Danilo B. Medinas

1.7k total citations
29 papers, 998 citations indexed

About

Danilo B. Medinas is a scholar working on Cell Biology, Neurology and Molecular Biology. According to data from OpenAlex, Danilo B. Medinas has authored 29 papers receiving a total of 998 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Cell Biology, 14 papers in Neurology and 11 papers in Molecular Biology. Recurrent topics in Danilo B. Medinas's work include Endoplasmic Reticulum Stress and Disease (13 papers), Amyotrophic Lateral Sclerosis Research (13 papers) and Neurogenetic and Muscular Disorders Research (6 papers). Danilo B. Medinas is often cited by papers focused on Endoplasmic Reticulum Stress and Disease (13 papers), Amyotrophic Lateral Sclerosis Research (13 papers) and Neurogenetic and Muscular Disorders Research (6 papers). Danilo B. Medinas collaborates with scholars based in Chile, United States and Brazil. Danilo B. Medinas's co-authors include Claudio Hetz, Ohára Augusto, Giselle Cerchiaro, Pablo Rozas, Vicente Valenzuela, Soledad Matus, Ute Woehlbier, Víctor Hugo Cornejo, Claudio Soto and Francisca Martínez Traub and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Journal of Biological Chemistry.

In The Last Decade

Danilo B. Medinas

26 papers receiving 987 citations

Peers

Danilo B. Medinas
Daniel Little United Kingdom
Scott P. Allen United Kingdom
María E. Solesio United States
Kristine M. Robinson United States
Megan Whitnall Australia
Daniel Little United Kingdom
Danilo B. Medinas
Citations per year, relative to Danilo B. Medinas Danilo B. Medinas (= 1×) peers Daniel Little

Countries citing papers authored by Danilo B. Medinas

Since Specialization
Citations

This map shows the geographic impact of Danilo B. Medinas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Danilo B. Medinas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Danilo B. Medinas more than expected).

Fields of papers citing papers by Danilo B. Medinas

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Danilo B. Medinas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Danilo B. Medinas. The network helps show where Danilo B. Medinas may publish in the future.

Co-authorship network of co-authors of Danilo B. Medinas

This figure shows the co-authorship network connecting the top 25 collaborators of Danilo B. Medinas. A scholar is included among the top collaborators of Danilo B. Medinas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Danilo B. Medinas. Danilo B. Medinas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Cabral‐Miranda, Felipe, Ana Paula Bérgamo Araújo, Danilo B. Medinas, & Flávia Carvalho Alcântara Gomes. (2025). Astrocytic Hevin/SPARCL‐1 Regulates Cognitive Decline in Pathological and Normal Brain Aging. Aging Cell. 24(5). e14493–e14493. 4 indexed citations
2.
Sepulveda, Martin, Viviana Pérez, Jorge Ojeda, et al.. (2025). Expression of protein disulfide isomerase A3Q481K variant associated with amyotrophic lateral sclerosis triggers disease features in mice. Neurobiology of Disease. 212. 106947–106947.
3.
Rodríguez, Leonardo, et al.. (2024). Overexpression of autophagy enhancer PACER/RUBCNL in neurons accelerates disease in the SOD1G93A ALS mouse model. Biological Research. 57(1). 86–86. 2 indexed citations
4.
Medinas, Danilo B., et al.. (2023). Immunohistochemical characterisation of the adult Nothobranchius furzeri intestine. Cell and Tissue Research. 395(1). 21–38. 2 indexed citations
5.
Medinas, Danilo B., et al.. (2022). Emerging roles of endoplasmic reticulum proteostasis in brain development. PubMed. 170. 203781–203781. 15 indexed citations
6.
Medinas, Danilo B., Pablo Rozas, & Claudio Hetz. (2022). Critical roles of protein disulfide isomerases in balancing proteostasis in the nervous system. Journal of Biological Chemistry. 298(7). 102087–102087. 29 indexed citations
7.
Rozas, Pablo, Francisca Martínez Traub, Viviana Pérez, et al.. (2021). Protein disulfide isomerase ERp57 protects early muscle denervation in experimental ALS. Acta Neuropathologica Communications. 9(1). 21–21. 12 indexed citations
8.
Nassif, Melissa, Cristian Cortéz, Sergio Espinoza, et al.. (2019). Network approach identifies Pacer as an autophagy protein involved in ALS pathogenesis. Molecular Neurodegeneration. 14(1). 14–14. 25 indexed citations
9.
Medinas, Danilo B., Jaime Maripillán, Ariel F. Castro, et al.. (2019). β-catenin aggregation in models of ALS motor neurons: GSK3β inhibition effect and neuronal differentiation. Neurobiology of Disease. 130. 104497–104497. 20 indexed citations
10.
Medinas, Danilo B., Vicente Valenzuela, & Claudio Hetz. (2017). Proteostasis disturbance in amyotrophic lateral sclerosis. Human Molecular Genetics. 26(R2). R91–R104. 41 indexed citations
11.
Duran‐Aniotz, Claudia, Víctor Hugo Cornejo, Sandra Espinoza, et al.. (2017). IRE1 signaling exacerbates Alzheimer’s disease pathogenesis. Acta Neuropathologica. 134(3). 489–506. 159 indexed citations
12.
Medinas, Danilo B., Francisca Martínez Traub, Pablo Rozas, et al.. (2017). Disulfide cross-linked multimers of TDP-43 and spinal motoneuron loss in a TDP-43A315T ALS/FTD mouse model. Scientific Reports. 7(1). 14266–14266. 19 indexed citations
13.
Medinas, Danilo B., et al.. (2017). Fine-Tuning ER Stress Signal Transducers to Treat Amyotrophic Lateral Sclerosis. Frontiers in Molecular Neuroscience. 10. 216–216. 17 indexed citations
14.
Ramírez, Marcos, et al.. (2016). Desafíos en el diagnóstico de enfermedad de Creutzfeldt-Jakob: Caso clínico. Revista médica de Chile. 144(6). 796–806.
15.
Rozas, Pablo, et al.. (2016). The ER proteostasis network in ALS: Determining the differential motoneuron vulnerability. Neuroscience Letters. 636. 9–15. 35 indexed citations
16.
Medinas, Danilo B., Adriano B. Chaves‐Filho, Thiago C. Genaro‐Mattos, et al.. (2015). Oligomerization of Cu,Zn-Superoxide Dismutase (SOD1) by Docosahexaenoic Acid and Its Hydroperoxides In Vitro: Aggregation Dependence on Fatty Acid Unsaturation and Thiols. PLoS ONE. 10(4). e0125146–e0125146. 16 indexed citations
17.
Matus, Soledad, Danilo B. Medinas, & Claudio Hetz. (2014). Common Ground: Stem Cell Approaches Find Shared Pathways Underlying ALS. Cell stem cell. 14(6). 697–699. 17 indexed citations
18.
Medinas, Danilo B., et al.. (2011). Generation of Singlet Oxygen by the Glyoxal–Peroxynitrite System. Journal of the American Chemical Society. 133(51). 20761–20768. 28 indexed citations
19.
Medinas, Danilo B., et al.. (2007). The carbonate radical and related oxidants derived from bicarbonate buffer. IUBMB Life. 59(4-5). 255–262. 153 indexed citations
20.
Medinas, Danilo B., et al.. (2004). Tempol diverts peroxynitrite/carbon dioxide reactivity toward albumin and cells from protein–tyrosine nitration to protein–cysteine nitrosation. Free Radical Biology and Medicine. 38(2). 189–200. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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