Daigo Miyazaki

613 total citations
31 papers, 362 citations indexed

About

Daigo Miyazaki is a scholar working on Molecular Biology, Neurology and Genetics. According to data from OpenAlex, Daigo Miyazaki has authored 31 papers receiving a total of 362 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 12 papers in Neurology and 5 papers in Genetics. Recurrent topics in Daigo Miyazaki's work include Muscle Physiology and Disorders (7 papers), Amyotrophic Lateral Sclerosis Research (4 papers) and Inflammatory Myopathies and Dermatomyositis (3 papers). Daigo Miyazaki is often cited by papers focused on Muscle Physiology and Disorders (7 papers), Amyotrophic Lateral Sclerosis Research (4 papers) and Inflammatory Myopathies and Dermatomyositis (3 papers). Daigo Miyazaki collaborates with scholars based in Japan and Canada. Daigo Miyazaki's co-authors include Akinori Nakamura, Shu‐ichi Ikeda, Shin’ichi Takeda, Kunihiro Yoshida, Kazuhiro Fukushima, Naoko Shiba, Yusuke Echigoya, Toshifumi Yokota, Noboru Fueki and Hitomi Nishizawa and has published in prestigious journals such as Biochemical and Biophysical Research Communications, Human Molecular Genetics and eLife.

In The Last Decade

Daigo Miyazaki

27 papers receiving 357 citations

Peers

Daigo Miyazaki
Jahannaz Dastgir United States
Daigo Miyazaki
Citations per year, relative to Daigo Miyazaki Daigo Miyazaki (= 1×) peers Jahannaz Dastgir

Countries citing papers authored by Daigo Miyazaki

Since Specialization
Citations

This map shows the geographic impact of Daigo Miyazaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daigo Miyazaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daigo Miyazaki more than expected).

Fields of papers citing papers by Daigo Miyazaki

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daigo Miyazaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daigo Miyazaki. The network helps show where Daigo Miyazaki may publish in the future.

Co-authorship network of co-authors of Daigo Miyazaki

This figure shows the co-authorship network connecting the top 25 collaborators of Daigo Miyazaki. A scholar is included among the top collaborators of Daigo Miyazaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daigo Miyazaki. Daigo Miyazaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Shiba, Naoko, Shin Kadota, Masaki Izumi, et al.. (2023). Efficacy of exon-skipping therapy for DMD cardiomyopathy with mutations in actin binding domain 1. Molecular Therapy — Nucleic Acids. 34. 102060–102060.
3.
Shimojima, Yasuhiro, et al.. (2020). Clinical characteristics of patients with myalgia as the initial manifestation of small and medium-sized vasculitis: a retrospective study. Rheumatology International. 40(10). 1667–1674. 9 indexed citations
4.
Shiba, Naoko, Daigo Miyazaki, Yuji Shiba, et al.. (2019). Amelioration of intracellular Ca2+ regulation by exon-45 skipping in Duchenne muscular dystrophy-induced pluripotent stem cell-derived cardiomyocytes. Biochemical and Biophysical Research Communications. 520(1). 179–185. 13 indexed citations
5.
Kodaira, Minori, et al.. (2017). Japanese siblings with woodhouse-sakati syndrome: The first family in East Asia. Journal of the Neurological Sciences. 381. 457–457. 1 indexed citations
6.
Miyazaki, Daigo, et al.. (2016). Increasing Incidence and Age at Onset of Amyotrophic Lateral Sclerosis in Nagano Prefecture, Japan. 64(5). 239–246. 1 indexed citations
7.
Miyazaki, Daigo, Akinori Nakamura, Akiyo Hineno, et al.. (2016). Elevation of serum heat-shock protein levels in amyotrophic lateral sclerosis. Neurological Sciences. 37(8). 1277–1281. 22 indexed citations
8.
Kodaira, Minori, et al.. (2016). Squatting-induced bilateral peroneal nerve palsy in a sewer pipe worker. Occupational Medicine. 67(1). 75–77. 8 indexed citations
9.
Nakamura, Akinori, Naoko Shiba, Daigo Miyazaki, et al.. (2016). Comparison of the phenotypes of patients harboring in-frame deletions starting at exon 45 in the Duchenne muscular dystrophy gene indicates potential for the development of exon skipping therapy. Journal of Human Genetics. 62(4). 459–463. 53 indexed citations
10.
Nakamura, Akinori, Noboru Fueki, Naoko Shiba, et al.. (2016). Deletion of exons 3−9 encompassing a mutational hot spot in the DMD gene presents an asymptomatic phenotype, indicating a target region for multiexon skipping therapy. Journal of Human Genetics. 61(7). 663–667. 50 indexed citations
11.
Kinoshita, Tomomi, Yasuhiro Shimojima, Dai Kishida, et al.. (2016). Hypertrophic Pachymeningitis as an Early Manifestation of Relapsing Polychondritis: Case Report and Review of the Literature. Case Reports in Neurology. 8(3). 211–217. 9 indexed citations
12.
Shiba, Naoko, Daigo Miyazaki, Takahiro Yoshizawa, et al.. (2015). Differential roles of MMP-9 in early and late stages of dystrophic muscles in a mouse model of Duchenne muscular dystrophy. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1852(10). 2170–2182. 22 indexed citations
13.
Nakamura, Akinori, Satoshi Kuru, Akiyo Hineno, et al.. (2014). Slowly progressing lower motor neuron disease caused by a novel duplication mutation in exon 1 of the SOD1 gene. Neurobiology of Aging. 35(10). 2420.e7–2420.e12. 7 indexed citations
14.
Miyazaki, Daigo, Akinori Nakamura, Kazuhiro Fukushima, et al.. (2011). Matrix metalloproteinase-2 ablation in dystrophin-deficient mdx muscles reduces angiogenesis resulting in impaired growth of regenerated muscle fibers. Human Molecular Genetics. 20(9). 1787–1799. 63 indexed citations
15.
Ohta, Kouichi, et al.. (2011). CANCER-ASSOCIATED RETINOPATHY IN A PATIENT WITH SEMINOMA. Retinal Cases & Brief Reports. 6(2). 159–162. 2 indexed citations
16.
Yoshinaga, Tsuneaki, Daigo Miyazaki, Kazuhiro Fukushima, et al.. (2010). Dropped head sign and tongue atrophy in systemic sclerosis-associated myopathy: a case report. Rinsho Shinkeigaku. 50(10). 732–735. 2 indexed citations
17.
Nakamura, Katsuya, Kunihiro Yoshida, Daigo Miyazaki, Hiroshi Morita, & Shu‐ichi Ikeda. (2009). Spinocerebellar ataxia type 6 (SCA6): Clinical pilot trial with gabapentin. Journal of the Neurological Sciences. 278(1-2). 107–111. 17 indexed citations
18.
Miyazaki, Daigo, Kunihiro Yoshida, Kazuhiro Fukushima, et al.. (2009). Characterization of deletion breakpoints in patients with dystrophinopathy carrying a deletion of exons 45–55 of the Duchenne muscular dystrophy (DMD) gene. Journal of Human Genetics. 54(2). 127–130. 18 indexed citations
19.
Miyazaki, Daigo, Masahide Yazaki, Takahisa Gono, et al.. (2008). AH amyloidosis associated with an immunoglobulin heavy chain variable region (VH1) fragment: a case report. Amyloid. 15(2). 125–128. 16 indexed citations
20.
Yazaki, Masahide, Kazuma Kaneko, Kana Tojo, et al.. (2008). An Unusual Case of Klippel-Trenaunay-Weber Syndrome Presenting with Portosystemic Encephalopathy. Internal Medicine. 47(18). 1621–1625. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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