Yale Liu

982 total citations
41 papers, 558 citations indexed

About

Yale Liu is a scholar working on Immunology, Molecular Biology and Cancer Research. According to data from OpenAlex, Yale Liu has authored 41 papers receiving a total of 558 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Immunology, 12 papers in Molecular Biology and 12 papers in Cancer Research. Recurrent topics in Yale Liu's work include NF-κB Signaling Pathways (11 papers), Autoimmune Bullous Skin Diseases (9 papers) and Psoriasis: Treatment and Pathogenesis (8 papers). Yale Liu is often cited by papers focused on NF-κB Signaling Pathways (11 papers), Autoimmune Bullous Skin Diseases (9 papers) and Psoriasis: Treatment and Pathogenesis (8 papers). Yale Liu collaborates with scholars based in China, United States and Poland. Yale Liu's co-authors include Yumin Xia, Liang Li, Shengxiang Xiao, Lingling Peng, Meifeng Xu, Kunyi Wu, Chengfei Liu, Raymond J. Cho, Jeffrey P. North and Jeffrey B. Cheng and has published in prestigious journals such as The Lancet, The Journal of Immunology and Scientific Reports.

In The Last Decade

Yale Liu

36 papers receiving 550 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yale Liu China 15 228 163 143 125 110 41 558
Pei Qiao China 13 369 1.6× 229 1.4× 155 1.1× 122 1.0× 120 1.1× 30 685
W. Nürnberg Germany 12 175 0.8× 208 1.3× 90 0.6× 61 0.5× 121 1.1× 27 536
Shengxian Shen China 10 222 1.0× 111 0.7× 98 0.7× 91 0.7× 81 0.7× 22 437
Alejandro Morales United States 12 121 0.5× 170 1.0× 151 1.1× 80 0.6× 123 1.1× 27 640
N. Vandeghinste Belgium 9 154 0.7× 182 1.1× 117 0.8× 44 0.4× 139 1.3× 15 586
Karline Guilloteau France 8 315 1.4× 202 1.2× 176 1.2× 38 0.3× 23 0.2× 10 618
Raquel Navarro Spain 8 302 1.3× 146 0.9× 121 0.8× 54 0.4× 50 0.5× 15 465
Stefan Frischbutter Germany 15 474 2.1× 140 0.9× 144 1.0× 196 1.6× 455 4.1× 33 916
Hirotsugu Hino Japan 13 53 0.2× 198 1.2× 40 0.3× 126 1.0× 114 1.0× 42 552
L. Dousset France 11 108 0.5× 205 1.3× 107 0.7× 59 0.5× 26 0.2× 34 618

Countries citing papers authored by Yale Liu

Since Specialization
Citations

This map shows the geographic impact of Yale Liu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yale Liu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yale Liu more than expected).

Fields of papers citing papers by Yale Liu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yale Liu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yale Liu. The network helps show where Yale Liu may publish in the future.

Co-authorship network of co-authors of Yale Liu

This figure shows the co-authorship network connecting the top 25 collaborators of Yale Liu. A scholar is included among the top collaborators of Yale Liu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yale Liu. Yale Liu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhu, Yan, Qin Chen, Jing Li, et al.. (2025). Carbon dot superoxide dismutase nanozyme enhances reactive oxygen species scavenging in diabetic skin wound repair. Journal of Advanced Research. 79. 691–706. 3 indexed citations
2.
Xia, Yumin, et al.. (2025). Periostin in Bullous Pemphigoid: A Potential Biomarker of Disease Activity and Severity. Experimental Dermatology. 34(2). e70067–e70067.
3.
4.
Duan, Xudong, Christopher Cook, Mei Lü, et al.. (2025). Synergistic effects of LCN2 and TWEAK on the progression of psoriasis. Cellular and Molecular Immunology. 22(7). 760–775. 2 indexed citations
5.
Wu, Kunyi, Christopher Cook, Liang Li, et al.. (2024). Effect of opioid receptor antagonist on mitigating tumor necrosis factor-like weak inducer of apoptosis (TWEAK)-induced apoptolysis in pemphigus pathogenesis. Journal of Autoimmunity. 149. 103307–103307. 4 indexed citations
6.
Hakimi, Marwa, Jeffrey P. North, Mark A. Taylor, et al.. (2023). Transcriptomics aids differentiation of IL-23 overactivity in a patient with atypical skin and joint disease. The Lancet. 401(10385). 1381–1381. 6 indexed citations
7.
Liu, Yale, Hao Wang, Christopher Cook, et al.. (2022). Defining Patient-Level Molecular Heterogeneity in Psoriasis Vulgaris Based on Single-Cell Transcriptomics. Frontiers in Immunology. 13. 842651–842651. 14 indexed citations
8.
Li, Liang, Christopher Cook, Yale Liu, et al.. (2022). Endothelial glycocalyx in hepatopulmonary syndrome: An indispensable player mediating vascular changes. Frontiers in Immunology. 13. 1039618–1039618. 1 indexed citations
9.
Lü, Mei, et al.. (2021). Plasma levels of D-dimer and fibrin degradation products correlate with bullous pemphigoid severity: a cross-sectional study. Scientific Reports. 11(1). 17746–17746. 1 indexed citations
10.
Liu, Yale, et al.. (2020). 511 TWEAK is a critical cytokine linking vulgaris, pustular, and erythrodermic psoriasis. Journal of Investigative Dermatology. 140(7). S69–S69. 1 indexed citations
11.
Wang, Huixia, Liang Li, Xiuying Wang, et al.. (2020). Involvement of the cytokine TWEAK in the pathogenesis of psoriasis vulgaris, pustular psoriasis, and erythrodermic psoriasis. Cytokine. 138. 155391–155391. 15 indexed citations
12.
Liu, Yale, Liang Li, & Yumin Xia. (2017). BP180 Is Critical in the Autoimmunity of Bullous Pemphigoid. Frontiers in Immunology. 8. 1752–1752. 78 indexed citations
13.
Liu, Yale, Lingling Peng, Liang Li, et al.. (2017). TWEAK/Fn14 Activation Contributes to the Pathogenesis of Bullous Pemphigoid. Journal of Investigative Dermatology. 137(7). 1512–1522. 39 indexed citations
14.
Liu, Yale, Meifeng Xu, Kunyi Wu, et al.. (2017). TWEAK/Fn14 Activation Participates in Ro52-Mediated Photosensitization in Cutaneous Lupus Erythematosus. Frontiers in Immunology. 8. 651–651. 35 indexed citations
15.
Liu, Yale, et al.. (2016). Hypoxia stimulates the proliferation of rat neural stem cells by regulating the expression of metabotropic glutamate receptors: an in vitro study.. PubMed. 62(3). 105–14. 3 indexed citations
16.
Liu, Yale, Weihui Zeng, Die Hu, et al.. (2016). The long-term effect of 1550 nm erbium:glass fractional laser in acne vulgaris. Lasers in Medical Science. 31(3). 453–457. 14 indexed citations
17.
Ren, Jianwen, et al.. (2016). Meta-Analysis of the Association between Vitiligo and Human Leukocyte Antigen-A. BioMed Research International. 2016. 1–13. 11 indexed citations
18.
Liu, Yale, et al.. (2016). A transcription factor 7-like 1-lipocalin 2 axis in the differentiation of keratinocytes. Cell Death and Disease. 7(6). e2241–e2241. 2 indexed citations
19.
Liu, Yale, et al.. (2015). Gold nanoparticles protect against skin photoaging through photothermal effects in ICR mice: a preliminary study. Chinese Journal of Dermatology. 48(10). 713–718. 1 indexed citations
20.
Liu, Yale, et al.. (2011). A novel mutation of CYLD in a Chinese family with multiple familial trichoepithelioma. Journal of the European Academy of Dermatology and Venereology. 26(11). 1420–1423. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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