David A. Foster

10.1k total citations · 1 hit paper
144 papers, 8.4k citations indexed

About

David A. Foster is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, David A. Foster has authored 144 papers receiving a total of 8.4k indexed citations (citations by other indexed papers that have themselves been cited), including 102 papers in Molecular Biology, 30 papers in Oncology and 24 papers in Cancer Research. Recurrent topics in David A. Foster's work include Protein Kinase Regulation and GTPase Signaling (53 papers), PI3K/AKT/mTOR signaling in cancer (37 papers) and Metabolism, Diabetes, and Cancer (20 papers). David A. Foster is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (53 papers), PI3K/AKT/mTOR signaling in cancer (37 papers) and Metabolism, Diabetes, and Cancer (20 papers). David A. Foster collaborates with scholars based in United States, Canada and Japan. David A. Foster's co-authors include Alfredo Toschi, Lizhong Xu, Zheng Yang, Mahesh Saqcena, Desmond N. Jackson, Noga Gadir, Zhimin Lu, Paige Yellen, Yingjie Shen and Evan Lee and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

David A. Foster

143 papers receiving 8.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Hypoxic induction of human vascular endothelial growth factor expression through c-Src activation

1995 517 citations David A. Foster et al. Nature profile →

Peers

Countries citing papers authored by David A. Foster

Since Specialization

Citations

This map shows the geographic impact of David A. Foster's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David A. Foster with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David A. Foster more than expected).

Fields of papers citing papers by David A. Foster

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David A. Foster. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David A. Foster. The network helps show where David A. Foster may publish in the future.

Co-authorship network of co-authors of David A. Foster

This figure shows the co-authorship network connecting the top 25 collaborators of David A. Foster. A scholar is included among the top collaborators of David A. Foster based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David A. Foster. David A. Foster is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Inhibiting glutamine utilization creates a synthetic lethality for suppression of ATP citrate lyase in KRas-driven cancer cells 2022 ·PLoS ONE ·Ahmet Hatipoğlu, Deepak Menon, Talia Levy, María A. Frías, David A. Foster	10
2	Lipid sensing by mTOR complexes via de novo synthesis of phosphatidic acid 2017 ·Journal of Biological Chemistry ·Deepak Menon, Darin Salloum, (unknown), (unknown), (unknown), María A. Frías, Jessica Sudderth, Pei-Hsuan Chen, Ralph J. DeBerardinis, David A. Foster	101
3	Synthetic lethality in KRas-driven cancer cells created by glutamine deprivation 2015 ·Oncoscience ·Suman Mukhopadhyay, Mahesh Saqcena, David A. Foster	47
4	Mutant Ras Elevates Dependence on Serum Lipids and Creates a Synthetic Lethality for Rapamycin 2014 ·Molecular Cancer Therapeutics ·Darin Salloum, Suman Mukhopadhyay, Kaity Tung, (unknown), David A. Foster	23
5	BxPC3 pancreatic cancer cells express a truncated Smad4 protein upon PI3K and mTOR inhibition 2014 ·Oncology Letters ·(unknown), (unknown), David A. Foster	2
6	Suppression of Hypoxia-Inducible Factor 2α Restores p53 Activity via Hdm2 and Reverses Chemoresistance of Renal Carcinoma Cells 2009 ·Cancer Research ·Andrew M. Roberts, Ian R. Watson, Andrew Evans, David A. Foster, Meredith S. Irwin, Michael Ohh	67
7	Suppression of cell migration by protein kinase Cδ 2005 ·Oncogene ·Desmond N. Jackson, Zheng Yang, (unknown), (unknown), Keiko Nakayama, Keiichi I. Nakayama, Michael Humphries, Mary E. Reyland, David A. Foster	44
8	mTOR-dependent Suppression of Protein Phosphatase 2A Is Critical for Phospholipase D Survival Signals in Human Breast Cancer Cells 2005 ·Journal of Biological Chemistry ·Hui Li, Vanessa Rodrik-Outmezguine, Rafal M. Pielak, (unknown), Zheng Yang, David A. Foster	48
9	Elevated Phospholipase D Activity in H-Ras- but Not K-Ras-Transformed Cells by the Synergistic Action of RalA and ARF6 2002 ·Molecular and Cellular Biology ·Lizhong Xu, Paul Frankel, Desmond N. Jackson, (unknown), Rita L. Boshans, Crislyn D’Souza‐Schorey, David A. Foster	47
10	Phospholipase D overcomes cell cycle arrest induced by high-intensity Raf signaling 2002 ·Oncogene ·Troy Joseph, (unknown), Paul Frankel, (unknown), Eugen Kerkhoff, Ulf R. Rapp, David A. Foster	32
11	Downregulating PKC δ provides a PI3K/Akt-independent survival signal that overcomes apoptotic signals generated by c-Src overexpression 2002 ·Oncogene ·Minghao Zhong, Zhimin Lu, David A. Foster	42
12	RalA Mediates v-Src, v-Ras, and v-Raf Regulation of CD44 and Fibronectin Expression in NIH3T3 Fibroblasts 2001 ·Biochemical and Biophysical Research Communications ·Virginia Ladeda, Paul Frankel, Larry A. Feig, David A. Foster, Elisa Bal de Kier Joffé, Julio A. Aguirre‐Ghiso	11
13	Epidemiology of Reye’s Syndrome, United States, 1991–1994: Comparison of CDC Surveillance and Hospital Admission Data 2000 ·Neuroepidemiology ·Kevin M. Sullivan, Ermias D. Belay, (unknown), David A. Foster, Dale Nordenberg	14
14	Novel Recruitment of Shc, Grb2, and Sos by Fibroblast Growth Factor Receptor-1 in v-Src-Transformed Cells 1998 ·Biochemical and Biophysical Research Communications ·Marcello Curto, Paul Frankel, (unknown), David A. Foster	12
15	The engineering of creativity: a review of Boden's the creative mind 1995 ·Artificial Intelligence ·Roger C. Schank, David A. Foster	8
16	Involvement of Ral GTPase in v-Src-induced phospholipase D activation 1995 ·Nature ·Hong Jiang, Jing-Qing Luo, Takeshi Urano, Paul Frankel, Zhimin Lu, David A. Foster, Larry A. Feig	232
17	Ras Mediates the Activation of Phospholipase D by v-Src 1995 ·Journal of Biological Chemistry ·Hong Jiang, Zhimin Lu, Jing-Qing Luo, Alan Wolfman, David A. Foster	84
18	A Dominant Negative RAF-1 Mutant Prevents V-SRC-Induced Transformation 1993 ·Biochemical and Biophysical Research Communications ·Sajjad A. Qureshi, Cecil K. Joseph, (unknown), Jian Song, Ruchika Gupta, (unknown), Ulf R. Rapp, David A. Foster	19
19	Influenza Vaccine Effectiveness in Preventing Hospitalization for Pneumonia in the Elderly 1992 ·American Journal of Epidemiology ·David A. Foster, AkkeNeel Talsma, Alice Furumoto-Dawson, Suzanne E. Ohmit, Janice R. Margulies, Nancy H. Arden, Arnold S. Monto	210
20	v-Src Increases Diacylglycerol Levels Via a Type D Phospholipase-Mediated Hydrolysis of Phosphatidylcholine 1991 ·Molecular and Cellular Biology ·Jianguo Song, Lawrence M. Pfeffer, David A. Foster	21

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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