Carlotta Cancelliere
- Oncology top 5%
- Colorectal Cancer Treatments and Studies 3
- Cancer Immunotherapy and Biomarkers 2
- Cancer Cells and Metastasis 1
- Cancer Research top 10%
- Pathology and Forensic Medicine top 10%
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- Protein Kinase Regulation and GTPase Signaling 3
- Cell death mechanisms and regulation 2
- Protein Degradation and Inhibitors 2
- Melanoma and MAPK Pathways 2
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- Chronic Lymphocytic Leukemia Research 2
- Co-authors
- Alberto BardelliFederica Di NicolantonioMariangela RussoSimona LambaSabrina ArenaDavide ZecchinMassimo GeunaMargherita Gallicchio
- Journals
- Proceedings of the National Academy of Sciences (1 paper)Journal of Clinical Investigation (1 paper)Nature Medicine (1 paper)
- Partner nations
- ItalyUnited StatesGermany
In The Last Decade
Carlotta Cancelliere
11 papers receiving 976 citations
Peers
Comparison fields: 5 of 75
- Oncology 466
- Cancer Research 203
- Pathology and Forensic Medicine 186
- Molecular Biology 529
- Immunology 141
Countries citing papers authored by Carlotta Cancelliere
This map shows the geographic impact of Carlotta Cancelliere's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Carlotta Cancelliere with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Carlotta Cancelliere more than expected).
Fields of papers citing papers by Carlotta Cancelliere
This network shows the impact of papers produced by Carlotta Cancelliere. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Carlotta Cancelliere. The network helps show where Carlotta Cancelliere may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Carlotta Cancelliere, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 4 | |
| 2 | 2019 | 38 | |
| 3 | 2018 | 1 | |
| 4 | 2016 | 215 | |
| 5 | 2015 | 217 | |
| 6 | 2015 | 12 | |
| 7 | 2014 | 87 | |
| 8 | 2013 | 16 | |
| 9 | 2010 | 288 | |
| 10 | 2008 | 75 | |
| 11 | 2007 | 38 |
About Carlotta Cancelliere
Carlotta Cancelliere is a scholar working on Oncology, Genetics and Biotechnology, having authored 11 papers that have together received 991 indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (3 papers), Colorectal Cancer Treatments and Studies (3 papers), Cell death mechanisms and regulation (2 papers), Chronic Lymphocytic Leukemia Research (2 papers), Cancer Immunotherapy and Biomarkers (2 papers), Protein Degradation and Inhibitors (2 papers), Melanoma and MAPK Pathways (2 papers) and Cancer Cells and Metastasis (1 paper). The work is most often cited by research in Oncology (466 citations), Cancer Research (203 citations) and Pathology and Forensic Medicine (186 citations). Carlotta Cancelliere has collaborated with scholars based in Italy, United States and Germany. Frequent co-authors include Alberto Bardelli, Federica Di Nicolantonio, Mariangela Russo, Simona Lamba, Sabrina Arena, Davide Zecchin, Massimo Geuna, Margherita Gallicchio, Enzo Médico and Stefano Biffo. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and Nature Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.