Benjamin L. Unger

502 total citations
11 papers, 382 citations indexed

About

Benjamin L. Unger is a scholar working on Epidemiology, Immunology and Molecular Biology. According to data from OpenAlex, Benjamin L. Unger has authored 11 papers receiving a total of 382 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Epidemiology, 5 papers in Immunology and 4 papers in Molecular Biology. Recurrent topics in Benjamin L. Unger's work include Respiratory viral infections research (3 papers), Immune Response and Inflammation (2 papers) and Liver physiology and pathology (2 papers). Benjamin L. Unger is often cited by papers focused on Respiratory viral infections research (3 papers), Immune Response and Inflammation (2 papers) and Liver physiology and pathology (2 papers). Benjamin L. Unger collaborates with scholars based in United States, Yemen and Canada. Benjamin L. Unger's co-authors include Shyamala Ganesan, Adam T. Comstock, Marc B. Hershenson, Andrea N. Faris, Umadevi Sajjan, Uma S. Sajjan, Bernard Gonik, Kang Chen, Peter Mancuso and Fernando J. Martínez and has published in prestigious journals such as The Journal of Immunology, Journal of Virology and PLoS Pathogens.

In The Last Decade

Benjamin L. Unger

11 papers receiving 377 citations

Peers

Countries citing papers authored by Benjamin L. Unger

Since Specialization

Citations

This map shows the geographic impact of Benjamin L. Unger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin L. Unger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin L. Unger more than expected).

Fields of papers citing papers by Benjamin L. Unger

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin L. Unger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin L. Unger. The network helps show where Benjamin L. Unger may publish in the future.

Co-authorship network of co-authors of Benjamin L. Unger

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin L. Unger. A scholar is included among the top collaborators of Benjamin L. Unger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin L. Unger. Benjamin L. Unger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

#	Work	Indexed citations
1	Rhinovirus Delays Cell Repolarization in a Model of Injured/Regenerating Human Airway Epithelium 2016 ·American Journal of Respiratory Cell and Molecular Biology ·Andrea N. Faris, Shyamala Ganesan, Asamanja Chattoraj, Sangbrita Chattoraj, Adam T. Comstock, Benjamin L. Unger, Marc B. Hershenson, Umadevi Sajjan	26
2	TLR2 Activation Limits Rhinovirus-Stimulated CXCL-10 by Attenuating IRAK-1–Dependent IL-33 Receptor Signaling in Human Bronchial Epithelial Cells 2016 ·The Journal of Immunology ·Shyamala Ganesan, (unknown), (unknown), Mohammad Farazuddin, (unknown), Benjamin L. Unger, Adam T. Comstock, David Proud, Adam S. Lauring, Uma S. Sajjan	26
3	Maternal vaccination: moving the science forward 2014 ·Human Reproduction Update ·(unknown), Benjamin L. Unger, Bernard Gonik, Kang Chen	49
4	Nod-Like Receptor X-1 Is Required for Rhinovirus-Induced Barrier Dysfunction in Airway Epithelial Cells 2014 ·Journal of Virology ·Benjamin L. Unger, Shyamala Ganesan, Adam T. Comstock, Andrea N. Faris, Marc B. Hershenson, Uma S. Sajjan	68
5	A novel role for the Rho-associated kinase, ROCK, in IL-1-stimulated intestinal epithelial cell responses 2012 ·Cellular Immunology ·B. Rafferty, Benjamin L. Unger, (unknown), Steven P. Tammariello, (unknown), Dennis W. McGee	15
6	Rhinovirus Attenuates Non-typeable Hemophilus influenzae-stimulated IL-8 Responses via TLR2-dependent Degradation of IRAK-1 2012 ·PLoS Pathogens ·Benjamin L. Unger, Andrea N. Faris, Shyamala Ganesan, Adam T. Comstock, Marc B. Hershenson, Umadevi Sajjan	51
7	Aberrantly activated EGFR contributes to enhanced IL-8 expression in COPD airways epithelial cells via regulation of nuclear FoxO3A 2012 ·Thorax ·Shyamala Ganesan, Benjamin L. Unger, Adam T. Comstock, (unknown), Peter Mancuso, Fernando J. Martínez, Umadevi Sajjan	74
8	Social Stress Promotes and γ-Aminobutyric Acid Inhibits Tumor Growth in Mouse Models of Non–Small Cell Lung Cancer 2011 ·Cancer Prevention Research ·Hussein A.N. Al-Wadei, Howard K. Plummer, Mohammad Fahad Ullah, Benjamin L. Unger, Joel R. Brody, Hildegard M. Schuller	55
9	Hepatocyte growth factor and keratinocyte growth factor enhance IL-1-induced IL-8 secretion through different mechanisms in Caco-2 epithelial cells 2010 ·In Vitro Cellular & Developmental Biology - Animal ·Benjamin L. Unger, Dennis W. McGee	6
10	Hepatocyte growth factor enhances IL-1β stimulated IL-8 secretion by Caco-2 epithelial cells 2007 ·In Vitro Cellular & Developmental Biology - Animal ·(unknown), (unknown), (unknown), Benjamin L. Unger, Dennis W. McGee	4
11	Increased cell proliferation characterizes Crohn's disease. 1998 ·PubMed ·Amy Noffsinger, Benjamin L. Unger, (unknown)	8

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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