Daria Diodato

2.9k citations
44 papers · 1.2k indexed · h-index 18
Topics
Mitochondrial Function and Pathology (29 papers)Metabolism and Genetic Disorders (19 papers)ATP Synthase and ATPases Research (10 papers)

In The Last Decade

Daria Diodato

42 papers receiving 1.2k citations

Peers

Daria Diodato
Comparison fields: 5 of 88
  • Molecular Biology 868
  • Clinical Biochemistry 535
  • Rheumatology 215
  • Genetics 89
  • Physiology 88
Replace Alberto Blázquez with:
Alberto Blázquez Spain
Saba Tadesse United States
K Naess Sweden
Langping He United Kingdom
Anna Ardissone Italy
Nataliya Povalko Japan
Alessandra Maresca Italy
Inés García‐Consuegra Spain
Lynne A. Wolfe United States
K. Tada Japan
Daria Diodato relative to Alberto Blázquez Spain Alberto Blázquez's profile →
Citations per field
00.5×1.5×1.9×
Alberto Blázquez · 1×
Citations per year

Countries citing papers authored by Daria Diodato

Since Specialization
Citations

This map shows the geographic impact of Daria Diodato's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daria Diodato with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daria Diodato more than expected).

Fields of papers citing papers by Daria Diodato

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daria Diodato. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daria Diodato. The network helps show where Daria Diodato may publish in the future.

Co-authorship network of co-authors of Daria Diodato

This figure shows the co-authorship network connecting the top 25 collaborators of Daria Diodato. A scholar is included among the top collaborators of Daria Diodato based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daria Diodato. Daria Diodato is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 0
2 4
3 0
4 9
5 13
6 17
7 4
8 4
9
UFM1 founder mutation in the Roma population causes severe variant of Hypomyelination with Atrophy of the Basal ganglia and Cerebellum (H-ABC)
1
10 8
11 18
12 34
13 13
14 23
15 45
16 14
17 16
18 131
19 39
20 1

About Daria Diodato

Daria Diodato is a scholar working on Clinical Biochemistry, Molecular Biology and Rheumatology, having authored 44 papers that have together received 1.2k indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (29 papers), Metabolism and Genetic Disorders (19 papers) and ATP Synthase and ATPases Research (10 papers). The work is most often cited by research in Clinical Biochemistry (535 citations), Rheumatology (215 citations) and Molecular Biology (868 citations). Daria Diodato has collaborated with scholars based in Italy, United Kingdom and United States. Frequent co-authors include Daniele Ghezzi, Valeria Tiranti, Carlo Dionisi‐Vici, Sara Boenzi, Massimo Zeviani, Enrico Bertini, Diego Martinelli, Costanza Lamperti, Rosalba Carrozzo and Eleonora Lamantea. Their work appears in journals such as Nature Genetics, Brain and Neurology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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