Christoph Höck

13.7k total citations · 3 hit papers
144 papers, 7.9k citations indexed

About

Christoph Höck is a scholar working on Physiology, Molecular Biology and Psychiatry and Mental health. According to data from OpenAlex, Christoph Höck has authored 144 papers receiving a total of 7.9k indexed citations (citations by other indexed papers that have themselves been cited), including 79 papers in Physiology, 43 papers in Molecular Biology and 39 papers in Psychiatry and Mental health. Recurrent topics in Christoph Höck's work include Alzheimer's disease research and treatments (77 papers), Dementia and Cognitive Impairment Research (36 papers) and Functional Brain Connectivity Studies (19 papers). Christoph Höck is often cited by papers focused on Alzheimer's disease research and treatments (77 papers), Dementia and Cognitive Impairment Research (36 papers) and Functional Brain Connectivity Studies (19 papers). Christoph Höck collaborates with scholars based in Switzerland, United States and Germany. Christoph Höck's co-authors include Roger M. Nitsch, Dominique J.‐F. de Quervain, U. Otten, Klaus Heese, Andreas Papassotiropoulos, Benno Roozendaal, F. Müller‐Spahn, Christine Hulette, James L. McGaugh and Katharina Henke and has published in prestigious journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Christoph Höck

136 papers receiving 7.7k citations

Hit Papers

Antibodies against β-Amyloid Slow Cognitive Decline in Al... 2000 2026 2008 2017 2003 2000 2023 100 200 300 400 500

Peers

Christoph Höck
Scott A. Small United States
Robert K. McNamara United States
Jesse M. Cedarbaum United States
Heii Arai Japan
Nabeel Nabulsi United States
Christoph Höck
Citations per year, relative to Christoph Höck Christoph Höck (= 1×) peers Andreas Papassotiropoulos

Countries citing papers authored by Christoph Höck

Since Specialization
Citations

This map shows the geographic impact of Christoph Höck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christoph Höck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christoph Höck more than expected).

Fields of papers citing papers by Christoph Höck

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christoph Höck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christoph Höck. The network helps show where Christoph Höck may publish in the future.

Co-authorship network of co-authors of Christoph Höck

This figure shows the co-authorship network connecting the top 25 collaborators of Christoph Höck. A scholar is included among the top collaborators of Christoph Höck based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christoph Höck. Christoph Höck is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kirabali, Tunahan, Uwe Konietzko, Daniel Razansky, et al.. (2025). Reduced synaptic vesicle protein 2A in extracellular vesicles and brains of Alzheimer’s disease: associations with Aβ, tau, synaptic proteins and APOE ε4. Translational Neurodegeneration. 14(1). 48–48.
2.
Rauen, Katrin, Andreas Buchmann, Roger M. Nitsch, et al.. (2025). Protective Effects of Socioeconomic Status and Lifestyle on Amyloid‐ and White Matter Hyperintensity ‐Related Longitudinal Brain Atrophy and Cognitive Decline. Annals of Neurology. 98(6). 1222–1236.
3.
4.
Michels, Lars, Susanne Müller, Katrin Rauen, et al.. (2024). The links among age, sex, and glutathione: A cross-sectional magnetic resonance spectroscopy study. Neurobiology of Aging. 144. 19–29.
5.
Michalon, Aubin, Matthias Macháček, Chandrasekhar Udata, et al.. (2024). Prediction of Cardiac ATTR Depletion by NI006 (ALXN2220) Using Mechanistic PK/PD Modeling. Clinical Pharmacology & Therapeutics. 117(1). 261–269. 1 indexed citations
6.
Chen, Tianle, John O’Gorman, Carmen Castrillo‐Viguera, et al.. (2024). Results from the long‐term extension of PRIME: A randomized Phase 1b trial of aducanumab. Alzheimer s & Dementia. 20(5). 3406–3415. 11 indexed citations
7.
Schreiner, Simon J., Jiri M.G. Van Bergen, Anton Gietl, et al.. (2024). Gray matter gamma‐hydroxy‐butyric acid and glutamate reflect beta‐amyloid burden at old age. Alzheimer s & Dementia Diagnosis Assessment & Disease Monitoring. 16(2). e12587–e12587. 5 indexed citations
8.
Buchmann, Andreas, Katrin Rauen, Roger M. Nitsch, et al.. (2024). Explaining variability in early stages of [18F]‐flortaucipir tau‐PET binding: Focus on sex differences. Alzheimer s & Dementia Diagnosis Assessment & Disease Monitoring. 16(1). e12565–e12565. 3 indexed citations
9.
Heitz, Fabrice D., Karin Breu, Heather C. Denroche, et al.. (2023). A human antibody against pathologic IAPP aggregates protects beta cells in type 2 diabetes models. Nature Communications. 14(1). 6294–6294. 13 indexed citations
10.
Ni, Ruiqing, Xosé Luís Deán‐Ben, Valérie Treyer, et al.. (2022). Coregistered transcranial optoacoustic and magnetic resonance angiography of the human brain. Optics Letters. 48(3). 648–648. 9 indexed citations
11.
Buchmann, Andreas, Katrin Rauen, Roger M. Nitsch, et al.. (2022). Lifestyle Affects Amyloid Burden and Cognition Differently in Men and Women. Annals of Neurology. 92(3). 451–463. 19 indexed citations
12.
Treyer, Valérie, Anton Gietl, Rafael Meyer, et al.. (2020). Reduced uptake of [11C]‐ABP688, a PET tracer for metabolic glutamate receptor 5 in hippocampus and amygdala in Alzheimer’s dementia. Brain and Behavior. 10(6). e01632–e01632. 18 indexed citations
13.
Kulic, Luka, Charlotte E. Teunissen, Adeola Shobo, et al.. (2019). Aβ34 is a BACE1-derived degradation intermediate associated with amyloid clearance and Alzheimer’s disease progression. Nature Communications. 10(1). 2240–2240. 47 indexed citations
14.
Maier, Marcel, Tobias Welt, Fabio Montrasio, et al.. (2018). A human-derived antibody targets misfolded SOD1 and ameliorates motor symptoms in mouse models of amyotrophic lateral sclerosis. Science Translational Medicine. 10(470). 58 indexed citations
16.
Lindvall, Olle, et al.. (2009). Aβ Immunotherapy Protects Morphology and Survival of Adult-Born Neurons in Doubly Transgenic APP/PS1 Mice. Journal of Neuroscience. 29(45). 14108–14119. 51 indexed citations
17.
Höck, Christoph, Uwe Konietzko, Johannes Streffer, et al.. (2003). Antibodies against β-Amyloid Slow Cognitive Decline in Alzheimer's Disease. Neuron. 38(4). 547–554. 581 indexed citations breakdown →
18.
Höck, Christoph, Alessia Maddalena, F. Müller‐Spahn, et al.. (2003). Treatment with the selective muscarinic ml agonist talsaclidine decreases cerebrospinal fluid levels of Aβ42in patients with Alzheimer's disease. Amyloid. 10(1). 1–6. 82 indexed citations
19.
Drewe, Jürgen, et al.. (2003). Safety of injectable opioid maintenance treatment for heroin dependence. Biological Psychiatry. 54(8). 854–861. 27 indexed citations
20.
Höck, Christoph, Klaus Heese, Christine Hulette, Carlyn K. Rosenberg, & U. Otten. (2000). Region-Specific Neurotrophin Imbalances in Alzheimer Disease. Archives of Neurology. 57(6). 846–846. 452 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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