Michael Didié

4.8k total citations · 2 hit papers
40 papers, 3.0k citations indexed

About

Michael Didié is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Surgery. According to data from OpenAlex, Michael Didié has authored 40 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 18 papers in Cardiology and Cardiovascular Medicine and 16 papers in Surgery. Recurrent topics in Michael Didié's work include Tissue Engineering and Regenerative Medicine (14 papers), Electrospun Nanofibers in Biomedical Applications (11 papers) and Cardiac electrophysiology and arrhythmias (8 papers). Michael Didié is often cited by papers focused on Tissue Engineering and Regenerative Medicine (14 papers), Electrospun Nanofibers in Biomedical Applications (11 papers) and Cardiac electrophysiology and arrhythmias (8 papers). Michael Didié collaborates with scholars based in Germany, United States and Czechia. Michael Didié's co-authors include Thomas Eschenhagen, Wolfram‐Hubertus Zimmermann, Ivan Melnychenko, Karin Schneiderbanger, Hiroshi Naito, Winfried Neuhuber, Jürgen F. Heubach, Philipp Schubert, Felix Münzel and Sawa Kostin and has published in prestigious journals such as Circulation, Journal of Clinical Investigation and Nature Medicine.

In The Last Decade

Michael Didié

39 papers receiving 3.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Engineered heart tissue grafts improve systolic and diastolic function in infarcted rat hearts

2006 733 citations Wolfram‐Hubertus Zimmermann, Ivan Melnychenko et al. profile →
Tissue Engineering of a Differentiated Cardiac Muscle Construct

2002 695 citations Wolfram‐Hubertus Zimmermann, Karin Schneiderbanger et al. Circulation Research profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Michael Didié Germany	21	2.0k	1.5k	1.2k	920	544	40	3.0k
Wolfram H. Zimmermann Germany	20	1.3k 0.6×	879 0.6×	1.2k 1.0×	698 0.8×	572 1.1×	37	2.5k
Claudia Bearzi Italy	26	1.6k 0.8×	674 0.4×	1.7k 1.4×	588 0.6×	470 0.9×	58	3.0k
Maria Papadaki United States	18	934 0.5×	766 0.5×	470 0.4×	616 0.7×	398 0.7×	36	1.7k
Wuqiang Zhu United States	29	1.1k 0.6×	564 0.4×	1.8k 1.6×	420 0.5×	987 1.8×	78	3.2k
Kiwon Ban Hong Kong	25	1.1k 0.6×	384 0.3×	1.4k 1.2×	589 0.6×	672 1.2×	55	3.5k
Jürgen F. Heubach Germany	22	758 0.4×	518 0.3×	1.1k 1.0×	395 0.4×	700 1.3×	30	2.4k
Elina Minami United States	16	1.8k 0.9×	743 0.5×	2.4k 2.0×	557 0.6×	511 0.9×	27	3.4k
Gil Arbel Israel	22	2.0k 1.0×	852 0.6×	3.3k 2.8×	1.1k 1.2×	1.1k 2.0×	31	4.6k
Xiulan Yang United States	18	1.1k 0.6×	350 0.2×	1.8k 1.5×	793 0.9×	647 1.2×	30	2.8k
Elisa Garbayo Spain	28	575 0.3×	787 0.5×	790 0.7×	515 0.6×	129 0.2×	52	2.1k

Countries citing papers authored by Michael Didié

Since Specialization

Citations

This map shows the geographic impact of Michael Didié's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Didié with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Didié more than expected).

Fields of papers citing papers by Michael Didié

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael Didié. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Didié. The network helps show where Michael Didié may publish in the future.

Co-authorship network of co-authors of Michael Didié

This figure shows the co-authorship network connecting the top 25 collaborators of Michael Didié. A scholar is included among the top collaborators of Michael Didié based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael Didié. Michael Didié is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kutschka, Ingo, Sören Brandenburg, Felix Bremmer, et al.. (2025). Two-Center Surgical Experience from the First-in-Human BioVAT-HF-DZHK20 Clinical Trial. The Journal of Heart and Lung Transplantation. 44(4). S64–S65.

Tiburcy, Malte, Daniel Biermann, Paul Balfanz, et al.. (2022). Transmural myocardial repair with engineered heart muscle in a rat model of heterotopic heart transplantation – A proof-of-concept study. Journal of Molecular and Cellular Cardiology. 168. 3–12. 7 indexed citations

Schnieder, Marlena, Michael Didié, Mostafa Z. Badr, et al.. (2021). Comparing the diagnostic value of Echocardiography In Stroke (CEIS) – results of a prospective observatory cohort study. BMC Neurology. 21(1). 118–118. 1 indexed citations

Mohamed, Belal A., Nico Hartmann, Petros Tirilomis, et al.. (2018). Sarcoplasmic reticulum calcium leak contributes to arrhythmia but not to heart failure progression. Science Translational Medicine. 10(458). 27 indexed citations

Monecke, Sebastian, Leslie Elsner, Michael Didié, et al.. (2018). CD8+-T Cells With Specificity for a Model Antigen in Cardiomyocytes Can Become Activated After Transverse Aortic Constriction but Do Not Accelerate Progression to Heart Failure. Frontiers in Immunology. 9. 2665–2665. 22 indexed citations

Monecke, Sebastian, Michael Didié, Leslie Elsner, et al.. (2017). T helper cells with specificity for an antigen in cardiomyocytes promote pressure overload-induced progression from hypertrophy to heart failure. Scientific Reports. 7(1). 15998–15998. 29 indexed citations

Didié, Michael, et al.. (2017). Immunological Properties of Murine Parthenogenetic Stem Cell-Derived Cardiomyocytes and Engineered Heart Muscle. Frontiers in Immunology. 8. 955–955. 7 indexed citations

Bremer, Sebastian, Kristian Hellenkamp, Nico Hartmann, et al.. (2016). Enhanced cardiac TBC1D10C expression lowers heart rate and enhances exercise capacity and survival. Scientific Reports. 6(1). 33853–33853. 4 indexed citations

Yang, Tao, Michael Rubart, Mark H. Soonpaa, et al.. (2015). Cardiac Engraftment of Genetically-Selected Parthenogenetic Stem Cell-Derived Cardiomyocytes. PLoS ONE. 10(6). e0131511–e0131511. 3 indexed citations

10.

Li, Xiao, Henrike J. Fischer, Michael Didié, et al.. (2015). Inducible Knock-Down of the Mineralocorticoid Receptor in Mice Disturbs Regulation of the Renin-Angiotensin-Aldosterone System and Attenuates Heart Failure Induced by Pressure Overload. PLoS ONE. 10(11). e0143954–e0143954. 10 indexed citations

11.

Leifheit‐Nestler, Maren, Nana‐Maria Wagner, Rajinikanth Gogiraju, et al.. (2013). Importance of leptin signaling and signal transducer and activator of transcription-3 activation in mediating the cardiac hypertrophy associated with obesity. Journal of Translational Medicine. 11(1). 170–170. 43 indexed citations

12.

Heilmann, Andreas, Michael Didié, Saskia Schlossarek, et al.. (2012). Impact of AT2 Receptor Deficiency on Postnatal Cardiovascular Development. PLoS ONE. 7(10). e47916–e47916. 10 indexed citations

13.

Grebe, Cornelia, Karl Toischer, Michael Didié, et al.. (2011). Enhanced expression of DYRK1A in cardiomyocytes inhibits acute NFAT activation but does not prevent hypertrophy in vivo. Cardiovascular Research. 90(3). 521–528. 16 indexed citations

14.

Vantler, Marius, Bijoy Chandapillai Karikkineth, Hiroshi Naito, et al.. (2010). PDGF-BB protects cardiomyocytes from apoptosis and improves contractile function of engineered heart tissue. Journal of Molecular and Cellular Cardiology. 48(6). 1316–1323. 34 indexed citations

15.

Sossalla, Samuel, Hanna Schotola, Nico Hartmann, et al.. (2010). Diastolic dysfunction and arrhythmias caused by overexpression of CaMKIIδC can be reversed by inhibition of late Na+ current. Basic Research in Cardiology. 106(2). 263–272. 81 indexed citations

16.

El‐Armouche, Ali, Katrin Wittköpper, Florian Weinberger, et al.. (2008). Phosphatase inhibitor-1-deficient mice are protected from catecholamine-induced arrhythmias and myocardial hypertrophy. Cardiovascular Research. 80(3). 396–406. 80 indexed citations

17.

El‐Armouche, Ali, Hiroshi Naito, Katrin Wittköpper, et al.. (2007). Adenovirus-delivered short hairpin RNA targeting PKCα improves contractile function in reconstituted heart tissue. Journal of Molecular and Cellular Cardiology. 43(3). 371–376. 27 indexed citations

18.

Rust, Marco B., Jörg Faulhaber, Carsten K. Pfeffer, et al.. (2006). Neurogenic Mechanisms Contribute to Hypertension in Mice With Disruption of the K-Cl Cotransporter KCC3. Circulation Research. 98(4). 549–556. 40 indexed citations

19.

El‐Armouche, Ali, et al.. (2006). Role of calcineurin and protein phosphatase-2A in the regulation of phosphatase inhibitor-1 in cardiac myocytes. Biochemical and Biophysical Research Communications. 346(3). 700–706. 55 indexed citations

20.

Münzel, Felix, Wolfram‐Hubertus Zimmermann, Michael Didié, et al.. (2005). Endothelin-1 and isoprenaline co-stimulation causes contractile failure which is partially reversed by MEK inhibition. Cardiovascular Research. 68(3). 464–474. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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