Bernardo Ramírez‐Zavala

679 total citations
28 papers, 518 citations indexed

About

Bernardo Ramírez‐Zavala is a scholar working on Infectious Diseases, Molecular Biology and Epidemiology. According to data from OpenAlex, Bernardo Ramírez‐Zavala has authored 28 papers receiving a total of 518 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Infectious Diseases, 16 papers in Molecular Biology and 12 papers in Epidemiology. Recurrent topics in Bernardo Ramírez‐Zavala's work include Antifungal resistance and susceptibility (20 papers), Fungal Infections and Studies (10 papers) and Fungal and yeast genetics research (10 papers). Bernardo Ramírez‐Zavala is often cited by papers focused on Antifungal resistance and susceptibility (20 papers), Fungal Infections and Studies (10 papers) and Fungal and yeast genetics research (10 papers). Bernardo Ramírez‐Zavala collaborates with scholars based in Germany, United States and Mexico. Bernardo Ramírez‐Zavala's co-authors include Joachim Morschhäuser, Knut Ohlsen, Oliver Reuß, Inès Krüger, Tsvia Gildor, Robert A. Arkowitz, Daniel Kornitzer, Ángel Domínguez, Ute Römling and Birgitta Agerberth and has published in prestigious journals such as Nature Communications, Genetics and Antimicrobial Agents and Chemotherapy.

In The Last Decade

Bernardo Ramírez‐Zavala

26 papers receiving 512 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Bernardo Ramírez‐Zavala Germany 14 336 256 235 85 78 28 518
Grazyna J. Sosinska Netherlands 6 281 0.8× 167 0.7× 202 0.9× 108 1.3× 68 0.9× 7 417
Sophia Ushinsky Canada 6 275 0.8× 178 0.7× 326 1.4× 122 1.4× 46 0.6× 6 498
Tricia L. Lo Australia 14 380 1.1× 292 1.1× 322 1.4× 95 1.1× 49 0.6× 19 661
Michael Tscherner Austria 14 304 0.9× 234 0.9× 169 0.7× 80 0.9× 51 0.7× 22 479
Antonia Geber United States 8 364 1.1× 239 0.9× 167 0.7× 68 0.8× 50 0.6× 10 562
Murielle Chauvel France 12 516 1.5× 329 1.3× 253 1.1× 76 0.9× 102 1.3× 24 634
Frank Stehr Germany 7 280 0.8× 223 0.9× 153 0.7× 48 0.6× 67 0.9× 7 450
Brooke D. Esquivel United States 8 282 0.8× 217 0.8× 148 0.6× 80 0.9× 43 0.6× 11 452
Roy A. Khalaf Lebanon 13 385 1.1× 299 1.2× 245 1.0× 79 0.9× 68 0.9× 33 582
Fernando L. Hernando Spain 14 291 0.9× 179 0.7× 131 0.6× 96 1.1× 23 0.3× 31 457

Countries citing papers authored by Bernardo Ramírez‐Zavala

Since Specialization
Citations

This map shows the geographic impact of Bernardo Ramírez‐Zavala's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bernardo Ramírez‐Zavala with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bernardo Ramírez‐Zavala more than expected).

Fields of papers citing papers by Bernardo Ramírez‐Zavala

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bernardo Ramírez‐Zavala. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bernardo Ramírez‐Zavala. The network helps show where Bernardo Ramírez‐Zavala may publish in the future.

Co-authorship network of co-authors of Bernardo Ramírez‐Zavala

This figure shows the co-authorship network connecting the top 25 collaborators of Bernardo Ramírez‐Zavala. A scholar is included among the top collaborators of Bernardo Ramírez‐Zavala based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bernardo Ramírez‐Zavala. Bernardo Ramírez‐Zavala is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Günther, K, Axel Dietschmann, Hrant Hovhannisyan, et al.. (2025). Host albumin redirects Candida albicans metabolism to engage an alternative pathogenicity pathway. Nature Communications. 16(1). 6447–6447.
4.
Ramírez‐Zavala, Bernardo, et al.. (2024). The zinc cluster transcription factor Znc1 regulates Rta3-dependent miltefosine resistance in Candida albicans. mSphere. 9(7). e0027024–e0027024. 2 indexed citations
5.
Cruz, Melissa R., et al.. (2023). Host-derived reactive oxygen species trigger activation of the Candida albicans transcription regulator Rtg1/3. PLoS Pathogens. 19(9). e1011692–e1011692. 1 indexed citations
6.
Ramírez‐Zavala, Bernardo, et al.. (2022). The protein kinase Ire1 has a Hac1-independent essential role in iron uptake and virulence of Candida albicans. PLoS Pathogens. 18(2). e1010283–e1010283. 17 indexed citations
7.
Brandt, Philipp, Franziska Gerwien, Lysett Wagner, et al.. (2022). Candida albicans SR-Like Protein Kinases Regulate Different Cellular Processes: Sky1 Is Involved in Control of Ion Homeostasis, While Sky2 Is Important for Dipeptide Utilization. Frontiers in Cellular and Infection Microbiology. 12. 850531–850531. 4 indexed citations
8.
Ramírez‐Zavala, Bernardo, et al.. (2021). The zinc cluster transcription factor Rha1 is a positive filamentation regulator in Candida albicans. Genetics. 220(1). 5 indexed citations
9.
Ramírez‐Zavala, Bernardo, et al.. (2021). The zinc cluster transcription factor Czf1 regulates cell wall architecture and integrity in Candida albicans. Molecular Microbiology. 116(2). 483–497. 13 indexed citations
10.
Ramírez‐Zavala, Bernardo, et al.. (2021). A Suppressor Mutation in the β-Subunit Kis1 Restores Functionality of the SNF1 Complex in Candida albicans snf4 Δ Mutants. mSphere. 6(6). e0092921–e0092921. 3 indexed citations
13.
Gildor, Tsvia, Bernardo Ramírez‐Zavala, Ziva Weissman, et al.. (2018). A Global Analysis of Kinase Function in Candida albicans Hyphal Morphogenesis Reveals a Role for the Endocytosis Regulator Akl1. Frontiers in Cellular and Infection Microbiology. 8. 17–17. 18 indexed citations
14.
Ramírez‐Zavala, Bernardo, et al.. (2014). SAGA/ADA Complex Subunit Ada2 Is Required for Cap1- but Not Mrr1-Mediated Upregulation of the Candida albicans Multidrug Efflux Pump MDR1. Antimicrobial Agents and Chemotherapy. 58(9). 5102–5110. 19 indexed citations
15.
Ramírez‐Zavala, Bernardo, et al.. (2013). Activation of the Cph1-Dependent MAP Kinase Signaling Pathway Induces White-Opaque Switching in Candida albicans. PLoS Pathogens. 9(10). e1003696–e1003696. 47 indexed citations
16.
Ramírez‐Zavala, Bernardo & Ángel Domínguez. (2008). Evolution and phylogenetic relationships of APSES proteins from Hemiascomycetes. FEMS Yeast Research. 8(4). 511–519. 17 indexed citations
17.
Ramírez‐Zavala, Bernardo, et al.. (2008). Environmental Induction of White–Opaque Switching in Candida albicans. PLoS Pathogens. 4(6). e1000089–e1000089. 114 indexed citations
18.
Mercado-Flores, Yuridia, et al.. (2005). Purification and characterization of an intracellular aspartyl acid proteinase (pumAi) fromUstilago maydis. Canadian Journal of Microbiology. 51(2). 171–175. 4 indexed citations
19.
Ramírez‐Zavala, Bernardo. (2004). Purification and characterization of a lysine aminopeptidase from Kluyveromyces marxianus. FEMS Microbiology Letters. 235(2). 369–375. 51 indexed citations
20.
Ramírez‐Zavala, Bernardo, et al.. (2003). Purification and characterization of a serine carboxypeptidase from Kluyveromyces marxianus. International Journal of Food Microbiology. 91(3). 245–252. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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