Chris Kay
Impact in
-
- Genetic Neurodegenerative Diseases
- Neurology top 2%
- Neurological disorders and treatments
- Parkinson's Disease Mechanisms and Treatments
Papers in
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- Genetic Neurodegenerative Diseases 23
- Co-authors
- Michael R. HaydenBlair R. LeavittChristopher A. RossGillian P. BatesJames F. GusellaSarah J. TabriziEdward J. WildMartha Nance
- Journals
- Biochemistry (6 papers)Biochemical Journal (5 papers)Journal of Biological Chemistry (4 papers)Genetics in Medicine (3 papers)FEBS Letters (3 papers)
- Partner nations
- United StatesUnited KingdomCanada
In The Last Decade
Chris Kay
63 papers receiving 2.7k citations
Hit Papers
Peers
Comparison fields: 5 of 133
- Cellular and Molecular Neuroscience 1.4k
- Neurology 591
- Molecular Biology 1.8k
- Aging 27
- Cell Biology 233
Countries citing papers authored by Chris Kay
This map shows the geographic impact of Chris Kay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chris Kay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chris Kay more than expected).
Fields of papers citing papers by Chris Kay
This network shows the impact of papers produced by Chris Kay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chris Kay. The network helps show where Chris Kay may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Chris Kay, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2024 | 0 | |
| 3 | 2023 | 15 | |
| 4 | 2022 | 6 | |
| 5 | 2021 | 17 | |
| 6 | 2020 | 11 | |
| 7 | 2020 | 13 | |
| 8 | 2020 | 8 | |
| 9 | 2019 | 120 | |
| 10 | 2018 | 17 | |
| 11 | 2017 | 10 | |
| 12 | 2015 | 64 | |
| 13 | Huntington disease Hit paper breakdown → | 2015 | 1082 |
| 14 | 2013 | 34 | |
| 15 | 2012 | 7 | |
| 16 | 2012 | 4 | |
| 17 | 1997 | 25 | |
| 18 | 1996 | 22 | |
| 19 | 1990 | 9 | |
| 20 | 1988 | 34 |
About Chris Kay
Chris Kay is a scholar working on Cellular and Molecular Neuroscience, Biophysics, Electrochemistry, Insect Science and Neurology, having authored 66 papers that have together received 2.8k indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (23 papers), Mitochondrial Function and Pathology (17 papers), Photosynthetic Processes and Mechanisms (10 papers), Trypanosoma species research and implications (9 papers), Neurological disorders and treatments (8 papers), Insect symbiosis and bacterial influences (8 papers), Muscle Physiology and Disorders (5 papers) and Research on Leishmaniasis Studies (5 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (1.4k citations), Neurology (591 citations), Molecular Biology (1.8k citations), Aging (27 citations) and Cell Biology (233 citations). Chris Kay has collaborated with scholars based in United States, United Kingdom and Canada. Frequent co-authors include Michael R. Hayden, Blair R. Leavitt, Christopher A. Ross, Gillian P. Bates, James F. Gusella, Sarah J. Tabrizi, Edward J. Wild, Martha Nance, Rachael I. Scahill and Ronald Wetzel. Their work appears in journals such as Biochemistry, Biochemical Journal, Journal of Biological Chemistry, Genetics in Medicine and FEBS Letters.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.