif Erik Vinge, et al.. (2008). Uncovering G protein-coupled receptor kinase-5 as a histone deacetylase kinase in the nucleus of cardiomyocytes. Proceedings of the National Academy of Sciences. 105(34). 12457–12462. 177 indexed citations

11.

Chen, Xiongwen, Xiaoying Zhang, David M. Harris, et al.. (2008). Reduced effects of BAY K 8644 on L-type Ca²⁺ current in failing human cardiac myocytes are related to abnormal adrenergic regulation. American Journal of Physiology-Heart and Circulatory Physiology. 294(5). H2257–H2267. 22 indexed citations

12.

Cohn, Heather I., et al.. (2008). Inhibition of vascular smooth muscle G protein-coupled receptor kinase 2 enhances α_1D-adrenergic receptor constriction. American Journal of Physiology-Heart and Circulatory Physiology. 295(4). H1695–H1704. 38 indexed citations

13.

Harris, David M., Xiongwen Chen, Stéphanie Pesant, et al.. (2008). Inhibition of angiotensin II Gq signaling augments β-adrenergic receptor mediated effects in a renal artery stenosis model of high blood pressure. Journal of Molecular and Cellular Cardiology. 46(1). 100–107. 6 indexed citations

14.

MacDonnell, Scott M., Hajime Kubo, David M. Harris, et al.. (2007). Calcineurin inhibition normalizes β-adrenergic responsiveness in the spontaneously hypertensive rat. American Journal of Physiology-Heart and Circulatory Physiology. 293(5). H3122–H3129. 17 indexed citations

15.

Harris, David M., Heather I. Cohn, Stéphanie Pesant, Ruihai Zhou, & Andrea D. Eckhart. (2007). Vascular smooth muscle G_qsignaling is involved in high blood pressure in both induced renal and genetic vascular smooth muscle-derived models of hypertension. American Journal of Physiology-Heart and Circulatory Physiology. 293(5). H3072–H3079. 22 indexed citations

16.

Mills, Geoffrey, David M. Harris, Xiongwen Chen, & Steven R. Houser. (2006). Intracellular sodium determines frequency-dependent alterations in contractility in hypertrophied feline ventricular myocytes. American Journal of Physiology-Heart and Circulatory Physiology. 292(2). H1129–H1138. 14 indexed citations

17.

Harris, David M., et al.. (2004). Therapeutic ratio quantifies laser antisepsis: Ablation of Porphyromonas gingivalis with dental lasers. Lasers in Surgery and Medicine. 35(3). 206–213. 44 indexed citations

18.

Wenig, Barry L., Leonard I. Grossweiner, Mahmood F. Mafee, et al.. (1990). Photodynamic Therapy in the Treatment of Squamous Cell Carcinoma of the Head and Neck. Archives of Otolaryngology - Head and Neck Surgery. 116(11). 1267–1270. 92 indexed citations

19.

Siedentop, Karl H., et al.. (1988). Autologous fibrin tissue adhesive: Factors influencing bonding power. The Laryngoscope. 98(7). 731–733. 46 indexed citations

20.

Harris, David M., et al.. (1987). Autologous fibrin tissue adhesive biodegration and systemic effects. The Laryngoscope. 97(10). 1141–1144. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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