Karin Klinga‐Levan

958 total citations
54 papers, 768 citations indexed

About

Karin Klinga‐Levan is a scholar working on Molecular Biology, Genetics and Cancer Research. According to data from OpenAlex, Karin Klinga‐Levan has authored 54 papers receiving a total of 768 indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Molecular Biology, 20 papers in Genetics and 14 papers in Cancer Research. Recurrent topics in Karin Klinga‐Levan's work include RNA Research and Splicing (15 papers), MicroRNA in disease regulation (10 papers) and Genomics and Chromatin Dynamics (8 papers). Karin Klinga‐Levan is often cited by papers focused on RNA Research and Splicing (15 papers), MicroRNA in disease regulation (10 papers) and Genomics and Chromatin Dynamics (8 papers). Karin Klinga‐Levan collaborates with scholars based in Sweden, Belgium and United States. Karin Klinga‐Levan's co-authors include Björn Olsson, G. Levan, Gisela Helenius, Jessica Carlsson, Mats G. Karlsson, Claude Szpirer, Gunnar C. Hansson, Khalil Helou, H. Stålhammar and I Carlstedt and has published in prestigious journals such as Journal of Biological Chemistry, Oncogene and Biochemical and Biophysical Research Communications.

In The Last Decade

Karin Klinga‐Levan

52 papers receiving 733 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Karin Klinga‐Levan Sweden 17 531 241 211 133 71 54 768
Marc Hirschfeld Germany 19 656 1.2× 435 1.8× 41 0.2× 112 0.8× 89 1.3× 45 905
Qi Shen China 17 707 1.3× 534 2.2× 60 0.3× 113 0.8× 144 2.0× 41 1.1k
Nupur Mukherjee India 17 415 0.8× 154 0.6× 46 0.2× 202 1.5× 32 0.5× 33 689
Lisa K. Mullany United States 16 342 0.6× 135 0.6× 68 0.3× 159 1.2× 134 1.9× 18 633
Umarani Pugazhenthi United States 13 557 1.0× 122 0.5× 154 0.7× 253 1.9× 28 0.4× 23 958
Ding-Yen Lin Taiwan 11 702 1.3× 115 0.5× 138 0.7× 170 1.3× 27 0.4× 15 854
Elena Alexandrova Italy 17 455 0.9× 137 0.6× 114 0.5× 95 0.7× 19 0.3× 47 669
Bing Sun China 14 311 0.6× 103 0.4× 57 0.3× 59 0.4× 120 1.7× 51 639
Ying-Fei Wei United States 8 860 1.6× 288 1.2× 78 0.4× 198 1.5× 11 0.2× 8 1.1k

Countries citing papers authored by Karin Klinga‐Levan

Since Specialization
Citations

This map shows the geographic impact of Karin Klinga‐Levan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Karin Klinga‐Levan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Karin Klinga‐Levan more than expected).

Fields of papers citing papers by Karin Klinga‐Levan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Karin Klinga‐Levan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Karin Klinga‐Levan. The network helps show where Karin Klinga‐Levan may publish in the future.

Co-authorship network of co-authors of Karin Klinga‐Levan

This figure shows the co-authorship network connecting the top 25 collaborators of Karin Klinga‐Levan. A scholar is included among the top collaborators of Karin Klinga‐Levan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Karin Klinga‐Levan. Karin Klinga‐Levan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Klinga‐Levan, Karin, et al.. (2016). Verification of microRNA expression in human endometrial adenocarcinoma. BMC Cancer. 16(1). 261–261. 24 indexed citations
2.
Olsson, Björn, et al.. (2013). Validation of suitable endogenous control genes for quantitative PCR analysis of microRNA gene expression in a rat model of endometrial cancer. Cancer Cell International. 13(1). 45–45. 12 indexed citations
3.
Behboudi, Afrouz, et al.. (2012). The impact of the genetic background on the genome make‐up of tumor cells. Genes Chromosomes and Cancer. 51(5). 438–446. 2 indexed citations
4.
Hedberg, Carola, et al.. (2011). SKY analysis revealed recurrent numerical and structural chromosome changes in BDII rat endometrial carcinomas. Cancer Cell International. 11(1). 20–20. 3 indexed citations
5.
Carlsson, Jessica, Sabina Davidsson, Gisela Helenius, et al.. (2011). A miRNA expression signature that separates between normal and malignant prostate tissues. Cancer Cell International. 11(1). 14–14. 44 indexed citations
6.
Carlsson, Jessica, et al.. (2010). Loss of glutathione peroxidase 3 expression is correlated with epigenetic mechanisms in endometrial adenocarcinoma. Cancer Cell International. 10(1). 46–46. 48 indexed citations
7.
Carlsson, Jessica, Gisela Helenius, Mats G. Karlsson, et al.. (2010). Validation of suitable endogenous control genes for expression studies of miRNA in prostate cancer tissues. Cancer Genetics and Cytogenetics. 202(2). 71–75. 28 indexed citations
8.
Adamović, Tatjana, et al.. (2008). Rearrangement and allelic imbalance on chromosome 5 leads to homozygous deletions in the CDKN2A/2B tumor suppressor gene region in rat endometrial cancer. Cancer Genetics and Cytogenetics. 184(1). 9–21. 6 indexed citations
9.
Mallon, Patrick, Dirk Wedekind, Josiane Szpirer, et al.. (2005). Genetic analysis of susceptibility to endometrial adenocarcinoma in the BDII rat model. Cancer Genetics and Cytogenetics. 158(2). 137–141. 21 indexed citations
10.
Klinga‐Levan, Karin, Agneta Andersson, Charles Hanson, et al.. (2004). Mapping of Glutathione Transferase (GST) Genes in the Rat. Hereditas. 119(3). 285–296. 2 indexed citations
11.
Olson, Fredrik J., Malin Johansson, Karin Klinga‐Levan, et al.. (2002). Blood Group A Glycosyltransferase Occurring as Alleles with High Sequence Difference Is Transiently Induced during aNippostrongylus brasiliensis Parasite Infection. Journal of Biological Chemistry. 277(17). 15044–15052. 20 indexed citations
12.
Helou, Khalil, Anna Walentinsson, Åsa Johansson, et al.. (2001). Analysis of genetic changes in rat endometrial carcinomas by means of comparative genomic hybridization. Cancer Genetics and Cytogenetics. 127(2). 118–127. 27 indexed citations
14.
Szpirer, Claude, Josiane Szpirer, P. van Vooren, et al.. (1999). Gene-based anchoring of the rat genetic linkage and cytogenetic maps. Transplantation Proceedings. 31(3). 1541–1543. 2 indexed citations
15.
Helou, Khalil, Ville Wallenius, Yan Qiu, et al.. (1999). Amplification and overexpression of the hepatocyte growth factor receptor (HGFR/MET) in rat DMBA sarcomas. Oncogene. 18(21). 3226–3234. 19 indexed citations
17.
Szpirer, Claude, Josiane Szpirer, P. van Vooren, et al.. (1998). Gene-based anchoring of the rat genetic linkage and cytogenetic maps: new regional localizations, orientation of the linkage groups, and insights into mammalian chromosome evolution. Mammalian Genome. 9(9). 721–734. 43 indexed citations
18.
Sjöling, Åsa, Karin Klinga‐Levan, & G. Levan. (1996). Sublocalization of the rat brown gene (B=Tyrp1) by linkage mapping. Mammalian Genome. 7(9). 710–711. 2 indexed citations
20.
Kobayashi, Toshihide, Tatsuya Kishino, Naomichi Matsumoto, et al.. (1995). Isolation of microdissection clones from rat Chromosome 10. Mammalian Genome. 6(3). 216–218. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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