Eiji Satoh

603 total citations
22 papers, 503 citations indexed

About

Eiji Satoh is a scholar working on Oncology, Molecular Biology and Immunology and Allergy. According to data from OpenAlex, Eiji Satoh has authored 22 papers receiving a total of 503 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Oncology, 8 papers in Molecular Biology and 6 papers in Immunology and Allergy. Recurrent topics in Eiji Satoh's work include Cell Adhesion Molecules Research (6 papers), Glioma Diagnosis and Treatment (4 papers) and TGF-β signaling in diseases (4 papers). Eiji Satoh is often cited by papers focused on Cell Adhesion Molecules Research (6 papers), Glioma Diagnosis and Treatment (4 papers) and TGF-β signaling in diseases (4 papers). Eiji Satoh collaborates with scholars based in Japan. Eiji Satoh's co-authors include Hirofumi Naganuma, Hideaki NUKUI, Atsushi Sasaki, Takayuki Asahara, Tomoyuki Kawataki, Shin Nakano, Toru Horikoshi, Hiroyuki Kinouchi, Shuichiro Maeda and Tadashi Mabuchi and has published in prestigious journals such as Journal of neurosurgery, Neurosurgery and Journal of Neuro-Oncology.

In The Last Decade

Eiji Satoh

22 papers receiving 499 citations

Peers

Eiji Satoh

ONCOL

IMMUN

GENET

M. Mirakhur United Kingdom

Tomoyuki Kawataki Japan

Marcel van der Weiden Netherlands

Eva Maria Deiner Austria

Isabel Burghardt Switzerland

Zhongbo Yang United States

Noriyuki Tsukamoto Japan

N Shimizu Japan

Jumpei Homma Japan

Stacey Ivanchuk Canada

M. Mirakhur United Kingdom

Eiji Satoh

175 ×0.9

69 ×0.4

ONCOL

92 ×0.7

IMMUN

43 ×0.4

GENET

52 ×0.8

Citations per year, relative to Eiji Satoh Eiji Satoh (= 1×) peers M. Mirakhur

Countries citing papers authored by Eiji Satoh

Since Specialization

Citations

This map shows the geographic impact of Eiji Satoh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eiji Satoh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eiji Satoh more than expected).

Fields of papers citing papers by Eiji Satoh

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eiji Satoh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eiji Satoh. The network helps show where Eiji Satoh may publish in the future.

Co-authorship network of co-authors of Eiji Satoh

This figure shows the co-authorship network connecting the top 25 collaborators of Eiji Satoh. A scholar is included among the top collaborators of Eiji Satoh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eiji Satoh. Eiji Satoh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kawataki, Tomoyuki, et al.. (2013). Expression of Indoleamine 2,3-Dioxygenase and Correlation With Pathological Malignancy in Gliomas. Neurosurgery. 72(6). 1031–1039. 76 indexed citations

Naganuma, Hirofumi, et al.. (2007). Growth Potential of Orbital Cavernous Hemangioma Suggested by Vascular Endothelial Growth Factor and its Receptor Flk-1. Neurologia medico-chirurgica. 47(1). 5–10. 21 indexed citations

Satoh, Eiji, et al.. (2006). Reduced expression of the transporter associated with antigen processing 1 molecule in malignant glioma cells, and its restoration by interferon-γ and -β. Journal of neurosurgery. 104(2). 264–271. 12 indexed citations

Watanabe, Akira, Takeo Horikoshi, Hirofumi Naganuma, Eiji Satoh, & Hideaki NUKUI. (2005). Intramedullary spinal cord germinoma expresses the protooncogene c-kit. Acta Neurochirurgica. 147(3). 303–308. 11 indexed citations

Watanabe, Arata, Tadashi Mabuchi, Eiji Satoh, et al.. (2005). Expression of syndecans, a heparan sulfate proteoglycan, in malignant gliomas: participation of nuclear factor-κB in upregulation of syndecan-1 expression. Journal of Neuro-Oncology. 77(1). 25–32. 60 indexed citations

Zhang, Lei, Tetsu Yamane, Eiji Satoh, et al.. (2005). Establishment and partial characterization of five malignant glioma cell lines. Neuropathology. 25(2). 136–143. 15 indexed citations

Naganuma, Hirofumi, Eiji Satoh, Takayuki Asahara, et al.. (2004). Quantification of thrombospondin-1 secretion and expression of ?v?3 and ?3?1 integrins and syndecan-1 as cell-surface receptors for thrombospondin-1 in malignant glioma cells. Journal of Neuro-Oncology. 70(3). 309–317. 35 indexed citations

Zhang, Lei, Tadashi Mabuchi, Eiji Satoh, et al.. (2004). Overexpression of Heparin-Binding Growth-Associated Molecule in Malignant Glioma Cells. Neurologia medico-chirurgica. 44(12). 637–645. 9 indexed citations

Naganuma, Hirofumi, et al.. (2004). Solitary Plasmacytoma of the Skull: Immunohistochemical Study of Angiogenic Factors and Syndecan-1-Two Case Reports-. Neurologia medico-chirurgica. 44(4). 195–200. 5 indexed citations

10.

Naganuma, Hirofumi, Eiji Satoh, & Hideaki NUKUI. (2002). Technical Considerations of Transsphenoidal Removal of Fibrous Pituitary Adenomas and Evaluation of Collagen Content and Subtype in the Adenomas.. Neurologia medico-chirurgica. 42(5). 202–213. 63 indexed citations

11.

Sasaki, Atsushi, Hirofumi Naganuma, Eiji Satoh, et al.. (2001). Participation of Thrombospondin-1 in the Activation of Latent Transforming Growth Factor-.BETA. in Malignant Glioma Cells.. Neurologia medico-chirurgica. 41(5). 253–259. 20 indexed citations

12.

Satoh, Eiji, et al.. (2000). [Renal inflammatory pseudotumor after embolization for arteriovenous malformation: a case report].. PubMed. 46(1). 23–6. 4 indexed citations

13.

Naganuma, Hirofumi, et al.. (1998). Down-regulation of transforming growth factor-β and interleukin-10 secretion from malignant glioma cells by cytokines and anticancer drugs. Journal of Neuro-Oncology. 39(3). 229–236. 11 indexed citations

14.

Naganuma, Hirofumi, S Nakano, Atsushi Sasaki, et al.. (1998). Resistance to growth inhibition by transforming growth factor—β in malignant glioma cells with functional receptors. Journal of neurosurgery. 88(3). 529–534. 22 indexed citations

15.

Takano, Y., et al.. (1998). [Study on the duration of pyuria after transurethral resection of prostate].. PubMed. 44(6). 391–6. 3 indexed citations

16.

Satoh, Eiji, et al.. (1997). Effect of irradiation on transforming growth factor-β secretion by malignant glioma cells. Journal of Neuro-Oncology. 33(3). 195–200. 16 indexed citations

17.

Naganuma, Hirofumi, et al.. (1997). Long-term survival in a young patient with anaplastic glioma. Brain Tumor Pathology. 14(1). 71–74. 11 indexed citations

18.

Naganuma, Hirofumi, et al.. (1996). Modulation of Transforming Growth Factor-β Secretion from Malignant Glioma Cells by Interleukin-1β. Neurologia medico-chirurgica. 36(3). 145–150. 6 indexed citations

19.

Sasaki, Atsushi, et al.. (1995). <I>Secretion of Transforming Growth Factor-β1 and -β2 by Malignant Glioma Cells</I>. Neurologia medico-chirurgica. 35(7). 423–430. 39 indexed citations

20.

Naganuma, Hirofumi, Atsushi Sasaki, Eiji Satoh, et al.. (1994). Inhibition of Tumor Necrosis Factor‐α and β Secretion by Lymphokine Activated Killer Cells by Transforming Growth Factor‐β. Japanese Journal of Cancer Research. 85(9). 952–957. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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