Ke Tang
- Cancer Research top 1%
- Cancer, Hypoxia, and Metabolism 10
- Cancer-related molecular mechanisms research 7
- Immunology top 1%
- Immune Cell Function and Interaction 13
- Immune cells in cancer 13
- Immunotherapy and Immune Responses 7
- Molecular Biology top 2%
- Extracellular vesicles in disease 12
- RNA Interference and Gene Delivery 8
- RNA modifications and cancer 8
- Oncology top 2%
- Cell Biology top 5%
- Co-authors
- Jingwei MaBo HuangPingwei XuJiadi LvYuying LiuHuafeng ZhangXiaoyu LiangJing Xie
- Journals
- Science (1 paper)Proceedings of the National Academy of Sciences (1 paper)Journal of Clinical Investigation (1 paper)
- Partner nations
- ChinaUnited StatesCanada
In The Last Decade
Ke Tang
101 papers receiving 5.4k citations
Hit Papers
Peers
Comparison fields: 5 of 136
- Cancer Research 1.3k
- Immunology 1.6k
- Molecular Biology 2.9k
- Oncology 1.1k
- Cell Biology 421
Countries citing papers authored by Ke Tang
This map shows the geographic impact of Ke Tang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ke Tang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ke Tang more than expected).
Fields of papers citing papers by Ke Tang
This network shows the impact of papers produced by Ke Tang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ke Tang. The network helps show where Ke Tang may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Ke Tang, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 2 | |
| 2 | 2024 | 0 | |
| 3 | 2024 | 28 | |
| 4 | 2023 | 20 | |
| 5 | 2022 | 29 | |
| 6 | 2022 | 45 | |
| 7 | 2021 | 75 | |
| 8 | 2021 | 2 | |
| 9 | 2020 | 43 | |
| 10 | 2020 | 88 | |
| 11 | Exosomal CircGDI2 Suppresses Oral Squamous Cell Carcinoma Progression Through the Regulation of MiR-424-5p/SCAI Axis | 2020 | 1 |
| 12 | 2019 | 189 | |
| 13 | 2019 | 24 | |
| 14 | 2018 | 88 | |
| 15 | 2018 | 64 | |
| 16 | 2018 | 48 | |
| 17 | 2015 | 68 | |
| 18 | 2014 | 125 | |
| 19 | 2012 | 384 | |
| 20 | 2002 | 1 |
About Ke Tang
Ke Tang is a scholar working on Immunology, Cancer Research and Biological Psychiatry, having authored 105 papers that have together received 5.5k indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (13 papers), Immune cells in cancer (13 papers), Extracellular vesicles in disease (12 papers), Cancer, Hypoxia, and Metabolism (10 papers), RNA Interference and Gene Delivery (8 papers), RNA modifications and cancer (8 papers), Immunotherapy and Immune Responses (7 papers) and Cancer-related molecular mechanisms research (7 papers). The work is most often cited by research in Cancer Research (1.3k citations), Immunology (1.6k citations) and Molecular Biology (2.9k citations). Ke Tang has collaborated with scholars based in China, United States and Canada. Frequent co-authors include Jingwei Ma, Bo Huang, Pingwei Xu, Bo Huang, Jiadi Lv, Yuying Liu, Huafeng Zhang, Xiaoyu Liang, Huafeng Zhang and Jing Xie. Their work appears in journals such as Science, Proceedings of the National Academy of Sciences and Journal of Clinical Investigation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.