Dagmar Hildebrand

1.3k total citations
36 papers, 1.1k citations indexed

About

Dagmar Hildebrand is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Dagmar Hildebrand has authored 36 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Immunology, 9 papers in Molecular Biology and 8 papers in Oncology. Recurrent topics in Dagmar Hildebrand's work include Microbial infections and disease research (6 papers), Immune Response and Inflammation (6 papers) and Immune Cell Function and Interaction (4 papers). Dagmar Hildebrand is often cited by papers focused on Microbial infections and disease research (6 papers), Immune Response and Inflammation (6 papers) and Immune Cell Function and Interaction (4 papers). Dagmar Hildebrand collaborates with scholars based in Germany, United States and Israel. Dagmar Hildebrand's co-authors include Klaus Heeg, Margot Zöller, Katharina F. Kubatzky, Jürgen Weitz, Mario Vitacolonna, Rachid Marhaba, Aline Sähr, Moritz Koch, Markus Ladwein and Pamela Klingbeil and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Immunology and Clinical Cancer Research.

In The Last Decade

Dagmar Hildebrand

36 papers receiving 1.1k citations

Peers

Countries citing papers authored by Dagmar Hildebrand

Since Specialization

Citations

This map shows the geographic impact of Dagmar Hildebrand's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dagmar Hildebrand with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dagmar Hildebrand more than expected).

Fields of papers citing papers by Dagmar Hildebrand

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dagmar Hildebrand. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dagmar Hildebrand. The network helps show where Dagmar Hildebrand may publish in the future.

Co-authorship network of co-authors of Dagmar Hildebrand

This figure shows the co-authorship network connecting the top 25 collaborators of Dagmar Hildebrand. A scholar is included among the top collaborators of Dagmar Hildebrand based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dagmar Hildebrand. Dagmar Hildebrand is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Inflammatory Response Against Staphylococcus aureus via Intracellular Sensing of Nucleic Acids in Keratinocytes 2022 ·Frontiers in Immunology ·(unknown), (unknown), Aline Sähr, Dagmar Hildebrand, Tatjana Eigenbrod, Klaus Heeg, Dennis Nurjadi	19
2	Host factors facilitating SARS‐CoV‐2 virus infection and replication in the lungs 2021 ·Cellular and Molecular Life Sciences ·Sébastien Boutin, Dagmar Hildebrand, Steeve Boulant, Michael Kreuter, (unknown), Srinivas Reddy Pallerla, Thirumalaisamy P. Velavan, Dennis Nurjadi	16
3	Pitfalls in genotypic antimicrobial susceptibility testing caused by low expression of blaKPC in Escherichia coli 2021 ·Journal of Antimicrobial Chemotherapy ·(unknown), Sabrina Klein, Dagmar Hildebrand, (unknown), Klaus Heeg, Sébastien Boutin, Dennis Nurjadi	9
4	Silencing SOCS1 via Liposome-Packed siRNA Sustains TLR4-Ligand Adjuvant 2019 ·Frontiers in Immunology ·Dagmar Hildebrand, (unknown), (unknown), Klaus Heeg	9
5	The Interplay of Notch Signaling and STAT3 in TLR-Activated Human Primary Monocytes 2018 ·Frontiers in Cellular and Infection Microbiology ·Dagmar Hildebrand, Florian Uhle, (unknown), (unknown), Markus Weigand, Klaus Heeg	55
6	Hsa-miR-99b/let-7e/miR-125a Cluster Regulates Pathogen Recognition Receptor-Stimulated Suppressive Antigen-Presenting Cells 2018 ·Frontiers in Immunology ·Dagmar Hildebrand, Mariel-Esther Eberle, (unknown), (unknown), (unknown), Aline Sähr, Konrad A. Bode, Klaus Heeg	24
7	Pasteurella multocida Toxin Manipulates T Cell Differentiation 2015 ·Frontiers in Microbiology ·Dagmar Hildebrand, Klaus Heeg, Katharina F. Kubatzky	16
8	Erythropoietin acts as an anti-inflammatory signal on murine mast cells 2015 ·Molecular Immunology ·(unknown), (unknown), (unknown), Dagmar Hildebrand, Sabrina Klein, Klaus Heeg, Katharina F. Kubatzky	10
9	Granzyme A Produces Bioactive IL-1β through a Nonapoptotic Inflammasome-Independent Pathway 2014 ·Cell Reports ·Dagmar Hildebrand, Konrad A. Bode, (unknown), Daniela Cerny, Anna Waldhuber, Franziska Römmler, (unknown), Simone Korten, J. Orth, Thomas Miethke, Klaus Heeg, Katharina F. Kubatzky	36
10	Histone deacetylase inhibitors block IFNγ-induced STAT1 phosphorylation 2012 ·Cellular Signalling ·(unknown), Carolin Bier, Shirley K. Knauer, Kalsoom Sughra, Dagmar Hildebrand, (unknown), (unknown), Regine Heller, Andreas Henke, Roland H. Stauber, W. Reichardt, Johannes A. Schmid, Katharina F. Kubatzky, Thorsten Heinzel, Oliver H. Krämer	39
11	Regulation of Toll-like receptor 4-mediated immune responses through Pasteurella multocida toxin-induced G protein signalling 2012 ·Cell Communication and Signaling ·Dagmar Hildebrand, Aline Sähr, (unknown), Klaus Heeg, Katharina F. Kubatzky	24
12	Pasteurella multocida Toxin-induced Pim-1 expression disrupts suppressor of cytokine signalling (SOCS)-1 activity 2010 ·Cellular Microbiology ·Dagmar Hildebrand, (unknown), Alexander H. Dalpke, Klaus Heeg, Katharina F. Kubatzky	19
13	The haematopoietic GTPase RhoH modulates IL3 signalling through regulation of STAT activity and IL3 receptor expression 2010 ·Molecular Cancer ·(unknown), He Liu, (unknown), Dagmar Hildebrand, Michael Aigner, Klaus Aktories, Klaus Heeg, Katharina F. Kubatzky	22
14	Pasteurella multocida toxin is a potent activator of anti-apoptotic signalling pathways 2010 ·Cellular Microbiology ·Inga Preuß, Dagmar Hildebrand, J. Orth, Klaus Aktories, Katharina F. Kubatzky	34
15	Tumorigenicity of IL-1α– and IL-1β–Deficient Fibrosarcoma Cells 2008 ·Neoplasia ·Irina Nazarenko, Rachid Marhaba, Eli Reich, Elena Voronov, Mario Vitacolonna, Dagmar Hildebrand, (unknown), (unknown), Ron N. Apte, Margot Zöller	25
16	A Complex of EpCAM, Claudin-7, CD44 Variant Isoforms, and Tetraspanins Promotes Colorectal Cancer Progression 2007 ·Molecular Cancer Research ·Sebastian Kühn, Moritz Koch, Tobias Nübel, Markus Ladwein, Dalibor Antolovic, Pamela Klingbeil, Dagmar Hildebrand, Gerhard Moldenhauer, Lutz Langbein, Werner W. Franke, Jürgen Weitz, Margot Zöller	213
17	The Importance of Myeloid-Derived Suppressor Cells in the Regulation of Autoimmune Effector Cells by a Chronic Contact Eczema 2007 ·The Journal of Immunology ·Rachid Marhaba, Mario Vitacolonna, Dagmar Hildebrand, Michal Baniyash, Pia Freyschmidt‐Paul, Margot Zöller	68
18	T Cell Responses in Mammalian Diaphanous-related Formin mDia1 Knock-out Mice 2007 ·Journal of Biological Chemistry ·Kathryn M. Eisenmann, Richard West, Dagmar Hildebrand, (unknown), Jun Peng, Robert E. Sigler, Jinyi Zhang, Katherine A. Siminovitch, Arthur S. Alberts	103
19	C4.4A as a candidate marker in the diagnosis of colorectal cancer 2007 ·British Journal of Cancer ·Claudia Paret, Dagmar Hildebrand, Jürgen Weitz, Annette Kopp‐Schneider, Annegret Kuhn, A.-M. Beer, Richard E. Hautmann, Margot Zöller	32
20	Colocalization of the Tetraspanins, CO-029 and CD151, with Integrins in Human Pancreatic Adenocarcinoma: Impact on Cell Motility 2005 ·Clinical Cancer Research ·Sabine Gesierich, Claudia Paret, Dagmar Hildebrand, Jürgen Weitz, Kaspar Z’graggen, Friedrich H. Schmitz-Winnenthal, Václav Hořejšı́, Osamu Yoshie, Dorothee Herlyn, Leonie K. Ashman, Margot Zöller	96

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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