David A. van Heel

26.6k total citations · 2 hit papers
97 papers, 6.5k citations indexed

About

David A. van Heel is a scholar working on Genetics, Immunology and Surgery. According to data from OpenAlex, David A. van Heel has authored 97 papers receiving a total of 6.5k indexed citations (citations by other indexed papers that have themselves been cited), including 59 papers in Genetics, 33 papers in Immunology and 25 papers in Surgery. Recurrent topics in David A. van Heel's work include Inflammatory Bowel Disease (30 papers), Celiac Disease Research and Management (23 papers) and Helicobacter pylori-related gastroenterology studies (18 papers). David A. van Heel is often cited by papers focused on Inflammatory Bowel Disease (30 papers), Celiac Disease Research and Management (23 papers) and Helicobacter pylori-related gastroenterology studies (18 papers). David A. van Heel collaborates with scholars based in United Kingdom, United States and Netherlands. David A. van Heel's co-authors include Cisca Wijmenga, Karen A. Hunt, Derek P. Jewell, Matthew A. Brown, Graham Heap, Miles Parkes, Adrián Cortés, Gosia Trynka, P Dubois and Dermot McGovern and has published in prestigious journals such as New England Journal of Medicine, The Lancet and Nucleic Acids Research.

In The Last Decade

David A. van Heel

95 papers receiving 6.4k citations

Hit Papers

Shared and Distinct Genetic Variants in Type 1 Diabetes a... 2008 2026 2014 2020 2008 2023 100 200 300 400 500

Peers

David A. van Heel
Marco Londei United Kingdom
Richard H. Duerr United States
Vijay Yajnik United States
Stephen P. James United States
Marco Londei United Kingdom
David A. van Heel
Citations per year, relative to David A. van Heel David A. van Heel (= 1×) peers Marco Londei

Countries citing papers authored by David A. van Heel

Since Specialization
Citations

This map shows the geographic impact of David A. van Heel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David A. van Heel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David A. van Heel more than expected).

Fields of papers citing papers by David A. van Heel

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David A. van Heel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David A. van Heel. The network helps show where David A. van Heel may publish in the future.

Co-authorship network of co-authors of David A. van Heel

This figure shows the co-authorship network connecting the top 25 collaborators of David A. van Heel. A scholar is included among the top collaborators of David A. van Heel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David A. van Heel. David A. van Heel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Walter, Klaudia, Qin Huang, Juha Karjalainen, et al.. (2025). Widespread recessive effects on common diseases in a cohort of 44,000 British Pakistanis and Bangladeshis with high autozygosity. The American Journal of Human Genetics. 112(6). 1316–1329.
2.
Magavern, Emma, Benjamin M. Jacobs, Helen R. Warren, et al.. (2023). CYP2C19 Genotype Prevalence and Association With Recurrent Myocardial Infarction in British–South Asians Treated With Clopidogrel. JACC Advances. 2(7). 100573–100573. 13 indexed citations
3.
Patel, Aniruddh P., Minxian Wang, Yunfeng Ruan, et al.. (2023). A multi-ancestry polygenic risk score improves risk prediction for coronary artery disease. Nature Medicine. 29(7). 1793–1803. 98 indexed citations breakdown →
4.
Hodgson, Sam, Qin Qin Huang, Neneh Sallah, et al.. (2022). Integrating polygenic risk scores in the prediction of type 2 diabetes risk and subtypes in British Pakistanis and Bangladeshis: A population-based cohort study. PLoS Medicine. 19(5). e1003981–e1003981. 33 indexed citations
5.
Huang, Qin Qin, Neneh Sallah, Bhavi Trivedi, et al.. (2022). Transferability of genetic loci and polygenic scores for cardiometabolic traits in British Pakistani and Bangladeshi individuals. Nature Communications. 13(1). 4664–4664. 34 indexed citations
6.
Arciero, Elena, Sufyan Abid Dogra, Daniel Malawsky, et al.. (2021). Fine-scale population structure and demographic history of British Pakistanis. Nature Communications. 12(1). 7189–7189. 14 indexed citations
7.
Minikel, Eric Vallabh, Konrad J. Karczewski, Beryl B. Cummings, et al.. (2020). Evaluating drug targets through human loss-of-function genetic variation. Figshare. 92 indexed citations
8.
Sharp, Seth A., Samuel E. Jones, Michael N. Weedon, et al.. (2020). A single nucleotide polymorphism genetic risk score to aid diagnosis of coeliac disease: a pilot study in clinical care. Alimentary Pharmacology & Therapeutics. 52(7). 1165–1173. 14 indexed citations
9.
Festen, Eleonora A., Philippe Goyette, Todd J. Green, et al.. (2011). A Meta-Analysis of Genome-Wide Association Scans Identifies IL18RAP, PTPN2, TAGAP, and PUS10 As Shared Risk Loci for Crohn's Disease and Celiac Disease. PLoS Genetics. 7(1). e1001283–e1001283. 162 indexed citations
10.
Blaydon, Diana C., Paolo Biancheri, Wei‐Li Di, et al.. (2011). Inflammatory Skin and Bowel Disease Linked toADAM17Deletion. New England Journal of Medicine. 365(16). 1502–1508. 235 indexed citations
11.
Zhernakova, Alexandra, Clara C. Elbers, Bart Ferwerda, et al.. (2010). Evolutionary and Functional Analysis of Celiac Risk Loci Reveals SH2B3 as a Protective Factor against Bacterial Infection. The American Journal of Human Genetics. 86(6). 970–977. 127 indexed citations
12.
Heap, Graham, Jennie H. M. Yang, Kate Downes, et al.. (2009). Genome-wide analysis of allelic expression imbalance in human primary cells by high-throughput transcriptome resequencing. Human Molecular Genetics. 19(1). 122–134. 144 indexed citations
13.
Monsuur, Alienke J., Paul I. W. de Bakker, Alexandra Zhernakova, et al.. (2008). Effective Detection of Human Leukocyte Antigen Risk Alleles in Celiac Disease Using Tag Single Nucleotide Polymorphisms. PLoS ONE. 3(5). e2270–e2270. 117 indexed citations
14.
Cartwright, Neil, Oliver Murch, Shaun K. McMaster, et al.. (2007). Selective NOD1 Agonists Cause Shock and Organ Injury/Dysfunction in Vivo. American Journal of Respiratory and Critical Care Medicine. 175(6). 595–603. 54 indexed citations
15.
Heel, David A. van. (2005). Kirsner’s Inflammatory Bowel Disease, 6th edn. Gut. 54(8). 1208.1–1208. 8 indexed citations
16.
Heel, David A. van, Sheila Fisher, Andrew Kirby, et al.. (2004). Inflammatory bowel disease susceptibility loci defined by genome scan meta-analysis of 1952 affected relative pairs. Human Molecular Genetics. 13(7). 763–770. 184 indexed citations
17.
Heel, David A. van, Irina A. Udalova, Dermot McGovern, et al.. (2002). Inflammatory bowel disease is associated with functional TNF polymorphism affecting OCT1/NF-kappa B interaction. Gut. 50. 1 indexed citations
18.
Heel, David A. van, Irina A. Udalova, Dermot McGovern, et al.. (2002). Inflammatory bowel disease is associated with a functional TNF polymorphism that affects an OCT1/NF-KB transcription factor interaction. Gastroenterology. 122. 1 indexed citations
19.
Crane, Alison M., L Bradbury, David A. van Heel, et al.. (2002). Role of NOD2 variants in spondylarthritis. Queensland's institutional digital repository (The University of Queensland). 7 indexed citations
20.
Heel, David A. van, et al.. (1998). DIABETIC NEUROPATHIC CACHEXIA: THE IMPORTANCE OF POSITIVE RECOGNITION AND EARLY NUTRITIONAL SUPPORT. International Journal of Clinical Practice. 52(8). 591–592. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026