Davi A. G. Mázala

1.1k total citations
21 papers, 556 citations indexed

About

Davi A. G. Mázala is a scholar working on Molecular Biology, Genetics and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Davi A. G. Mázala has authored 21 papers receiving a total of 556 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 7 papers in Genetics and 5 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Davi A. G. Mázala's work include Muscle Physiology and Disorders (16 papers), Neurogenetic and Muscular Disorders Research (7 papers) and Cardiomyopathy and Myosin Studies (4 papers). Davi A. G. Mázala is often cited by papers focused on Muscle Physiology and Disorders (16 papers), Neurogenetic and Muscular Disorders Research (7 papers) and Cardiomyopathy and Myosin Studies (4 papers). Davi A. G. Mázala collaborates with scholars based in United States, France and Canada. Davi A. G. Mázala's co-authors include Eva R. Chin, Jyoti K. Jaiswal, Eleonora Guadagnin, Yiwen Chen, Marshall W. Hogarth, Dapeng Chen, Christopher B. Tully, Terence A. Partridge, James S. Novak and Robert W. Grange and has published in prestigious journals such as The Journal of Cell Biology, The FASEB Journal and International Journal of Molecular Sciences.

In The Last Decade

Davi A. G. Mázala

21 papers receiving 556 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Davi A. G. Mázala United States 15 461 142 77 73 71 21 556
James S. Novak United States 14 599 1.3× 154 1.1× 92 1.2× 68 0.9× 49 0.7× 21 678
Ermelinda Ceco United States 10 479 1.0× 136 1.0× 92 1.2× 58 0.8× 54 0.8× 11 655
Cíntia Yuri Matsumura Brazil 12 398 0.9× 131 0.9× 40 0.5× 57 0.8× 91 1.3× 22 510
Claudia Beyer Australia 7 613 1.3× 198 1.4× 79 1.0× 50 0.7× 101 1.4× 7 736
Pradyut K. Paul United States 12 618 1.3× 221 1.6× 76 1.0× 78 1.1× 126 1.8× 16 795
Kitipong Uaesoontrachoon United States 15 473 1.0× 120 0.8× 85 1.1× 51 0.7× 53 0.7× 25 615
Huibin Tang United States 15 541 1.2× 101 0.7× 51 0.7× 129 1.8× 98 1.4× 23 812
Amalia Stantzou France 7 426 0.9× 131 0.9× 75 1.0× 50 0.7× 82 1.2× 9 490
Adam Hagg Australia 12 436 0.9× 203 1.4× 63 0.8× 50 0.7× 129 1.8× 14 564
Annarita Scaramozza Italy 7 324 0.7× 201 1.4× 97 1.3× 43 0.6× 34 0.5× 9 534

Countries citing papers authored by Davi A. G. Mázala

Since Specialization
Citations

This map shows the geographic impact of Davi A. G. Mázala's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Davi A. G. Mázala with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Davi A. G. Mázala more than expected).

Fields of papers citing papers by Davi A. G. Mázala

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Davi A. G. Mázala. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Davi A. G. Mázala. The network helps show where Davi A. G. Mázala may publish in the future.

Co-authorship network of co-authors of Davi A. G. Mázala

This figure shows the co-authorship network connecting the top 25 collaborators of Davi A. G. Mázala. A scholar is included among the top collaborators of Davi A. G. Mázala based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Davi A. G. Mázala. Davi A. G. Mázala is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mázala, Davi A. G., Dapeng Chen, & Eva R. Chin. (2024). SERCA1 Overexpression in Skeletal Muscle Attenuates Muscle Atrophy and Improves Motor Function in a Mouse Model of ALS. Journal of Neuromuscular Diseases. 11(2). 315–326. 4 indexed citations
2.
Mázala, Davi A. G., Young Jae Moon, Michèle Weiss‐Gayet, et al.. (2023). Altered muscle niche contributes to myogenic deficit in the D2-mdx model of severe DMD. Cell Death Discovery. 9(1). 224–224. 15 indexed citations
3.
Uapinyoying, Prech, Marshall W. Hogarth, Davi A. G. Mázala, et al.. (2023). Single-cell transcriptomic analysis of the identity and function of fibro/adipogenic progenitors in healthy and dystrophic muscle. iScience. 26(8). 107479–107479. 12 indexed citations
4.
Haug, Michael, Stefanie Nübler, Davi A. G. Mázala, et al.. (2022). Absence of the Z-disc protein α-actinin-3 impairs the mechanical stability of Actn3KO mouse fast-twitch muscle fibres without altering their contractile properties or twitch kinetics. Skeletal Muscle. 12(1). 14–14. 5 indexed citations
5.
Uapinyoying, Prech, Marshall W. Hogarth, Davi A. G. Mázala, et al.. (2022). Single Cell Transcriptomic Analysis of the Identity and Function of Fibro/Adipogenic Progenitors in Healthy and Dystrophic Muscle. SSRN Electronic Journal. 1 indexed citations
6.
Novak, James S., Davi A. G. Mázala, Marie Nearing, et al.. (2021). Human muscle stem cells are refractory to aging. Aging Cell. 20(7). e13411–e13411. 19 indexed citations
7.
Chandra, Goutam, Sen Chandra Sreetama, Davi A. G. Mázala, et al.. (2021). Endoplasmic reticulum maintains ion homeostasis required for plasma membrane repair. The Journal of Cell Biology. 220(5). 17 indexed citations
8.
Novak, James S., Utkarsh J. Dang, Alyson A. Fiorillo, et al.. (2021). Interrogation of Dystrophin and Dystroglycan Complex Protein Turnover After Exon Skipping Therapy. Journal of Neuromuscular Diseases. 8(s2). S383–S402. 15 indexed citations
9.
Hogarth, Marshall W., Prech Uapinyoying, Davi A. G. Mázala, & Jyoti K. Jaiswal. (2021). Pathogenic role and therapeutic potential of fibro-adipogenic progenitors in muscle disease. Trends in Molecular Medicine. 28(1). 8–11. 14 indexed citations
10.
Mázala, Davi A. G., James S. Novak, Marshall W. Hogarth, et al.. (2020). TGF-β–driven muscle degeneration and failed regeneration underlie disease onset in a DMD mouse model. JCI Insight. 5(6). 97 indexed citations
11.
Debattisti, Valentina, Adam Horn, R. P. Singh, et al.. (2019). Dysregulation of Mitochondrial Ca2+ Uptake and Sarcolemma Repair Underlie Muscle Weakness and Wasting in Patients and Mice Lacking MICU1. Cell Reports. 29(5). 1274–1286.e6. 74 indexed citations
12.
Michailowsky, Vladimir, Bidyottam Mittra, Shama R. Iyer, et al.. (2019). Defects in sarcolemma repair and skeletal muscle function after injury in a mouse model of Niemann-Pick type A/B disease. Skeletal Muscle. 9(1). 1–1. 18 indexed citations
13.
Heier, Christopher R., Qing Yu, Alyson A. Fiorillo, et al.. (2019). Vamorolone targets dual nuclear receptors to treat inflammation and dystrophic cardiomyopathy. Life Science Alliance. 2(1). e201800186–e201800186. 53 indexed citations
14.
Vila, Maria Candida, James S. Novak, J Boehler, et al.. (2018). NEW THERAPEUTIC APPROACHES AND THEIR READOUT. Neuromuscular Disorders. 28. S90–S90. 1 indexed citations
15.
Guadagnin, Eleonora, Davi A. G. Mázala, & Yiwen Chen. (2018). STAT3 in Skeletal Muscle Function and Disorders. International Journal of Molecular Sciences. 19(8). 2265–2265. 65 indexed citations
16.
Amici, David R., Iago Pinal‐Fernandez, Davi A. G. Mázala, et al.. (2017). Calcium dysregulation, functional calpainopathy, and endoplasmic reticulum stress in sporadic inclusion body myositis. Acta Neuropathologica Communications. 5(1). 24–24. 36 indexed citations
17.
18.
Mázala, Davi A. G., Stephen J. P. Pratt, Dapeng Chen, et al.. (2015). SERCA1 overexpression minimizes skeletal muscle damage in dystrophic mouse models. American Journal of Physiology-Cell Physiology. 308(9). C699–C709. 51 indexed citations
19.
Chin, Eva R., et al.. (2014). Perturbations in intracellular Ca2+ handling in skeletal muscle in the G93A*SOD1 mouse model of amyotrophic lateral sclerosis. American Journal of Physiology-Cell Physiology. 307(11). C1031–C1038. 21 indexed citations
20.
Chen, Dapeng, et al.. (2012). BiP deficiency and ER stress in skeletal muscle of a mouse model of amyotrophic lateral sclerosis. The FASEB Journal. 26(S1). 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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