Daniela Asslaber

841 citations
22 papers · 557 indexed · h-index 14

Impact in

  • Genetics top 5%
    • Chronic Lymphocytic Leukemia Research
    • Immunodeficiency and Autoimmune Disorders
    • Immune Cell Function and Interaction

Papers in

    • Chronic Lymphocytic Leukemia Research 16
    • Glycosylation and Glycoproteins Research 3
    • PI3K/AKT/mTOR signaling in cancer 3

Daniela Asslaber

20 papers receiving 551 citations

Peers

Daniela Asslaber
Comparison fields: 5 of 63
  • Genetics 231
  • Immunology 172
  • Pathology and Forensic Medicine 134
  • Cancer Research 107
  • Biological Psychiatry 15
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Citations per field
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Citations per year

Countries citing papers authored by Daniela Asslaber

Since Specialization
Citations

This map shows the geographic impact of Daniela Asslaber's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniela Asslaber with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniela Asslaber more than expected).

Fields of papers citing papers by Daniela Asslaber

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniela Asslaber. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniela Asslaber. The network helps show where Daniela Asslaber may publish in the future.

Co-authors

The 25 scholars most cited alongside Daniela Asslaber, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Daniela Asslaber Line = papers co-authored together Daniela Asslaber links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 22 papers — load more, or switch the sort, to bring in the rest.

#Work
1 201078
2 201174
3 201062
4 201460
5 201045
6 202239
7 201232
8 201832
9 200826
10 201821
11 201817
12 201917
13 201515
14 201913
15 201612
16 20168
17 20102
18 20112
19 20111
20 20101

About Daniela Asslaber

Daniela Asslaber is a scholar working on Genetics, Molecular Biology, Immunology, Oncology and Cancer Research, having authored 22 papers that have together received 557 indexed citations. Recurring topics across this work include Chronic Lymphocytic Leukemia Research (16 papers), Immunodeficiency and Autoimmune Disorders (6 papers), Lymphoma Diagnosis and Treatment (3 papers), MicroRNA in disease regulation (3 papers), Glycosylation and Glycoproteins Research (3 papers), PI3K/AKT/mTOR signaling in cancer (3 papers), Cancer-related Molecular Pathways (2 papers) and Phagocytosis and Immune Regulation (2 papers). The work is most often cited by research in Genetics (231 citations), Immunology (172 citations), Pathology and Forensic Medicine (134 citations), Cancer Research (107 citations) and Biological Psychiatry (15 citations). Daniela Asslaber has collaborated with scholars based in Austria, Germany and Slovakia. Frequent co-authors include Richard Greil, Alexander Egle, Tanja Nicole Hartmann, Olaf Merkel, Josefina Piñón Hofbauer, Ingeborg Tinhofer, Inge Tinhofer, Markus Stöcher, Ursula Denk and Christoph Heyder. Their work appears in journals such as Blood, Cancer Research, Oncotarget, Leukemia and Clinical Cancer Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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