Clive Bate

4.0k total citations
96 papers, 3.3k citations indexed

About

Clive Bate is a scholar working on Molecular Biology, Physiology and Neurology. According to data from OpenAlex, Clive Bate has authored 96 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 53 papers in Molecular Biology, 49 papers in Physiology and 37 papers in Neurology. Recurrent topics in Clive Bate's work include Prion Diseases and Protein Misfolding (48 papers), Alzheimer's disease research and treatments (46 papers) and Malaria Research and Control (25 papers). Clive Bate is often cited by papers focused on Prion Diseases and Protein Misfolding (48 papers), Alzheimer's disease research and treatments (46 papers) and Malaria Research and Control (25 papers). Clive Bate collaborates with scholars based in United Kingdom, Italy and Australia. Clive Bate's co-authors include Steven Williams, Alun Williams, Janice Taverne, J. H. L. Playfair, Dominic Kwiatkowski, Mario Salmona, Mourad Tayebi, Piet Eikelenboom, Robert Veerhuis and Sarah Kempster and has published in prestigious journals such as Journal of Biological Chemistry, SHILAP Revista de lepidopterología and PLoS ONE.

In The Last Decade

Clive Bate

96 papers receiving 3.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Clive Bate United Kingdom 33 1.3k 958 937 817 570 96 3.3k
Janetta G. Culvenor Australia 41 1.5k 1.1× 1.0k 1.1× 1.5k 1.6× 455 0.6× 559 1.0× 83 5.0k
William B. Cowden Australia 39 1.2k 0.9× 1.9k 2.0× 570 0.6× 504 0.6× 2.1k 3.7× 96 5.7k
Tanweer Zaidi United States 34 3.1k 2.3× 472 0.5× 2.9k 3.1× 488 0.6× 393 0.7× 61 6.2k
Cameron R. Stewart Australia 22 2.4k 1.8× 178 0.2× 806 0.9× 865 1.1× 1.8k 3.2× 40 4.6k
Yvonne Kress United States 39 2.1k 1.6× 182 0.2× 2.5k 2.6× 914 1.1× 646 1.1× 88 6.5k
Rhonda Kean United States 24 717 0.5× 165 0.2× 452 0.5× 398 0.5× 443 0.8× 38 2.7k
Damien Arnoult France 38 4.4k 3.3× 377 0.4× 623 0.7× 135 0.2× 1.7k 3.0× 72 6.6k
Sergei Spitsin United States 27 1.2k 0.9× 143 0.1× 479 0.5× 375 0.5× 532 0.9× 54 3.4k
Denise Galatis Australia 22 865 0.6× 854 0.9× 1.0k 1.1× 104 0.1× 55 0.1× 27 2.6k
Regine Heller Germany 34 1.2k 0.9× 125 0.1× 924 1.0× 135 0.2× 744 1.3× 84 4.1k

Countries citing papers authored by Clive Bate

Since Specialization
Citations

This map shows the geographic impact of Clive Bate's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Clive Bate with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Clive Bate more than expected).

Fields of papers citing papers by Clive Bate

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Clive Bate. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Clive Bate. The network helps show where Clive Bate may publish in the future.

Co-authorship network of co-authors of Clive Bate

This figure shows the co-authorship network connecting the top 25 collaborators of Clive Bate. A scholar is included among the top collaborators of Clive Bate based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Clive Bate. Clive Bate is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Puig, Berta, Hermann C. Altmeppen, Luise Linsenmeier, et al.. (2019). GPI-anchor signal sequence influences PrPC sorting, shedding and signalling, and impacts on different pathomechanistic aspects of prion disease in mice. PLoS Pathogens. 15(1). e1007520–e1007520. 31 indexed citations
2.
Bate, Clive. (2017). Can we switch production of toxic Aβ oligomers to neuroprotective Aβ monomers to allow synapse regeneration?. Neural Regeneration Research. 12(9). 1437–1437. 1 indexed citations
3.
Bate, Clive, et al.. (2016). Sialic Acid within the Glycosylphosphatidylinositol Anchor Targets the Cellular Prion Protein to Synapses. Journal of Biological Chemistry. 291(33). 17093–17101. 14 indexed citations
4.
5.
Bate, Clive & Alun Williams. (2011). Ethanol protects cultured neurons against amyloid-β and α-synuclein-induced synapse damage. Neuropharmacology. 61(8). 1406–1412. 28 indexed citations
6.
Bate, Clive & Steven Williams. (2011). Monoacylated Cellular Prion Protein Modifies Cell Membranes, Inhibits Cell Signaling, and Reduces Prion Formation. Journal of Biological Chemistry. 286(11). 8752–8758. 21 indexed citations
7.
Jones, Daryl, William A. Taylor, Clive Bate, Monique David, & Mourad Tayebi. (2010). A Camelid Anti-PrP Antibody Abrogates PrPSc Replication in Prion-Permissive Neuroblastoma Cell Lines. PLoS ONE. 5(3). e9804–e9804. 33 indexed citations
8.
Bate, Clive, Mourad Tayebi, & Alun Williams. (2010). Phospholipase A2 inhibitors protect against prion and Aβ mediated synapse degeneration. Molecular Neurodegeneration. 5(1). 13–13. 33 indexed citations
9.
Bate, Clive, Mourad Tayebi, Mario Salmona, Luisa Diomede, & Steven Williams. (2009). Polyunsaturated Fatty Acids Protect Against Prion-Mediated Synapse Damage In Vitro. Neurotoxicity Research. 17(3). 203–214. 12 indexed citations
10.
Bate, Clive, Mourad Tayebi, & Steven Williams. (2008). Sequestration of free cholesterol in cell membranes by prions correlates with cytoplasmic phospholipase A2activation. BMC Biology. 6(1). 8–8. 26 indexed citations
11.
Bate, Clive, et al.. (2007). Squalestatin alters the intracellular trafficking of a neurotoxic prion peptide. BMC Neuroscience. 8(1). 99–99. 11 indexed citations
12.
Bate, Clive, et al.. (2006). Interferon-gamma increases neuronal death in response to amyloid-beta1-42.. Journal of Neuroinflammation. 3(1). 7–7. 56 indexed citations
13.
Bate, Clive, Jan Langeveld, & Steven Williams. (2004). Manipulation of PrPres production in scrapie-infected neuroblastoma cells. Journal of Neuroscience Methods. 138(1-2). 217–223. 17 indexed citations
14.
Bate, Clive, Mario Salmona, & Steven Williams. (2004). The role of platelet activating factor in prion and amyloid-β neurotoxicity. Neuroreport. 15(3). 509–513. 34 indexed citations
15.
Eikelenboom, Piet, Clive Bate, Willem A. van Gool, et al.. (2002). Neuroinflammation in Alzheimer's disease and prion disease. Glia. 40(2). 232–239. 328 indexed citations
16.
Bate, Clive, et al.. (2002). Cyclo-oxygenase inhibitors protect against prion-induced neurotoxicity in vitro. Neuroreport. 13(15). 1933–1938. 23 indexed citations
17.
Jakobsen, Palle, Clive Bate, Janice Taverne, & J. H. L. Playfair. (1995). Malaria: toxins, cytokines and disease. Parasite Immunology. 17(5). 223–231. 61 indexed citations
18.
Bate, Clive, et al.. (1990). Malaria exoantigens induce T-independent antibody that blocks their ability to induce TNF.. PubMed Central. 70(3). 315–20. 52 indexed citations
19.
Playfair, J. H. L., Janice Taverne, Clive Bate, & J. Brian de Souza. (1990). The malaria vaccine: anti-parasite or anti-disease?. Immunology Today. 11(1). 25–27. 139 indexed citations
20.
Schanfield, Moses S., et al.. (1975). Immunogenetic factors in thalassemia and hepatitis B infection. A multicentre study.. PubMed. 30. 257–69. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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