Martin Moser

4.9k total citations
121 papers, 3.9k citations indexed

About

Martin Moser is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Surgery. According to data from OpenAlex, Martin Moser has authored 121 papers receiving a total of 3.9k indexed citations (citations by other indexed papers that have themselves been cited), including 56 papers in Molecular Biology, 45 papers in Cardiology and Cardiovascular Medicine and 26 papers in Surgery. Recurrent topics in Martin Moser's work include Antiplatelet Therapy and Cardiovascular Diseases (25 papers), Atrial Fibrillation Management and Outcomes (17 papers) and Acute Myocardial Infarction Research (17 papers). Martin Moser is often cited by papers focused on Antiplatelet Therapy and Cardiovascular Diseases (25 papers), Atrial Fibrillation Management and Outcomes (17 papers) and Acute Myocardial Infarction Research (17 papers). Martin Moser collaborates with scholars based in Germany, United States and Switzerland. Martin Moser's co-authors include Christoph Bode, Cam Patterson, Thomas Helbing, Christoph Bode, Sebastian Grundmann, Philipp Diehl, Johannes Ruef, Karlheinz Peter, Yaxu Wu and Jennifer Heinke and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and The Journal of Cell Biology.

In The Last Decade

Martin Moser

117 papers receiving 3.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Martin Moser Germany 37 1.9k 931 680 653 429 121 3.9k
Ivo Buschmann Germany 33 2.4k 1.3× 650 0.7× 473 0.7× 1.1k 1.7× 399 0.9× 108 4.5k
Asish K. Ghosh United States 30 1.8k 1.0× 649 0.7× 623 0.9× 430 0.7× 516 1.2× 46 3.7k
Jens W. Fischer Germany 38 1.7k 0.9× 957 1.0× 714 1.1× 608 0.9× 394 0.9× 139 4.5k
Hideaki Oda Japan 33 2.1k 1.1× 352 0.4× 536 0.8× 481 0.7× 528 1.2× 105 4.2k
Michael T. Chin United States 36 2.6k 1.4× 711 0.8× 781 1.1× 397 0.6× 292 0.7× 100 4.7k
Donald B. Bloch United States 42 2.3k 1.2× 637 0.7× 369 0.5× 876 1.3× 561 1.3× 120 5.8k
Kenshi Hayashi Japan 36 2.1k 1.1× 1.8k 1.9× 649 1.0× 939 1.4× 558 1.3× 244 5.4k
Masahito Tamura Japan 27 3.0k 1.6× 625 0.7× 547 0.8× 370 0.6× 457 1.1× 121 4.9k
Anna M. Randi United Kingdom 40 2.2k 1.2× 432 0.5× 557 0.8× 668 1.0× 517 1.2× 100 4.9k
Ralph V. Shohet United States 38 1.9k 1.0× 806 0.9× 507 0.7× 416 0.6× 256 0.6× 93 4.7k

Countries citing papers authored by Martin Moser

Since Specialization
Citations

This map shows the geographic impact of Martin Moser's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Moser with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Moser more than expected).

Fields of papers citing papers by Martin Moser

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martin Moser. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Moser. The network helps show where Martin Moser may publish in the future.

Co-authorship network of co-authors of Martin Moser

This figure shows the co-authorship network connecting the top 25 collaborators of Martin Moser. A scholar is included among the top collaborators of Martin Moser based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin Moser. Martin Moser is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Pankratz, Franziska, Gerhard Sengle, Christian Smolka, et al.. (2023). BMPER Improves Vascular Remodeling and the Contractile Vascular SMC Phenotype. International Journal of Molecular Sciences. 24(5). 4950–4950. 4 indexed citations
2.
Ruile, Philipp, et al.. (2023). Bilateral deafness, diabetes, and different types of cardiomyopathy in family members with m.3243A > g mutation: a case report. European Heart Journal - Case Reports. 7(2). ytad073–ytad073. 3 indexed citations
3.
Diehl, Philipp, et al.. (2022). Time from blood draw to multiple electrode aggregometry and association with platelet reactivity. Journal of Thrombosis and Thrombolysis. 55(1). 134–140. 6 indexed citations
4.
Smolka, Christian, Christoph Koentges, Oliver Gorka, et al.. (2021). Cardiomyocyte‐specific miR‐100 overexpression preserves heart function under pressure overload in mice and diminishes fatty acid uptake as well as ROS production by direct suppression of Nox4 and CD36. The FASEB Journal. 35(11). e21956–e21956. 12 indexed citations
5.
Smolka, Christian, Xavier Bemtgen, Laura Schneider, et al.. (2021). MiR-100 overexpression attenuates high fat diet induced weight gain, liver steatosis, hypertriglyceridemia and development of metabolic syndrome in mice. Molecular Medicine. 27(1). 101–101. 19 indexed citations
6.
Olivier, Christoph B., Nathaly Anto-Michel, Ingo Hilgendorf, et al.. (2021). Platelet Bone Morphogenetic Protein-4 Mediates Vascular Inflammation and Neointima Formation after Arterial Injury. Cells. 10(8). 2027–2027. 14 indexed citations
8.
Olivier, Christoph B., Susanne Weber, Philipp Diehl, et al.. (2016). Dabigatran and rivaroxaban do not affect AA- and ADP-induced platelet aggregation in patients receiving concomitant platelet inhibitors. Journal of Thrombosis and Thrombolysis. 42(2). 161–166. 21 indexed citations
9.
Zhou, Qian, Franziska Pankratz, Christoph B. Olivier, et al.. (2016). MnTBAP increases BMPR-II expression in endothelial cells and attenuates vascular inflammation. Vascular Pharmacology. 84. 67–73. 9 indexed citations
10.
Rilinger, Jonathan, Jennifer S. Esser, Qian Zhou, et al.. (2016). High platelet reactivity after P2Y12-inhibition in patients with atrial fibrillation and coronary stenting. Journal of Thrombosis and Thrombolysis. 42(4). 558–565. 4 indexed citations
11.
Olivier, Christoph B., Susanne Weber, Qian Zhou, et al.. (2016). Platelet reactivity after administration of third generation P2Y12-antagonists does not depend on body weight in contrast to clopidogrel. Journal of Thrombosis and Thrombolysis. 42(1). 84–89. 4 indexed citations
12.
Esser, Jennifer S., Anita L. Allen, Achim Lother, et al.. (2016). The neuronal transcription factor NPAS4 is a strong inducer of sprouting angiogenesis and tip cell formation. Cardiovascular Research. 113(2). 222–223. 14 indexed citations
13.
Olivier, Christoph B., Manfred Olschewski, Qian Zhou, et al.. (2014). A high ratio of ADP–TRAP induced platelet aggregation is associated more strongly with increased mortality after coronary stent implantation than high conventional ADP induced aggregation alone. Clinical Research in Cardiology. 103(12). 968–975. 7 indexed citations
14.
Pi, Xinchun, Pamela Lockyer, Laura A. Dyer, et al.. (2012). Bmper Inhibits Endothelial Expression of Inflammatory Adhesion Molecules and Protects Against Atherosclerosis. Arteriosclerosis Thrombosis and Vascular Biology. 32(9). 2214–2222. 36 indexed citations
15.
Willecke, Florian, Volker Auwärter, Stefan Kneisel, et al.. (2011). Cannabinoid Receptor 2 Signaling Does Not Modulate Atherogenesis in Mice. PLoS ONE. 6(4). e19405–e19405. 19 indexed citations
16.
Moser, Martin, et al.. (2010). MicroRNA Regulation of Angiogenesis and Arteriogenesis. Trends in Cardiovascular Medicine. 20(8). 253–262. 19 indexed citations
17.
Kelley, Rusty, Rongqin Ren, Xinchun Pi, et al.. (2009). A concentration-dependent endocytic trap and sink mechanism converts Bmper from an activator to an inhibitor of Bmp signaling. The Journal of Cell Biology. 184(4). 597–609. 105 indexed citations
18.
Grundmann, Sebastian, Stephan H. Schirmer, Liesbeth H.P. Hekking, et al.. (2009). Endothelial glycocalyx dimensions are reduced in growing collateral arteries and modulate leucocyte adhesion in arteriogenesis. Journal of Cellular and Molecular Medicine. 13(9b). 3463–3474. 32 indexed citations
19.
Nairz, Knud, et al.. (2006). Overgrowth caused by misexpression of a microRNA with dispensable wild-type function. Developmental Biology. 291(2). 314–324. 41 indexed citations
20.
Moser, Martin, et al.. (2003). Abciximab, Eptifibatide, and Tirofiban Exhibit Dose-dependent Potencies to Dissolve Platelet Aggregates. Journal of Cardiovascular Pharmacology. 41(4). 586–592. 80 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026