Corrie J.B. daCosta
- Molecular Biology top 10%
- Cellular and Molecular Neuroscience top 10%
- Pharmacology
- Spectroscopy
- Insect Science top 10%
- Co-authors
- John E. BaenzigerSteven M. SineJ. P. Daniel TherienMichael BlantonCasey L. CarswellJeremías CorradiCecilia BouzatShuzhi Wang
- Topics
- Nicotinic Acetylcholine Receptors Study (25 papers)Receptor Mechanisms and Signaling (20 papers)Ion channel regulation and function (16 papers)
- Partner nations
- CanadaUnited StatesAustralia
In The Last Decade
Corrie J.B. daCosta
27 papers receiving 866 citations
Peers
Comparison fields: 5 of 67
- Molecular Biology 830
- Cellular and Molecular Neuroscience 260
- Pharmacology 77
- Spectroscopy 52
- Insect Science 50
Countries citing papers authored by Corrie J.B. daCosta
This map shows the geographic impact of Corrie J.B. daCosta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Corrie J.B. daCosta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Corrie J.B. daCosta more than expected).
Fields of papers citing papers by Corrie J.B. daCosta
This network shows the impact of papers produced by Corrie J.B. daCosta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Corrie J.B. daCosta. The network helps show where Corrie J.B. daCosta may publish in the future.
Co-authorship network of co-authors of Corrie J.B. daCosta
This figure shows the co-authorship network connecting the top 25 collaborators of Corrie J.B. daCosta. A scholar is included among the top collaborators of Corrie J.B. daCosta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Corrie J.B. daCosta. Corrie J.B. daCosta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 2 | |
| 3 | 0 | |
| 4 | 3 | |
| 5 | 6 | |
| 6 | 0 | |
| 7 | 12 | |
| 8 | 7 | |
| 9 | 38 | |
| 10 | 23 | |
| 11 | 81 | |
| 12 | 19 | |
| 13 | 7 | |
| 14 | 63 | |
| 15 | 14 | |
| 16 | 57 | |
| 17 | 53 | |
| 18 | 21 | |
| 19 | 104 | |
| 20 | 48 |
About Corrie J.B. daCosta
Corrie J.B. daCosta is a scholar working on Aging, Molecular Biology and Cellular and Molecular Neuroscience, having authored 29 papers that have together received 875 indexed citations. Recurring topics across this work include Nicotinic Acetylcholine Receptors Study (25 papers), Receptor Mechanisms and Signaling (20 papers) and Ion channel regulation and function (16 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (260 citations), Molecular Biology (830 citations) and Pharmacology (77 citations). Corrie J.B. daCosta has collaborated with scholars based in Canada, United States and Australia. Frequent co-authors include John E. Baenziger, Steven M. Sine, J. P. Daniel Therien, Michael Blanton, Casey L. Carswell, Jeremías Corradi, Cecilia Bouzat, Shuzhi Wang, Nuriya Mukhtasimova and Christian Tessier. Their work appears in journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.