Verena Labi

3.3k total citations
50 papers, 2.3k citations indexed

About

Verena Labi is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Verena Labi has authored 50 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 35 papers in Molecular Biology, 26 papers in Immunology and 18 papers in Oncology. Recurrent topics in Verena Labi's work include Cell death mechanisms and regulation (23 papers), Immune Cell Function and Interaction (16 papers) and Cancer-related Molecular Pathways (14 papers). Verena Labi is often cited by papers focused on Cell death mechanisms and regulation (23 papers), Immune Cell Function and Interaction (16 papers) and Cancer-related Molecular Pathways (14 papers). Verena Labi collaborates with scholars based in Austria, Germany and Australia. Verena Labi's co-authors include Andreas Villunger, Miriam Erlacher, Andreas Strasser, Claudia Manzl, Florian Baumgartner, Alexandar Tzankov, Stephan Kießling, Ewa M. Michalak, Alexander Egle and Francesca Grespi and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and The Journal of Experimental Medicine.

In The Last Decade

Verena Labi

49 papers receiving 2.3k citations

Peers

Verena Labi
Tim Hon Man Chan United States
Dexing Fang United States
Richard Gedrich United States
David J. Panka United States
Meike Vogler Germany
Verena Labi
Citations per year, relative to Verena Labi Verena Labi (= 1×) peers Stephanie Grabow

Countries citing papers authored by Verena Labi

Since Specialization
Citations

This map shows the geographic impact of Verena Labi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Verena Labi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Verena Labi more than expected).

Fields of papers citing papers by Verena Labi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Verena Labi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Verena Labi. The network helps show where Verena Labi may publish in the future.

Co-authorship network of co-authors of Verena Labi

This figure shows the co-authorship network connecting the top 25 collaborators of Verena Labi. A scholar is included among the top collaborators of Verena Labi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Verena Labi. Verena Labi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Weiss, Julia Miriam, Irene González-Menéndez, Geoffroy Andrieux, et al.. (2025). PUMA-induced apoptosis drives bone marrow failure and genomic instability in telomerase-deficient mice. Cell Death and Differentiation. 33(1). 38–50.
2.
Hirai, Maretoshi, Michelle S. Levine, Eva Kiermaier, et al.. (2024). Centrioles are frequently amplified in early B cell development but dispensable for humoral immunity. Nature Communications. 15(1). 8890–8890. 2 indexed citations
3.
Schuler, Fabian, Diana C.J. Spierings, Andréa E. Tijhuis, et al.. (2024). Extra centrosomes delay DNA damage–driven tumorigenesis. Science Advances. 10(13). eadk0564–eadk0564. 6 indexed citations
4.
Labi, Verena, et al.. (2023). Age-related neuroimmune signatures in dorsal root ganglia of a Fabry disease mouse model. Immunity & Ageing. 20(1). 22–22. 7 indexed citations
5.
Jukic, Almina, Verena Labi, Tamás Szabó, et al.. (2020). SAFB2 Enables the Processing of Suboptimal Stem-Loop Structures in Clustered Primary miRNA Transcripts. Molecular Cell. 78(5). 876–889.e6. 44 indexed citations
6.
Labi, Verena, Siying Peng, Filippos Klironomos, et al.. (2019). Context-specific regulation of cell survival by a miRNA-controlled BIM rheostat. Genes & Development. 33(23-24). 1673–1687. 12 indexed citations
7.
Labi, Verena, et al.. (2019). miR-17∼92 in lymphocyte development and lymphomagenesis. Cancer Letters. 446. 73–80. 11 indexed citations
8.
Labi, Verena, Rostislav Novak, Renaud Gaujoux, et al.. (2016). A c-Myc/miR17-92/Pten Axis Controls PI3K-Mediated Positive and Negative Selection in B Cell Development and Reconstitutes CD19 Deficiency. Cell Reports. 16(2). 419–431. 40 indexed citations
9.
Labi, Verena & Miriam Erlacher. (2015). How cell death shapes cancer. Cell Death and Disease. 6(3). e1675–e1675. 209 indexed citations
10.
Tuzlak, Selma, et al.. (2015). Combined loss of the BH3-only proteins Bim and Bmf restores B-cell development and function in TACI-Ig transgenic mice. Cell Death and Differentiation. 22(9). 1477–1488. 14 indexed citations
11.
Schweingruber, Nils, Henrike J. Fischer, Lisa Fischer, et al.. (2014). Chemokine-mediated redirection of T cells constitutes a critical mechanism of glucocorticoid therapy in autoimmune CNS responses. Acta Neuropathologica. 127(5). 713–729. 44 indexed citations
12.
Wensveen, Felix M., Christian R. Geest, Sten F.W.M. Libregts, et al.. (2013). BH3-only protein Noxa contributes to apoptotic control of stress-erythropoiesis. APOPTOSIS. 18(11). 1306–1318. 9 indexed citations
13.
Manzl, Claudia, Lukas Peintner, Gerhard Krumschnabel, et al.. (2012). PIDDosome-independent tumor suppression by Caspase-2. Cell Death and Differentiation. 19(10). 1722–1732. 52 indexed citations
14.
Baumgartner, Florian, et al.. (2012). Minor cell-death defects but reduced tumor latency in mice lacking the BH3-only proteins Bad and Bmf. Oncogene. 32(5). 621–630. 14 indexed citations
15.
Labi, Verena & Andreas Villunger. (2010). PUMA-mediated tumor suppression: A tale of two stories. Cell Cycle. 9(21). 4269–4275. 14 indexed citations
16.
Labi, Verena, Miriam Erlacher, Gerhard Krumschnabel, et al.. (2010). Apoptosis of leukocytes triggered by acute DNA damage promotes lymphoma formation. Genes & Development. 24(15). 1602–1607. 91 indexed citations
17.
Manzl, Claudia, Gerhard Krumschnabel, Florian J. Bock, et al.. (2009). Caspase-2 activation in the absence of PIDDosome formation. The Journal of Cell Biology. 185(2). 291–303. 129 indexed citations
18.
Labi, Verena, Miriam Erlacher, Stephan Kießling, et al.. (2008). Loss of the BH3-only protein Bmf impairs B cell homeostasis and accelerates γ irradiation–induced thymic lymphoma development. The Journal of Experimental Medicine. 205(3). 641–655. 98 indexed citations
19.
Labi, Verena, Francesca Grespi, Florian Baumgartner, & Andreas Villunger. (2008). Targeting the Bcl-2-regulated apoptosis pathway by BH3 mimetics: a breakthrough in anticancer therapy?. Cell Death and Differentiation. 15(6). 977–987. 138 indexed citations
20.
Hofbauer, Josefina Piñón, Verena Labi, Alexander Egle, & Andreas Villunger. (2008). Bim and Bmf in tissue homeostasis and malignant disease. Oncogene. 27(S1). S41–S52. 102 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026