Clara L. Oeste
- Cell Biology top 10%
- Cellular transport and secretion 3
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- Immune Cell Function and Interaction 8
- T-cell and B-cell Immunology 8
- Immunotherapy and Immune Responses 6
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- PI3K/AKT/mTOR signaling in cancer 3
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- Calcium signaling and nucleotide metabolism 4
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- Inflammatory mediators and NSAID effects 3
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- Cancer-related Molecular Pathways 3
- Co-authors
- Dolores Pérez‐SalaIrene RamosKonstantinos StamatakisAlma E. MartínezF. Javier CañadaBalbino AlarcónBeatriz Díez-DacalPatricia Boya
- Cited by
- Cell BiologyImmunologyBiochemistry
- Journals
- Nature Communications (2 papers)The Journal of Experimental Medicine (1 paper)PLoS ONE (2 papers)
- Partner nations
- SpainUnited StatesUnited Kingdom
In The Last Decade
Clara L. Oeste
26 papers receiving 660 citations
Peers
Comparison fields: 5 of 84
- Cell Biology 158
- Immunology 139
- Biochemistry 36
- Molecular Biology 361
- Biochemistry 29
Countries citing papers authored by Clara L. Oeste
This map shows the geographic impact of Clara L. Oeste's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Clara L. Oeste with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Clara L. Oeste more than expected).
Fields of papers citing papers by Clara L. Oeste
This network shows the impact of papers produced by Clara L. Oeste. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Clara L. Oeste. The network helps show where Clara L. Oeste may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Clara L. Oeste, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 1 | |
| 2 | 2025 | 2 | |
| 3 | 2025 | 0 | |
| 4 | 2025 | 1 | |
| 5 | 2024 | 8 | |
| 6 | 2023 | 2 | |
| 7 | 2023 | 4 | |
| 8 | 2022 | 19 | |
| 9 | 2021 | 19 | |
| 10 | 2020 | 121 | |
| 11 | 2018 | 26 | |
| 12 | 2018 | 22 | |
| 13 | 2017 | 1 | |
| 14 | 2015 | 66 | |
| 15 | 2014 | 13 | |
| 16 | 2012 | 16 | |
| 17 | 2012 | 49 | |
| 18 | 2011 | 33 | |
| 19 | 2011 | 29 | |
| 20 | 2010 | 16 |
About Clara L. Oeste
Clara L. Oeste is a scholar working on Physiology, Immunology and Biochemistry, having authored 27 papers that have together received 666 indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (8 papers), T-cell and B-cell Immunology (8 papers), Immunotherapy and Immune Responses (6 papers), Calcium signaling and nucleotide metabolism (4 papers), Cellular transport and secretion (3 papers), PI3K/AKT/mTOR signaling in cancer (3 papers), Inflammatory mediators and NSAID effects (3 papers) and Cancer-related Molecular Pathways (3 papers). The work is most often cited by research in Cell Biology (158 citations), Immunology (139 citations) and Biochemistry (36 citations). Clara L. Oeste has collaborated with scholars based in Spain, United States and United Kingdom. Frequent co-authors include Dolores Pérez‐Sala, Irene Ramos, Konstantinos Stamatakis, Alma E. Martínez, F. Javier Cañada, Balbino Alarcón, Beatriz Díez-Dacal, Patricia Boya, Francisco J. Sánchez-Gómez and Wayne F. Patton. Their work appears in journals such as Nature Communications, The Journal of Experimental Medicine and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.