C. Gastón Bisig

451 citations
17 papers · 337 indexed · h-index 10
    • Microtubule and mitosis dynamics 9
    • Cellular Mechanics and Interactions 2
    • DNA Repair Mechanisms 3
    • Histone Deacetylase Inhibitors Research 3
    • 14-3-3 protein interactions 3
    • Nuclear Structure and Function 2
    • Ubiquitin and proteasome pathways 2
    • Neurogenesis and neuroplasticity mechanisms 2

C. Gastón Bisig

17 papers receiving 337 citations

Peers

C. Gastón Bisig
Comparison fields: 5 of 60
  • Cell Biology 99
  • Aging 8
  • Physiology 18
  • Molecular Biology 240
  • Developmental Neuroscience 12
Replace Huikyong Lee with:
Huikyong Lee South Korea
Claire Didszun Germany
Remo Lazzarini Italy
Safia Ladha Canada
Galdo Bustos Chile
Giovana da Silva Leandro Brazil
Zhong Yan Gan Australia
Ladislav Kuchař Czechia
Merle Mandel Estonia
Annika Pfeiffer Germany
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Citations per field
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Citations per year

Countries citing papers authored by C. Gastón Bisig

Since Specialization
Citations

This map shows the geographic impact of C. Gastón Bisig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by C. Gastón Bisig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites C. Gastón Bisig more than expected).

Fields of papers citing papers by C. Gastón Bisig

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by C. Gastón Bisig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by C. Gastón Bisig. The network helps show where C. Gastón Bisig may publish in the future.

Co-authorship network

The 25 scholars most cited alongside C. Gastón Bisig, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with C. Gastón Bisig Line = papers co-authored together C. Gastón Bisig links everyone, so they are left out of the graph.

All Works

17 of 17 papers shown
#Work
1 20218
2 202010
3 202019
4 20206
5 20183
6 20166
7 20144
8 20133
9 201299
10 20129
11 201019
12 20097
13 200640
14 200448
15 200310
16 200310
17 200236

About C. Gastón Bisig

C. Gastón Bisig is a scholar working on Cell Biology, Developmental Neuroscience and Molecular Biology, having authored 17 papers that have together received 337 indexed citations. Recurring topics across this work include Microtubule and mitosis dynamics (9 papers), DNA Repair Mechanisms (3 papers), Histone Deacetylase Inhibitors Research (3 papers), 14-3-3 protein interactions (3 papers), Cellular Mechanics and Interactions (2 papers), Nuclear Structure and Function (2 papers), Ubiquitin and proteasome pathways (2 papers) and Neurogenesis and neuroplasticity mechanisms (2 papers). The work is most often cited by research in Cell Biology (99 citations), Aging (8 citations) and Physiology (18 citations). C. Gastón Bisig has collaborated with scholars based in Argentina, United States and Uruguay. Frequent co-authors include Carlos A. Arce, Silvia A. Purro, Roberto J. Pezza, Héctor S. Barra, Christer Höög, Harry Scherthan, Michel F. Guiraldelli, Anna Kouznetsova, Dean Dawson and María A. Contín. Their work appears in journals such as Current Biology, Scientific Reports and Biochemical Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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