David B. Hawver

498 total citations
10 papers, 428 citations indexed

About

David B. Hawver is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, David B. Hawver has authored 10 papers receiving a total of 428 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 6 papers in Cellular and Molecular Neuroscience and 5 papers in Physiology. Recurrent topics in David B. Hawver's work include Alzheimer's disease research and treatments (5 papers), Nerve injury and regeneration (3 papers) and Nuclear Receptors and Signaling (2 papers). David B. Hawver is often cited by papers focused on Alzheimer's disease research and treatments (5 papers), Nerve injury and regeneration (3 papers) and Nuclear Receptors and Signaling (2 papers). David B. Hawver collaborates with scholars based in United States and Germany. David B. Hawver's co-authors include Clinton B. Wright, William Z. Potter, Husseini K. Manji, Guang Chen, Kathleen C. Flanders, Guang Chen, Baishen Pan, Husseini K. Manji, Samuel Schacher and Edward S. Kim and has published in prestigious journals such as Brain Research, Neuroscience and Journal of Neurochemistry.

In The Last Decade

David B. Hawver

10 papers receiving 413 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David B. Hawver United States	9	222	155	126	101	64	10	428
Josephine Heffernan Ireland	9	153 0.7×	176 1.1×	103 0.8×	89 0.9×	99 1.5×	18	410
K Morimoto Japan	10	101 0.5×	143 0.9×	79 0.6×	113 1.1×	21 0.3×	25	346
Mónica E. Ureña‐Guerrero Mexico	14	173 0.8×	254 1.6×	76 0.6×	71 0.7×	44 0.7×	25	488
Janet Kennedy Canada	9	171 0.8×	215 1.4×	37 0.3×	118 1.2×	65 1.0×	13	569
Mikael Ängehagen Sweden	8	182 0.8×	279 1.8×	167 1.3×	39 0.4×	83 1.3×	8	456
Shu-Hui Chuang United States	12	190 0.9×	191 1.2×	125 1.0×	30 0.3×	41 0.6×	13	442
Joeri Van Liefferinge Belgium	14	228 1.0×	279 1.8×	65 0.5×	69 0.7×	28 0.4×	19	597
Xiao-Xin Yan United States	11	160 0.7×	297 1.9×	178 1.4×	207 2.0×	90 1.4×	14	539
Marleisje Njunting Germany	7	205 0.9×	390 2.5×	198 1.6×	60 0.6×	67 1.0×	9	564
Jing-Quan Lan United States	7	219 1.0×	195 1.3×	50 0.4×	38 0.4×	20 0.3×	8	479

Countries citing papers authored by David B. Hawver

Since Specialization

Citations

This map shows the geographic impact of David B. Hawver's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David B. Hawver with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David B. Hawver more than expected).

Fields of papers citing papers by David B. Hawver

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David B. Hawver. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David B. Hawver. The network helps show where David B. Hawver may publish in the future.

Co-authorship network of co-authors of David B. Hawver

This figure shows the co-authorship network connecting the top 25 collaborators of David B. Hawver. A scholar is included among the top collaborators of David B. Hawver based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David B. Hawver. David B. Hawver is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

Luo, Jin Jun, Matthew Wallace, David B. Hawver, John W. Kusiak, & William Wallace. (2001). Characterization of the neurotrophic interaction between nerve growth factor and secreted ?-amyloid precursor protein. Journal of Neuroscience Research. 63(5). 410–420. 31 indexed citations

Luo, Jin Jun, Matthew Wallace, David B. Hawver, John W. Kusiak, & William Wallace. (2001). Characterization of the neurotrophic interaction between nerve growth factor and secreted α‐amyloid precursor protein. Journal of Neuroscience Research. 63(5). 410–420. 1 indexed citations

Ren, Rutong, James J. Lah, Anke Diehlmann, et al.. (1999). Differential effects of transforming growth factor-βs and glial cell line-derived neurotrophic factor on gene expression of presenilin-1 in human post-mitotic neurons and astrocytes. Neuroscience. 93(3). 1041–1049. 15 indexed citations

Kim, Edward S., et al.. (1998). Transforming growth factor-β inhibits apoptosis induced by β-amyloid peptide fragment 25–35 in cultured neuronal cells. Molecular Brain Research. 62(2). 122–130. 41 indexed citations

Luo, Ying, et al.. (1997). Physiological levels of β-amyloid peptide stimulate protein kinase C in PC12 cells. Brain Research. 769(2). 287–295. 32 indexed citations

Hawver, David B., et al.. (1997). Transforming growth factor-β protects human hNT cells from degeneration induced by β-amyloid peptide: involvement of the TGF-β type II receptor. Molecular Brain Research. 48(2). 315–322. 38 indexed citations

Chen, Guang, Baishen Pan, David B. Hawver, et al.. (1996). Attenuation of Cyclic AMP Production by Carbamazepine. Journal of Neurochemistry. 67(5). 2079–2086. 70 indexed citations

Chen, Guang, Husseini K. Manji, David B. Hawver, Clinton B. Wright, & William Z. Potter. (1994). Chronic Sodium Valproate Selectively Decreases Protein Kinase C α and ε In Vitro. Journal of Neurochemistry. 63(6). 2361–2364. 147 indexed citations

Hawver, David B. & Samuel Schacher. (1993). Selective fasciculation as a mechanism for the formation of specific chemical connections between Aplysia neurons in vitro. Journal of Neurobiology. 24(3). 368–383. 17 indexed citations

10.

Mahadik, Sahebarao P., et al.. (1989). GM1 Ganglioside treatment after global ischemia protects changes in membrane fatty acids and properties of Na+, K+‐ATPase and Mg2+‐ATPase. Journal of Neuroscience Research. 24(3). 402–412. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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