C. Jacquemin

1.2k total citations
18 papers, 700 citations indexed

About

C. Jacquemin is a scholar working on Immunology, Cell Biology and Molecular Biology. According to data from OpenAlex, C. Jacquemin has authored 18 papers receiving a total of 700 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Immunology, 11 papers in Cell Biology and 4 papers in Molecular Biology. Recurrent topics in C. Jacquemin's work include melanin and skin pigmentation (11 papers), T-cell and B-cell Immunology (7 papers) and Immune Cell Function and Interaction (6 papers). C. Jacquemin is often cited by papers focused on melanin and skin pigmentation (11 papers), T-cell and B-cell Immunology (7 papers) and Immune Cell Function and Interaction (6 papers). C. Jacquemin collaborates with scholars based in France, United States and Belgium. C. Jacquemin's co-authors include Julien Sénéschal, Katia Boniface, Alain Taı̈eb, Denis Thiolat, Jérôme Rambert, Christina Martins, Fabienne Lucchese, Khaled Ezzedine, A.‐S. Darrigade and Patrick Blanco and has published in prestigious journals such as PLoS ONE, Annals of the Rheumatic Diseases and Journal of Investigative Dermatology.

In The Last Decade

C. Jacquemin

17 papers receiving 693 citations

Peers

C. Jacquemin
Maggi Ahmed Refat United States
Christina Martins United States
Hui Cheng China
Martin B. Lyons United States
Sylviane Lambert United States
Yale Liu China
Maggi Ahmed Refat United States
C. Jacquemin
Citations per year, relative to C. Jacquemin C. Jacquemin (= 1×) peers Maggi Ahmed Refat

Countries citing papers authored by C. Jacquemin

Since Specialization
Citations

This map shows the geographic impact of C. Jacquemin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by C. Jacquemin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites C. Jacquemin more than expected).

Fields of papers citing papers by C. Jacquemin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by C. Jacquemin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by C. Jacquemin. The network helps show where C. Jacquemin may publish in the future.

Co-authorship network of co-authors of C. Jacquemin

This figure shows the co-authorship network connecting the top 25 collaborators of C. Jacquemin. A scholar is included among the top collaborators of C. Jacquemin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with C. Jacquemin. C. Jacquemin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Vallon, R., C. Jacquemin, Clara Cilindre, et al.. (2025). Mapping gaseous ethanol in the headspace of wine glasses with an interband cascade laser sensor. Sensors and Actuators B Chemical. 443. 138216–138216.
2.
Sénéschal, Julien, Katia Boniface, & C. Jacquemin. (2022). Alopecia areata: Recent advances and emerging therapies. Annales de Dermatologie et de Vénéréologie. 149(4). 222–227. 12 indexed citations
3.
Martins, Christina, Claire Drullion, C. Jacquemin, et al.. (2021). Vitiligo Skin T Cells Are Prone to Produce Type 1 and Type 2 Cytokines to Induce Melanocyte Dysfunction and Epidermal Inflammatory Response Through Jak Signaling. Journal of Investigative Dermatology. 142(4). 1194–1205.e7. 50 indexed citations
4.
Martins, Christina, A.‐S. Darrigade, Claire Drullion, et al.. (2020). Type-1 cytokines regulate matrix metalloprotease-9 production and E-cadherin disruption to promote melanocyte loss in vitiligo. JCI Insight. 5(11). 47 indexed citations
5.
Martins, Christina, A.‐S. Darrigade, C. Jacquemin, et al.. (2020). Phenotype and function of circulating memory T cells in human vitiligo*. British Journal of Dermatology. 183(5). 899–908. 25 indexed citations
6.
Jacquemin, C., Christina Martins, Fabienne Lucchese, et al.. (2019). NKG2D Defines a Subset of Skin Effector Memory CD8 T Cells with Proinflammatory Functions in Vitiligo. Journal of Investigative Dermatology. 140(6). 1143–1153.e5. 44 indexed citations
7.
Garnier, Anne‐Sophie, Jérémie Riou, C. Jacquemin, et al.. (2019). Pre-transplant CD45RC expression on blood T cells differentiates patients with cancer and rejection after kidney transplantation. PLoS ONE. 14(3). e0214321–e0214321. 5 indexed citations
8.
Jacquemin, C., Jean‐François Augusto, Marc Scherlinger, et al.. (2018). OX40L/OX40 axis impairs follicular and natural Treg function in human SLE. JCI Insight. 3(24). 62 indexed citations
9.
Jacquemin, C., Alain Taı̈eb, Katia Boniface, & Julien Sénéschal. (2018). 088 Imbalance of peripheral T follicular helper lymphocyte subsets in active vitiligo. Journal of Investigative Dermatology. 138(5). S15–S15. 1 indexed citations
10.
Jacquemin, C., et al.. (2018). Imbalance of peripheral follicular helper T lymphocyte subsets in active vitiligo. Pigment Cell & Melanoma Research. 32(4). 588–592. 8 indexed citations
11.
Boniface, Katia, C. Jacquemin, A.‐S. Darrigade, et al.. (2017). Vitiligo Skin Is Imprinted with Resident Memory CD8 T Cells Expressing CXCR3. Journal of Investigative Dermatology. 138(2). 355–364. 180 indexed citations
12.
Jacquemin, C., Jérôme Rambert, Denis Thiolat, et al.. (2017). Vitiligo-like lesions occurring in patients receiving anti-programmed cell death–1 therapies are clinically and biologically distinct from vitiligo. Journal of the American Academy of Dermatology. 76(5). 863–870. 112 indexed citations
13.
Geel, Nanja van, Katia Boniface, Julien Sénéschal, et al.. (2017). Meeting report: Vitiligo Global Issues Consensus Conference Workshop “Outcome measurement instruments” and Vitiligo International Symposium, Rome, Nov 30–Dec 3rd. Pigment Cell & Melanoma Research. 30(4). 436–443. 14 indexed citations
14.
Jacquemin, C., Jérôme Rambert, S. Guillet, et al.. (2017). Heat shock protein 70 potentiates interferon alpha production by plasmacytoid dendritic cells: relevance for cutaneous lupus and vitiligo pathogenesis. British Journal of Dermatology. 177(5). 1367–1375. 72 indexed citations
15.
Truchetet, Marie‐Elise, Valérie Jolivel, Isabelle Douchet, et al.. (2016). Platelets Induce Thymic Stromal Lymphopoietin Production by Endothelial Cells: Contribution to Fibrosis in Human Systemic Sclerosis. Arthritis & Rheumatology. 68(11). 2784–2794. 37 indexed citations
16.
Khoryati, Liliane, Jean‐François Augusto, Cécile Contin‐Bordes, et al.. (2016). IgE Inhibits Toll‐like Receptor 7– and Toll‐like Receptor 9–Mediated Expression of Interferon‐α by Plasmacytoid Dendritic Cells in Patients With Systemic Lupus Erythematosus. Arthritis & Rheumatology. 68(9). 2221–2231. 20 indexed citations
17.
Bentebibel, Salah-Eddine, C. Jacquemin, Nathalie Schmitt, & Hideki Ueno. (2015). Analysis of Human Blood Memory T Follicular Helper Subsets. Methods in molecular biology. 1291. 187–197. 10 indexed citations
18.
Jacquemin, C., Nathalie Schmitt, Cécile Contin‐Bordes, et al.. (2015). OP0009 OX40 Ligand Contributes to Human Lupus Pathogenesis by Promoting T Follicular Helper Response. Annals of the Rheumatic Diseases. 74. 67–67. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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