Bernhard M. Schmitt
- Molecular Biology top 10%
- Oncology top 10%
- Surgery top 10%
- Cellular and Molecular Neuroscience top 5%
- Genetics top 5%
- Co-authors
- Hermann KoepsellWalter F. BoronValentin GorboulevMichael F. RomeroMark O. BevenseeInyeong ChoiJochen RingeMichael Sittinger
- Topics
- Ion Transport and Channel Regulation (14 papers)Ion channel regulation and function (10 papers)Neuroscience and Neuropharmacology Research (6 papers)
- Cited by
- BiochemistryGeneticsUrology
- Partner nations
- GermanyUnited StatesNew Zealand
In The Last Decade
Bernhard M. Schmitt
29 papers receiving 1.9k citations
Peers
Comparison fields: 5 of 101
- Molecular Biology 1.1k
- Oncology 369
- Surgery 316
- Cellular and Molecular Neuroscience 296
- Genetics 266
Countries citing papers authored by Bernhard M. Schmitt
This map shows the geographic impact of Bernhard M. Schmitt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bernhard M. Schmitt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bernhard M. Schmitt more than expected).
Fields of papers citing papers by Bernhard M. Schmitt
This network shows the impact of papers produced by Bernhard M. Schmitt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bernhard M. Schmitt. The network helps show where Bernhard M. Schmitt may publish in the future.
Co-authorship network of co-authors of Bernhard M. Schmitt
This figure shows the co-authorship network connecting the top 25 collaborators of Bernhard M. Schmitt. A scholar is included among the top collaborators of Bernhard M. Schmitt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bernhard M. Schmitt. Bernhard M. Schmitt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 15 | |
| 2 | 12 | |
| 3 | 18 | |
| 4 | 5 | |
| 5 | 22 | |
| 6 | 34 | |
| 7 | 179 | |
| 8 | 8 | |
| 9 | 235 | |
| 10 | 36 | |
| 11 | 191 | |
| 12 | 33 | |
| 13 | 179 | |
| 14 | 44 | |
| 15 | 84 | |
| 16 | 94 | |
| 17 | 71 | |
| 18 | 56 | |
| 19 | 143 | |
| 20 | 9 |
About Bernhard M. Schmitt
Bernhard M. Schmitt is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Urology, having authored 29 papers that have together received 1.9k indexed citations. Recurring topics across this work include Ion Transport and Channel Regulation (14 papers), Ion channel regulation and function (10 papers) and Neuroscience and Neuropharmacology Research (6 papers). The work is most often cited by research in Biochemistry (185 citations), Genetics (266 citations) and Urology (123 citations). Bernhard M. Schmitt has collaborated with scholars based in Germany, United States and New Zealand. Frequent co-authors include Hermann Koepsell, Walter F. Boron, Valentin Gorboulev, Michael F. Romero, Mark O. Bevensee, Inyeong Choi, Jochen Ringe, Michael Sittinger, Christian Kaps and Thomas Häupl. Their work appears in journals such as Journal of Biological Chemistry, Journal of Neuroscience and Gastroenterology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.