Curtis T. Okamoto
- Molecular Biology top 5%
- Cell Biology top 2%
- Physiology top 5%
- Oncology top 10%
- Surgery top 10%
- Co-authors
- Sarah F. Hamm‐AlvarezJohn G. ForteShi XuBogdan OlenyukKeith E. MostovBenjamin AroetiGerard ApodacaYoung Y. Jeng
- Topics
- Cellular transport and secretion (27 papers)Glycosylation and Glycoproteins Research (15 papers)Ocular Surface and Contact Lens (14 papers)
- Partner nations
- United StatesGermanyNorway
In The Last Decade
Curtis T. Okamoto
77 papers receiving 2.7k citations
Peers
Comparison fields: 5 of 120
- Molecular Biology 1.5k
- Cell Biology 690
- Physiology 374
- Oncology 304
- Surgery 296
Countries citing papers authored by Curtis T. Okamoto
This map shows the geographic impact of Curtis T. Okamoto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Curtis T. Okamoto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Curtis T. Okamoto more than expected).
Fields of papers citing papers by Curtis T. Okamoto
This network shows the impact of papers produced by Curtis T. Okamoto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Curtis T. Okamoto. The network helps show where Curtis T. Okamoto may publish in the future.
Co-authorship network of co-authors of Curtis T. Okamoto
This figure shows the co-authorship network connecting the top 25 collaborators of Curtis T. Okamoto. A scholar is included among the top collaborators of Curtis T. Okamoto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Curtis T. Okamoto. Curtis T. Okamoto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 2 | |
| 3 | 2 | |
| 4 | 8 | |
| 5 | 1 | |
| 6 | Tear Proteins as Possible Biomarkers for Parkinson’s Disease | 2 |
| 7 | 4 | |
| 8 | 9 | |
| 9 | 11 | |
| 10 | 58 | |
| 11 | 21 | |
| 12 | 42 | |
| 13 | 35 | |
| 14 | 38 | |
| 15 | 22 | |
| 16 | 4 | |
| 17 | 72 | |
| 18 | 23 | |
| 19 | 10 | |
| 20 | The teleost cone cytoskeleton. Localization of actin, microtubules, and intermediate filaments. | 53 |
About Curtis T. Okamoto
Curtis T. Okamoto is a scholar working on Cell Biology, Immunology and Allergy and Molecular Biology, having authored 77 papers that have together received 2.7k indexed citations. Recurring topics across this work include Cellular transport and secretion (27 papers), Glycosylation and Glycoproteins Research (15 papers) and Ocular Surface and Contact Lens (14 papers). The work is most often cited by research in Cell Biology (690 citations), Immunology and Allergy (142 citations) and Molecular Biology (1.5k citations). Curtis T. Okamoto has collaborated with scholars based in United States, Germany and Norway. Frequent co-authors include Sarah F. Hamm‐Alvarez, John G. Forte, Shi Xu, Bogdan Olenyuk, Keith E. Mostov, Benjamin Aroeti, Gerard Apodaca, Young Y. Jeng, Austin K. Mircheff and Richard L. Wood. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Cell Biology and Gastroenterology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.