Belal A. Mohamed

961 total citations
28 papers, 649 citations indexed

About

Belal A. Mohamed is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Surgery. According to data from OpenAlex, Belal A. Mohamed has authored 28 papers receiving a total of 649 indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 17 papers in Cardiology and Cardiovascular Medicine and 4 papers in Surgery. Recurrent topics in Belal A. Mohamed's work include Cardiomyopathy and Myosin Studies (7 papers), Cardiovascular Function and Risk Factors (5 papers) and Cardiac Fibrosis and Remodeling (5 papers). Belal A. Mohamed is often cited by papers focused on Cardiomyopathy and Myosin Studies (7 papers), Cardiovascular Function and Risk Factors (5 papers) and Cardiac Fibrosis and Remodeling (5 papers). Belal A. Mohamed collaborates with scholars based in Germany, Egypt and United Kingdom. Belal A. Mohamed's co-authors include Karl Toischer, Gerd Hasenfuß, Ibrahim M. Adham, Dawid Lbik, Amal Z. Barakat, Tonatiuh Peña, Markus T. Bohnsack, Michael Sacherer, Vincenzo Capece and Susanne Burkhardt and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Circulation and Journal of Clinical Investigation.

In The Last Decade

Belal A. Mohamed

26 papers receiving 644 citations

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Belal A. Mohamed 444 194 113 93 56 28 649
George Caputa 259 0.6× 67 0.3× 49 0.4× 62 0.7× 13 0.2× 9 618
Ronald V. Wade 492 1.1× 457 2.4× 21 0.2× 85 0.9× 18 0.3× 20 883
Harry J. Whitwell 187 0.4× 26 0.1× 65 0.6× 62 0.7× 14 0.3× 26 504
Markus A. Engelen 454 1.0× 630 3.2× 53 0.5× 134 1.4× 9 0.2× 32 988
Gourav Bhardwaj 311 0.7× 70 0.4× 36 0.3× 43 0.5× 7 0.1× 24 537
Clémence Merlen 254 0.6× 118 0.6× 17 0.2× 43 0.5× 9 0.2× 26 414
Thomas A. Noland 629 1.4× 702 3.6× 15 0.1× 35 0.4× 19 0.3× 19 980
Hüseyin Güner 442 1.0× 102 0.5× 26 0.2× 55 0.6× 4 0.1× 22 706
Yoichi Inada 236 0.5× 56 0.3× 20 0.2× 88 0.9× 22 0.4× 34 548
Alpesh Thakker 184 0.4× 33 0.2× 50 0.4× 97 1.0× 7 0.1× 16 359

Countries citing papers authored by Belal A. Mohamed

Since Specialization
Citations

This map shows the geographic impact of Belal A. Mohamed's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Belal A. Mohamed with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Belal A. Mohamed more than expected).

Fields of papers citing papers by Belal A. Mohamed

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Belal A. Mohamed. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Belal A. Mohamed. The network helps show where Belal A. Mohamed may publish in the future.

Co-authorship network of co-authors of Belal A. Mohamed

This figure shows the co-authorship network connecting the top 25 collaborators of Belal A. Mohamed. A scholar is included among the top collaborators of Belal A. Mohamed based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Belal A. Mohamed. Belal A. Mohamed is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Barallobre‐Barreiro, Javier, Moritz Schnelle, Belal A. Mohamed, et al.. (2024). Cellular and extracellular proteomic profiling of paradoxical low-flow low-gradient aortic stenosis myocardium. Frontiers in Cardiovascular Medicine. 11. 1398114–1398114. 2 indexed citations
2.
Ashraf, Mohammad, et al.. (2024). Dundee Annual Neurosurgery Skills Event (DANSE)—Improving the Availability and Affordability of Neurosurgical Skills Workshops for Medical Students. Asian Journal of Neurosurgery. 19(1). 63–72. 3 indexed citations
3.
Mohamed, Belal A., et al.. (2023). A suspension of inactivated bacteria used for vaccination against recurrent urinary tract infections increases the phagocytic activity of murine macrophages. Frontiers in Immunology. 14. 1180785–1180785. 2 indexed citations
4.
Xu, Xingbo, Xiao-Ying Tan, Moritz Schnelle, et al.. (2023). DNA Methylation Analysis Identifies Novel Epigenetic Loci in Dilated Murine Heart upon Exposure to Volume Overload. International Journal of Molecular Sciences. 24(6). 5885–5885.
5.
Hartmann, Nico, Belal A. Mohamed, Moritz Schnelle, et al.. (2022). Different Activation of MAPKs and Akt/GSK3β After Preload vs. Afterload Elevation. ESC Heart Failure. 9(3). 1823–1831. 2 indexed citations
6.
Barakat, Amal Z., et al.. (2022). Heat shock protein A4 ablation leads to skeletal muscle myopathy associated with dysregulated autophagy and induced apoptosis. Journal of Translational Medicine. 20(1). 229–229. 11 indexed citations
7.
Bengel, Philipp, Nataliya Dybkova, Petros Tirilomis, et al.. (2021). Detrimental proarrhythmogenic interaction of Ca2+/calmodulin-dependent protein kinase II and NaV1.8 in heart failure. Nature Communications. 12(1). 6586–6586. 19 indexed citations
8.
Mohamed, Belal A., et al.. (2021). Murine Embryonic Stem Cell Culture, Self-Renewal, and Differentiation. Methods in molecular biology. 2520. 265–273. 2 indexed citations
9.
Khadjeh, Sara, Lukas Cyganek, Ramón Vidal, et al.. (2020). CRISPLD1: a novel conserved target in the transition to human heart failure. Basic Research in Cardiology. 115(3). 27–27. 9 indexed citations
10.
Markus, M. Andrea, et al.. (2020). X-ray diffraction and second harmonic imaging reveal new insights into structural alterations caused by pressure-overload in murine hearts. Scientific Reports. 10(1). 19317–19317. 10 indexed citations
11.
Schnelle, Moritz, Iain A. Sawyer, Narayana Anilkumar, et al.. (2019). NADPH oxidase-4 promotes eccentric cardiac hypertrophy in response to volume overload. Cardiovascular Research. 117(1). 178–187. 24 indexed citations
12.
Mohamed, Belal A., Dawid Lbik, Lutz Binder, et al.. (2019). Assessing the Role of Extracellular Signal-Regulated Kinases 1 and 2 in Volume Overload-Induced Cardiac Remodelling. ESC Heart Failure. 6(5). 1015–1026. 5 indexed citations
13.
14.
Mohamed, Belal A., Nico Hartmann, Petros Tirilomis, et al.. (2018). Sarcoplasmic reticulum calcium leak contributes to arrhythmia but not to heart failure progression. Science Translational Medicine. 10(458). 27 indexed citations
15.
Lbik, Dawid, Sara Khadjeh, Belal A. Mohamed, et al.. (2017). Abstract 20724: The Absence of the Chromatin Reader Brd2 Decreases Heart Function and Increases Mortality After Pressure Overload. Circulation. 1 indexed citations
16.
Toischer, Karl, Wuqiang Zhu, Mark Hünlich, et al.. (2017). Cardiomyocyte proliferation prevents failure in pressure overload but not volume overload. Journal of Clinical Investigation. 127(12). 4285–4296. 33 indexed citations
17.
Mohamed, Belal A., Abdul R. Asif, Moritz Schnelle, et al.. (2016). Proteomic analysis of short-term preload-induced eccentric cardiac hypertrophy. Journal of Translational Medicine. 14(1). 149–149. 11 indexed citations
18.
Mohamed, Belal A., Moritz Schnelle, Sara Khadjeh, et al.. (2015). Molecular and Structural Transition Mechanisms in Long-Term Volume Overload. European Journal of Heart Failure. 18(4). 362–371. 46 indexed citations
19.
Mohamed, Belal A., Amal Z. Barakat, Wolfram‐Hubertus Zimmermann, et al.. (2012). Targeted disruption of Hspa4 gene leads to cardiac hypertrophy and fibrosis. Journal of Molecular and Cellular Cardiology. 53(4). 459–468. 35 indexed citations
20.
Held, Torsten, Amal Z. Barakat, Belal A. Mohamed, et al.. (2011). Heat-shock protein HSPA4 is required for progression of spermatogenesis. Reproduction. 142(1). 133–144. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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