Bernard Terlain
- Rheumatology top 0.5%
- Osteoarthritis Treatment and Mechanisms 24
- Pharmacology top 0.5%
- Inflammatory mediators and NSAID effects 22
- Equine top 2%
- Cancer Research top 5%
- NF-κB Signaling Pathways 10
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- Immune Response and Inflammation 11
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- Peroxisome Proliferator-Activated Receptors 9
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- Eicosanoids and Hypertension Pharmacology 6
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- Nitric Oxide and Endothelin Effects 5
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- Adipokines, Inflammation, and Metabolic Diseases 5
Bernard Terlain
81 papers receiving 3.0k citations
Peers
Comparison fields: 5 of 119
- Rheumatology 1.3k
- Pharmacology 1.0k
- Equine 70
- Endocrine and Autonomic Systems 188
- Cancer Research 311
Countries citing papers authored by Bernard Terlain
This map shows the geographic impact of Bernard Terlain's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bernard Terlain with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bernard Terlain more than expected).
Fields of papers citing papers by Bernard Terlain
This network shows the impact of papers produced by Bernard Terlain. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bernard Terlain. The network helps show where Bernard Terlain may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Bernard Terlain, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2006 | 36 | |
| 2 | 2006 | 11 | |
| 3 | 2006 | 246 | |
| 4 | 2005 | 52 | |
| 5 | 2004 | 71 | |
| 6 | 2002 | 13 | |
| 7 | 2001 | 38 | |
| 8 | 2000 | 118 | |
| 9 | 1999 | 8 | |
| 10 | 1999 | 26 | |
| 11 | 1997 | 163 | |
| 12 | 1997 | 339 | |
| 13 | [Inducible cyclooxygenase. New relationships between non-steroidal anti-inflammatory agents and inhibition of synthesis of prostaglandins]. | 1995 | 11 |
| 14 | 1995 | 19 | |
| 15 | 1994 | 5 | |
| 16 | 1993 | 2 | |
| 17 | 1992 | 17 | |
| 18 | 1990 | 15 | |
| 19 | 1973 | 40 | |
| 20 | [Structure of griselimycin, polypeptide antibiotic extracted from streptomyces cultures. II. Structure of griselimycin]. | 1971 | 8 |
About Bernard Terlain
Bernard Terlain is a scholar working on Pharmacology, Rheumatology and Biochemistry, having authored 84 papers that have together received 3.2k indexed citations. Recurring topics across this work include Osteoarthritis Treatment and Mechanisms (24 papers), Inflammatory mediators and NSAID effects (22 papers), Immune Response and Inflammation (11 papers), NF-κB Signaling Pathways (10 papers), Peroxisome Proliferator-Activated Receptors (9 papers), Eicosanoids and Hypertension Pharmacology (6 papers), Nitric Oxide and Endothelin Effects (5 papers) and Adipokines, Inflammation, and Metabolic Diseases (5 papers). The work is most often cited by research in Rheumatology (1.3k citations), Pharmacology (1.0k citations) and Equine (70 citations). Bernard Terlain has collaborated with scholars based in France, Canada and Belgium. Frequent co-authors include Patrick Netter, Nathalie Presle, Pierre Gillet, P. Netter, Hélène Dumond, Didier Mainard, P. Pottié, Pascale Gégout-Pottie, Corinne Guingamp and Karim Bordji. Their work appears in journals such as Inflammation Research, Journal of Medicinal Chemistry, Osteoarthritis and Cartilage, Life Sciences and FEBS Letters.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.