Axel Dietschmann

427 total citations
18 papers, 246 citations indexed

About

Axel Dietschmann is a scholar working on Infectious Diseases, Immunology and Molecular Biology. According to data from OpenAlex, Axel Dietschmann has authored 18 papers receiving a total of 246 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Infectious Diseases, 7 papers in Immunology and 5 papers in Molecular Biology. Recurrent topics in Axel Dietschmann's work include Antifungal resistance and susceptibility (8 papers), IL-33, ST2, and ILC Pathways (6 papers) and Reproductive tract infections research (4 papers). Axel Dietschmann is often cited by papers focused on Antifungal resistance and susceptibility (8 papers), IL-33, ST2, and ILC Pathways (6 papers) and Reproductive tract infections research (4 papers). Axel Dietschmann collaborates with scholars based in Germany, United States and Netherlands. Axel Dietschmann's co-authors include David Voehringer, Mark S. Gresnigt, Sven Krappmann, N. Louise Glass, Vincent W. Wu, J. Philipp Benz, Nils Thieme, Igor V. Grigoriev, Vasanth Singan and Lori B. Huberman and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Nature Immunology.

In The Last Decade

Axel Dietschmann

17 papers receiving 244 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Axel Dietschmann Germany 9 122 56 52 45 43 18 246
Senthil Kumar Velusamy United States 11 94 0.8× 27 0.5× 35 0.7× 14 0.3× 18 0.4× 16 342
Yilin Zhao China 11 117 1.0× 25 0.4× 28 0.5× 41 0.9× 52 1.2× 30 304
Decong Kong China 12 125 1.0× 44 0.8× 21 0.4× 53 1.2× 20 0.5× 32 326
Shahida Shahana Sweden 9 133 1.1× 47 0.8× 61 1.2× 147 3.3× 12 0.3× 13 409
Kyung Eun Jung South Korea 10 47 0.4× 36 0.6× 37 0.7× 51 1.1× 7 0.2× 41 315
B. Tümmler Germany 10 182 1.5× 27 0.5× 37 0.7× 17 0.4× 16 0.4× 19 505
Anna Tai Australia 11 134 1.1× 18 0.3× 18 0.3× 35 0.8× 16 0.4× 30 307
Kaesi A. Morelli United States 5 147 1.2× 25 0.4× 31 0.6× 89 2.0× 10 0.2× 5 280
Y.‐S. Kim South Korea 7 123 1.0× 53 0.9× 13 0.3× 27 0.6× 11 0.3× 7 291
S.‐W. Hong South Korea 5 245 2.0× 43 0.8× 180 3.5× 38 0.8× 13 0.3× 6 534

Countries citing papers authored by Axel Dietschmann

Since Specialization
Citations

This map shows the geographic impact of Axel Dietschmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Axel Dietschmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Axel Dietschmann more than expected).

Fields of papers citing papers by Axel Dietschmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Axel Dietschmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Axel Dietschmann. The network helps show where Axel Dietschmann may publish in the future.

Co-authorship network of co-authors of Axel Dietschmann

This figure shows the co-authorship network connecting the top 25 collaborators of Axel Dietschmann. A scholar is included among the top collaborators of Axel Dietschmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Axel Dietschmann. Axel Dietschmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Günther, K, Axel Dietschmann, Hrant Hovhannisyan, et al.. (2025). Host albumin redirects Candida albicans metabolism to engage an alternative pathogenicity pathway. Nature Communications. 16(1). 6447–6447.
2.
Rosati, Diletta, Marisa Valentine, Mariolina Bruno, et al.. (2025). Lactic acid in the vaginal milieu modulates the Candida -host interaction. Virulence. 16(1). 2451165–2451165. 1 indexed citations
3.
Valentine, Marisa, Diletta Rosati, Axel Dietschmann, et al.. (2025). Probiotic Lactobacillus Species Modulate Immune Responses During Vaginal Epithelial Cell Colonization. The Journal of Infectious Diseases. 232(3). e403–e415. 1 indexed citations
4.
Jaeger, Martin, Axel Dietschmann, Larsen Vornholz, et al.. (2024). Alpha1-antitrypsin impacts innate host–pathogen interactions with Candida albicans by stimulating fungal filamentation. Virulence. 15(1). 2333367–2333367. 6 indexed citations
5.
Zelante, Teresa, et al.. (2024). Inflammatory cytokine signalling in vulvovaginal candidiasis: a hot mess driving immunopathology. PubMed. 5(1). iqae010–iqae010. 14 indexed citations
6.
Dietschmann, Axel, et al.. (2024). Microbial adaptive pathogenicity strategies to the host inflammatory environment. FEMS Microbiology Reviews. 49. 6 indexed citations
7.
Valentine, Marisa, Axel Dietschmann, Selene Mogavero, et al.. (2024). Nanobody-mediated neutralization of candidalysin prevents epithelial damage and inflammatory responses that drive vulvovaginal candidiasis pathogenesis. mBio. 15(3). e0340923–e0340923. 14 indexed citations
8.
Dietschmann, Axel, Peter J. Murray, Claudia Günther, et al.. (2023). Th2‐dependent disappearance and phenotypic conversion of mouse alveolar macrophages. European Journal of Immunology. 53(10). e2350475–e2350475. 4 indexed citations
9.
Dietschmann, Axel, et al.. (2023). Th2-dependent STAT6-regulated genes in intestinal epithelial cells mediate larval trapping during secondary Heligmosomoides polygyrus bakeri infection. PLoS Pathogens. 19(4). e1011296–e1011296. 2 indexed citations
10.
Chao, Ying-Yin, Thomas Seeholzer, Marlot van der Wal, et al.. (2023). Human TH17 cells engage gasdermin E pores to release IL-1α on NLRP3 inflammasome activation. Nature Immunology. 24(2). 295–308. 47 indexed citations
11.
Dietschmann, Axel, et al.. (2023). STAT6‐induced production of mucus and resistin‐like molecules in lung Club cells does not protect against helminth or influenza A virus infection. European Journal of Immunology. 54(1). e2350558–e2350558. 2 indexed citations
12.
Pekmezović, Marina, Axel Dietschmann, & Mark S. Gresnigt. (2022). Type I interferons during host–fungus interactions: Is antifungal immunity going viral?. PLoS Pathogens. 18(8). e1010740–e1010740. 8 indexed citations
13.
14.
Dietschmann, Axel, et al.. (2022). Siglec-F Promotes IL-33–Induced Cytokine Release from Bone Marrow–Derived Eosinophils Independently of the ITIM and ITIM-like Motif Phosphorylation. The Journal of Immunology. 208(3). 732–744. 11 indexed citations
15.
Dietschmann, Axel, et al.. (2020). Th2 cells promote eosinophil‐independent pathology in a murine model of allergic bronchopulmonary aspergillosis. European Journal of Immunology. 50(7). 1044–1056. 22 indexed citations
16.
Wu, Vincent W., Nils Thieme, Lori B. Huberman, et al.. (2020). The regulatory and transcriptional landscape associated with carbon utilization in a filamentous fungus. Proceedings of the National Academy of Sciences. 117(11). 6003–6013. 69 indexed citations
17.
Dietschmann, Axel, et al.. (2018). Murine eosinophil development and allergic lung eosinophilia are largely dependent on the signaling adaptor GRB2. European Journal of Immunology. 48(11). 1786–1795. 9 indexed citations
18.
Thieme, Nils, Vincent W. Wu, Axel Dietschmann, et al.. (2017). The transcription factor PDR-1 is a multi-functional regulator and key component of pectin deconstruction and catabolism in Neurospora crassa. Biotechnology for Biofuels. 10(1). 149–149. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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