Chantévy Pou
- Cellular and Molecular Neuroscience top 5%
- Molecular Biology
- Endocrine and Autonomic Systems top 5%
- Nutrition and Dietetics top 10%
- Physiology
- Co-authors
- Kemal PayzaClaude GodboutRalf SchmidtW. A. BrownTomas HökfeltDajan O’DonnellPaola LemboManon Pelletier
- Topics
- Receptor Mechanisms and Signaling (6 papers)Neuropeptides and Animal Physiology (6 papers)Cancer Genomics and Diagnostics (4 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryJournal of Clinical Oncology
- Partner nations
- CanadaUnited KingdomUnited States
In The Last Decade
Chantévy Pou
16 papers receiving 739 citations
Peers
Comparison fields: 5 of 64
- Cellular and Molecular Neuroscience 445
- Molecular Biology 443
- Endocrine and Autonomic Systems 230
- Nutrition and Dietetics 138
- Physiology 112
Countries citing papers authored by Chantévy Pou
This map shows the geographic impact of Chantévy Pou's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chantévy Pou with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chantévy Pou more than expected).
Fields of papers citing papers by Chantévy Pou
This network shows the impact of papers produced by Chantévy Pou. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chantévy Pou. The network helps show where Chantévy Pou may publish in the future.
Co-authorship network of co-authors of Chantévy Pou
This figure shows the co-authorship network connecting the top 25 collaborators of Chantévy Pou. A scholar is included among the top collaborators of Chantévy Pou based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chantévy Pou. Chantévy Pou is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 17 | |
| 2 | 4 | |
| 3 | 1 | |
| 4 | 17 | |
| 5 | 3 | |
| 6 | 20 | |
| 7 | 37 | |
| 8 | 13 | |
| 9 | 19 | |
| 10 | 190 | |
| 11 | 70 | |
| 12 | 212 | |
| 13 | 30 | |
| 14 | 111 | |
| 15 | 7 | |
| 16 | 7 |
About Chantévy Pou
Chantévy Pou is a scholar working on Cellular and Molecular Neuroscience, Behavioral Neuroscience and Cancer Research, having authored 16 papers that have together received 758 indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (6 papers), Neuropeptides and Animal Physiology (6 papers) and Cancer Genomics and Diagnostics (4 papers). The work is most often cited by research in Endocrine and Autonomic Systems (230 citations), Cellular and Molecular Neuroscience (445 citations) and Nutrition and Dietetics (138 citations). Chantévy Pou has collaborated with scholars based in Canada, United Kingdom and United States. Frequent co-authors include Kemal Payza, Claude Godbout, Ralf Schmidt, W. A. Brown, Tomas Hökfelt, Dajan O’Donnell, Paola Lembo, Manon Pelletier, Philippe Walker and Pablo R. Brumovsky. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Oncology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.