Claude Szpirer

7.0k citations
190 papers · 5.3k indexed · h-index 39

Claude Szpirer

189 papers receiving 5.2k citations

Peers

Claude Szpirer
Comparison fields: 5 of 131
  • Genetics 1.8k
  • Molecular Biology 3.0k
  • Endocrinology, Diabetes and Metabolism 661
  • Reproductive Medicine 296
  • Cancer Research 362
Replace Fumi Tashiro with:
Fumi Tashiro Japan
A Kahn France
Yukiko Kurihara Japan
Kimi Araki Japan
Kunio Shiota Japan
Andreas Hoeflich Germany
John W.M. Creemers Belgium
George E.O. Muscat Australia
Alexandra Belayew Belgium
Mona Nemer Canada
Claude Szpirer relative to Fumi Tashiro Japan Fumi Tashiro's profile →
Citations per field
00.5×1.5×1.9×
Fumi Tashiro · 1×
Citations per year

Countries citing papers authored by Claude Szpirer

Since Specialization
Citations

This map shows the geographic impact of Claude Szpirer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Claude Szpirer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Claude Szpirer more than expected).

Fields of papers citing papers by Claude Szpirer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Claude Szpirer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Claude Szpirer. The network helps show where Claude Szpirer may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Claude Szpirer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Claude Szpirer Line = papers co-authored together Claude Szpirer links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 200854
2 200610
3 20042
4 200332
5 20021
6 20012
7 200127
8 199810
9 199633
10 199632
11 199621
12 199618
13 1995143
14 19951
15 19935
16
Chromosomal assignment of five cancer-associated rat genes: two thyroid hormone receptor (ERBA) genes, two ERBB genes and the retinoblastoma gene.
199113
17 199150
18 19913
19 198840
20 1984105

About Claude Szpirer

Claude Szpirer is a scholar working on Genetics, Molecular Biology and Clinical Biochemistry, having authored 190 papers that have together received 5.3k indexed citations. Recurring topics across this work include Genetic Mapping and Diversity in Plants and Animals (23 papers), Genomics and Chromatin Dynamics (20 papers), RNA Research and Splicing (19 papers), RNA modifications and cancer (14 papers), Epigenetics and DNA Methylation (13 papers), Growth Hormone and Insulin-like Growth Factors (10 papers), Pancreatic function and diabetes (10 papers) and RNA and protein synthesis mechanisms (9 papers). The work is most often cited by research in Genetics (1.8k citations), Molecular Biology (3.0k citations) and Endocrinology, Diabetes and Metabolism (661 citations). Claude Szpirer has collaborated with scholars based in Belgium, Sweden and United States. Frequent co-authors include Josiane Szpirer, G. Levan, Göran Levan, Josiane Szpirer, M.Q. Islam, M. Rivière, Christelle De Mees, Vladimı́r Křen, Michal Pravenec and Julie Bakker. Their work appears in journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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