Beatriz Marcos‐Ramiro

969 total citations
15 papers, 740 citations indexed

About

Beatriz Marcos‐Ramiro is a scholar working on Molecular Biology, Cell Biology and Immunology. According to data from OpenAlex, Beatriz Marcos‐Ramiro has authored 15 papers receiving a total of 740 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 5 papers in Cell Biology and 5 papers in Immunology. Recurrent topics in Beatriz Marcos‐Ramiro's work include Cell Adhesion Molecules Research (4 papers), Signaling Pathways in Disease (3 papers) and Protein Degradation and Inhibitors (2 papers). Beatriz Marcos‐Ramiro is often cited by papers focused on Cell Adhesion Molecules Research (4 papers), Signaling Pathways in Disease (3 papers) and Protein Degradation and Inhibitors (2 papers). Beatriz Marcos‐Ramiro collaborates with scholars based in Spain, United Kingdom and United States. Beatriz Marcos‐Ramiro's co-authors include Jaime Millán, Natalia Reglero-Real, Isabel Correas, Laura Fernández‐Martín, Anne J. Ridley, Carolina L. Bigarella, Robert J. Cain, Eva Cernuda‐Morollón, Miguel A. Alonso and Silvia Ortega‐Gutiérrez and has published in prestigious journals such as Nature Communications, The Journal of Cell Biology and International Journal of Molecular Sciences.

In The Last Decade

Beatriz Marcos‐Ramiro

15 papers receiving 732 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Beatriz Marcos‐Ramiro Spain 12 398 197 116 93 84 15 740
Natalia Reglero-Real Spain 12 393 1.0× 265 1.3× 140 1.2× 101 1.1× 91 1.1× 15 761
Cora Beckers Netherlands 12 356 0.9× 136 0.7× 179 1.5× 110 1.2× 107 1.3× 15 730
Matthias Vockel Germany 9 388 1.0× 108 0.5× 161 1.4× 152 1.6× 105 1.3× 10 673
Michael Broman United States 18 615 1.5× 231 1.2× 123 1.1× 139 1.5× 94 1.1× 34 1.3k
I‐Chu Tseng United States 13 256 0.6× 111 0.6× 92 0.8× 87 0.9× 51 0.6× 15 630
Raudel Sandoval United States 13 504 1.3× 227 1.2× 184 1.6× 110 1.2× 48 0.6× 15 1.0k
Wenwen Tang China 18 533 1.3× 164 0.8× 240 2.1× 107 1.2× 24 0.3× 40 967
Giulia Germena Germany 13 399 1.0× 216 1.1× 199 1.7× 122 1.3× 18 0.2× 21 739
Ulrich A. Hirt Germany 9 389 1.0× 88 0.4× 209 1.8× 50 0.5× 56 0.7× 11 729
Dustin S. Chang United States 5 557 1.4× 74 0.4× 120 1.0× 49 0.5× 28 0.3× 6 858

Countries citing papers authored by Beatriz Marcos‐Ramiro

Since Specialization
Citations

This map shows the geographic impact of Beatriz Marcos‐Ramiro's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beatriz Marcos‐Ramiro with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beatriz Marcos‐Ramiro more than expected).

Fields of papers citing papers by Beatriz Marcos‐Ramiro

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Beatriz Marcos‐Ramiro. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beatriz Marcos‐Ramiro. The network helps show where Beatriz Marcos‐Ramiro may publish in the future.

Co-authorship network of co-authors of Beatriz Marcos‐Ramiro

This figure shows the co-authorship network connecting the top 25 collaborators of Beatriz Marcos‐Ramiro. A scholar is included among the top collaborators of Beatriz Marcos‐Ramiro based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Beatriz Marcos‐Ramiro. Beatriz Marcos‐Ramiro is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Angelina, Alba, Angel Maldonado, Begoña Lavín, et al.. (2025). A mucosal vaccine prevents eosinophilic allergic airway inflammation by modulating immune responses to allergens in a murine model of airway disease. Nature Communications. 16(1). 7129–7129. 1 indexed citations
2.
Marcos‐Ramiro, Beatriz, et al.. (2021). Small-Molecule Therapeutic Perspectives for the Treatment of Progeria. International Journal of Molecular Sciences. 22(13). 7190–7190. 15 indexed citations
3.
Marcos‐Ramiro, Beatriz, Nagore I. Marín‐Ramos, Pilar Gonzalo, et al.. (2021). Isoprenylcysteine Carboxylmethyltransferase-Based Therapy for Hutchinson–Gilford Progeria Syndrome. ACS Central Science. 7(8). 1300–1310. 16 indexed citations
4.
Marcos‐Ramiro, Beatriz, et al.. (2020). Chemistry for the Identification of Therapeutic Targets: Recent Advances and Future Directions. European Journal of Organic Chemistry. 2021(9). 1307–1320. 6 indexed citations
5.
Marín‐Ramos, Nagore I., Iván R. Torrecillas, Beatriz Marcos‐Ramiro, et al.. (2019). A Potent Isoprenylcysteine Carboxylmethyltransferase (ICMT) Inhibitor Improves Survival in Ras-Driven Acute Myeloid Leukemia. Journal of Medicinal Chemistry. 62(13). 6035–6046. 36 indexed citations
6.
Ortega, María Cristina, Beatriz Marcos‐Ramiro, Susana Barroso, et al.. (2016). Activation of Rac1 and RhoA Preserve Corneal Endothelial Barrier Function. Investigative Ophthalmology & Visual Science. 57(14). 6210–6210. 11 indexed citations
7.
Marcos‐Ramiro, Beatriz, Susana Barroso, Jorge Feito, et al.. (2016). RhoB controls endothelial barrier recovery by inhibiting Rac1 trafficking to the cell border. The Journal of Cell Biology. 213(3). 385–402. 56 indexed citations
8.
Marcos‐Ramiro, Beatriz, Esther Serrano‐Pertierra, Miguel Ángel Blanco-Gelaz, et al.. (2014). Microparticles in multiple sclerosis and clinically isolated syndrome: effect on endothelial barrier function. BMC Neuroscience. 15(1). 110–110. 83 indexed citations
9.
Reglero-Real, Natalia, Adrián Álvarez-Varela, Eva Cernuda‐Morollón, et al.. (2014). Apicobasal Polarity Controls Lymphocyte Adhesion to Hepatic Epithelial Cells. Cell Reports. 8(6). 1879–1893. 13 indexed citations
10.
Marcos‐Ramiro, Beatriz, et al.. (2014). TNF-induced endothelial barrier disruption: beyond actin and Rho. Thrombosis and Haemostasis. 112(12). 1088–1102. 152 indexed citations
11.
Reglero-Real, Natalia, Beatriz Marcos‐Ramiro, & Jaime Millán. (2012). Endothelial membrane reorganization during leukocyte extravasation. Cellular and Molecular Life Sciences. 69(18). 3079–3099. 44 indexed citations
12.
Aranda, Juan, Natalia Reglero-Real, Beatriz Marcos‐Ramiro, et al.. (2012). MYADM controls endothelial barrier function through ERM-dependent regulation of ICAM-1 expression. Molecular Biology of the Cell. 24(4). 483–494. 33 indexed citations
13.
Fernández‐Martín, Laura, Beatriz Marcos‐Ramiro, Carolina L. Bigarella, et al.. (2012). Crosstalk Between Reticular Adherens Junctions and Platelet Endothelial Cell Adhesion Molecule-1 Regulates Endothelial Barrier Function. Arteriosclerosis Thrombosis and Vascular Biology. 32(8). e90–102. 56 indexed citations
14.
Aranda, Juan, Natalia Reglero-Real, Leonor Kremer, et al.. (2011). MYADM regulates Rac1 targeting to ordered membranes required for cell spreading and migration. Molecular Biology of the Cell. 22(8). 1252–1262. 42 indexed citations
15.
Millán, Jaime, Robert J. Cain, Natalia Reglero-Real, et al.. (2010). Adherens junctions connect stress fibres between adjacent endothelial cells. BMC Biology. 8(1). 11–11. 176 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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