Beatriz Domínguez-Molina

456 total citations
13 papers, 234 citations indexed

About

Beatriz Domínguez-Molina is a scholar working on Virology, Immunology and Hepatology. According to data from OpenAlex, Beatriz Domínguez-Molina has authored 13 papers receiving a total of 234 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Virology, 7 papers in Immunology and 5 papers in Hepatology. Recurrent topics in Beatriz Domínguez-Molina's work include HIV Research and Treatment (10 papers), Hepatitis C virus research (5 papers) and HIV-related health complications and treatments (4 papers). Beatriz Domínguez-Molina is often cited by papers focused on HIV Research and Treatment (10 papers), Hepatitis C virus research (5 papers) and HIV-related health complications and treatments (4 papers). Beatriz Domínguez-Molina collaborates with scholars based in Spain, United States and Chile. Beatriz Domínguez-Molina's co-authors include Ezequiel Ruíz-Mateos, Laura Tarancón‐Díez, M. Leal, Pompeyo Viciana, Manuel Leal, Sara Ferrando‐Martínez, María Ángeles Muñoz‐Fernández, Núria Espinosa, Omar J. BenMarzouk‐Hidalgo and María Trujillo‐Rodríguez and has published in prestigious journals such as Clinical Infectious Diseases, Journal of Virology and The Journal of Infectious Diseases.

In The Last Decade

Beatriz Domínguez-Molina

13 papers receiving 232 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Beatriz Domínguez-Molina Spain 10 120 83 69 60 59 13 234
Kawthar Machmach Spain 11 236 2.0× 209 2.5× 92 1.3× 50 0.8× 40 0.7× 20 346
Qingxia Zhao China 9 139 1.2× 66 0.8× 154 2.2× 21 0.3× 32 0.5× 32 266
Manon Nayrac Canada 8 50 0.4× 52 0.6× 88 1.3× 59 1.0× 14 0.2× 14 171
Anne-Sophie Liovat France 7 240 2.0× 213 2.6× 100 1.4× 11 0.2× 65 1.1× 7 342
Desmond Persad Canada 6 138 1.1× 137 1.7× 60 0.9× 8 0.1× 31 0.5× 7 248
Surekha Tennakoon Australia 9 180 1.5× 91 1.1× 112 1.6× 23 0.4× 83 1.4× 13 326
Jette Victor Denmark 7 285 2.4× 257 3.1× 90 1.3× 27 0.5× 43 0.7× 8 397
Chiara Taibi Italy 8 61 0.5× 33 0.4× 58 0.8× 138 2.3× 10 0.2× 36 262
Matteo Surdo Italy 9 104 0.9× 21 0.3× 93 1.3× 62 1.0× 18 0.3× 16 237
Johanna Repits Sweden 9 208 1.7× 113 1.4× 124 1.8× 14 0.2× 19 0.3× 11 268

Countries citing papers authored by Beatriz Domínguez-Molina

Since Specialization
Citations

This map shows the geographic impact of Beatriz Domínguez-Molina's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beatriz Domínguez-Molina with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beatriz Domínguez-Molina more than expected).

Fields of papers citing papers by Beatriz Domínguez-Molina

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Beatriz Domínguez-Molina. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beatriz Domínguez-Molina. The network helps show where Beatriz Domínguez-Molina may publish in the future.

Co-authorship network of co-authors of Beatriz Domínguez-Molina

This figure shows the co-authorship network connecting the top 25 collaborators of Beatriz Domínguez-Molina. A scholar is included among the top collaborators of Beatriz Domínguez-Molina based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Beatriz Domínguez-Molina. Beatriz Domínguez-Molina is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Tarancón‐Díez, Laura, Beatriz Domínguez-Molina, Isabel Gallego, et al.. (2023). Toll-like receptor agonists enhance HIV-specific T cell response mediated by plasmacytoid dendritic cells in diverse HIV-1 disease progression phenotypes. EBioMedicine. 91. 104549–104549. 10 indexed citations
2.
Ruíz-Mateos, Ezequiel, Laura Tarancón‐Díez, Carolina Gutiérrez, et al.. (2019). High CD8 T cell percentage and HCV replication control are common features in HIV-1 controllers and HTLV-2-co-infected patients with a history of injection drug use. Virus Research. 264. 40–44. 6 indexed citations
3.
López‐Cortés, Luís F., María Trujillo‐Rodríguez, Omar J. BenMarzouk‐Hidalgo, et al.. (2018). Eradication of Hepatitis C Virus (HCV) Reduces Immune Activation, Microbial Translocation, and the HIV DNA Level in HIV/HCV-Coinfected Patients. The Journal of Infectious Diseases. 218(4). 624–632. 41 indexed citations
4.
Domínguez-Molina, Beatriz, Sara Ferrando‐Martínez, Laura Tarancón‐Díez, et al.. (2018). Immune Correlates of Natural HIV Elite Control and Simultaneous HCV Clearance—Supercontrollers. Frontiers in Immunology. 9. 2897–2897. 13 indexed citations
5.
Rodríguez‐Gallego, Esther, Laura Tarancón‐Díez, Felipe García, et al.. (2018). Proteomic Profile Associated With Loss of Spontaneous Human Immunodeficiency Virus Type 1 Elite Control. The Journal of Infectious Diseases. 219(6). 867–876. 23 indexed citations
6.
Domínguez-Molina, Beatriz, Kawthar Machmach, Celia Perales, et al.. (2018). Toll-Like Receptor 7 (TLR-7) and TLR-9 Agonists Improve Hepatitis C Virus Replication and Infectivity Inhibition by Plasmacytoid Dendritic Cells. Journal of Virology. 92(23). 19 indexed citations
7.
González‐Serna, Alejandro, Sara Ferrando‐Martínez, Laura Tarancón‐Díez, et al.. (2017). Increased CD127+ and decreased CD57+ T cell expression levels in HIV-infected patients on NRTI-sparing regimens. Journal of Translational Medicine. 15(1). 259–259. 8 indexed citations
8.
Ruíz-Mateos, Ezequiel, Laura Tarancón‐Díez, Ana Isabel Álvarez‐Ríos, et al.. (2017). Association of heterozygous CCR5Δ32 deletion with survival in HIV-infection: A cohort study. Antiviral Research. 150. 15–19. 5 indexed citations
9.
Tarancón‐Díez, Laura, Ana Isabel Álvarez‐Ríos, Isaac Rosado‐Sánchez, et al.. (2017). CCR5+ CD8 T-cell levels and monocyte activation precede the onset of acute coronary syndrome in HIV-infected patients on antiretroviral therapy. Thrombosis and Haemostasis. 117(6). 1141–1149. 11 indexed citations
10.
Domínguez-Molina, Beatriz, Agathe León, Carmen Rodríguez, et al.. (2016). Analysis of Non-AIDS-Defining Events in HIV Controllers. Clinical Infectious Diseases. 62(10). 1304–1309. 31 indexed citations
11.
Machmach, Kawthar, Cristina Abad‐Molina, Beatriz Domínguez-Molina, et al.. (2014). IFNL4 ss469415590 polymorphism is associated with unfavourable clinical and immunological status in HIV-infected individuals. Clinical Microbiology and Infection. 21(3). 289.e1–289.e4. 17 indexed citations
12.
Ferrando‐Martínez, Sara, Ezequiel Ruíz-Mateos, Joseph P. Casazza, et al.. (2014). IFNγ−TNFα−IL2−MIP1α−CD107a+PRF1+CD8 pp65-Specific T-Cell Response Is Independently Associated With Time to Death in Elderly Humans. The Journals of Gerontology Series A. 70(10). 1210–1218. 12 indexed citations
13.
Ruíz-Mateos, Ezequiel, Beatriz Domínguez-Molina, Antonio Carrillo‐Vico, et al.. (2014). TNF-  levels in HIV-infected patients after long-term suppressive cART persist as high as in elderly, HIV-uninfected subjects. Journal of Antimicrobial Chemotherapy. 69(11). 3041–3046. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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