Daniela Ehrlich

568 citations
21 papers · 461 indexed · h-index 11

Impact in

Papers in

    • Neuroinflammation and Neurodegeneration Mechanisms 4
    • Transcranial Magnetic Stimulation Studies 2
    • Neurological Disease Mechanisms and Treatments 2
    • Tryptophan and brain disorders 2

Daniela Ehrlich

20 papers receiving 453 citations

Peers

Daniela Ehrlich
Comparison fields: 5 of 86
  • Biological Psychiatry 78
  • Neurology 108
  • Physiology 161
  • Behavioral Neuroscience 22
  • Developmental Neuroscience 21
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Citations per field
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Citations per year

Countries citing papers authored by Daniela Ehrlich

Since Specialization
Citations

This map shows the geographic impact of Daniela Ehrlich's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniela Ehrlich with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniela Ehrlich more than expected).

Fields of papers citing papers by Daniela Ehrlich

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniela Ehrlich. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniela Ehrlich. The network helps show where Daniela Ehrlich may publish in the future.

Co-authors

The 25 scholars most cited alongside Daniela Ehrlich, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Daniela Ehrlich Line = papers co-authored together Daniela Ehrlich links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 21 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2021102
2 201256
3 201243
4 201242
5 201542
6 201231
7 201524
8 201223
9 202121
10 201220
11 201313
12 20129
13 20138
14 20227
15 20124
16 20134
17 20214
18 20134
19 20093
20 20211

About Daniela Ehrlich

Daniela Ehrlich is a scholar working on Neurology, Biological Psychiatry, Physiology, Clinical Biochemistry and Cellular and Molecular Neuroscience, having authored 21 papers that have together received 461 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (10 papers), Neuroinflammation and Neurodegeneration Mechanisms (4 papers), Neuropeptides and Animal Physiology (3 papers), Glaucoma and retinal disorders (2 papers), Transcranial Magnetic Stimulation Studies (2 papers), Tryptophan and brain disorders (2 papers), Advanced Glycation End Products research (2 papers) and Neurological Disease Mechanisms and Treatments (2 papers). The work is most often cited by research in Biological Psychiatry (78 citations), Neurology (108 citations), Physiology (161 citations), Behavioral Neuroscience (22 citations) and Developmental Neuroscience (21 citations). Daniela Ehrlich has collaborated with scholars based in Austria, Czechia and Germany. Frequent co-authors include Christian Humpel, Michael Pirchl, Katharina Kurz, Dietmar Fuchs, Tanja Hochstrasser, Kostja Steiner, Josef Marksteiner, Simon Geisler, Friedrich Leblhuber and Lukas Lanser. Their work appears in journals such as Current Alzheimer Research, Platelets, Journal of the Neurological Sciences, Neuropeptides and Neuroscience.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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